BID links ferroptosis to mitochondrial cell death pathways

Redox Biology

Volumn 12, Issue , 2017, Pages 558-570

  Neitemeier, Sandra ^a   Jelinek, Anja ^a   Laino, Vincenzo ^a   Hoffmann, Lena ^a   Eisenbach, Ina ^a   Eying, Roman ^a   Ganjam, Goutham K ^a   Dolga, Amalia M ^a   Oppermann, Sina ^a   Culmsee, Carsten ^a  

^a PHILIPPS UNIVERSITY MARBURG   (Germany)

Author keywords

BID; CRISPR; Ferroptosis; Mitochondria; Neuronal death; Oxytosis

Indexed keywords

CASPASE 9; CRISPR ASSOCIATED PROTEIN; PROTEIN BID; BID PROTEIN, MOUSE; ERASTIN; PIPERAZINE DERIVATIVE; REACTIVE OXYGEN METABOLITE;

ANIMAL CELL; ARTICLE; CELL DEATH; CELL VIABILITY; CONFOCAL MICROSCOPY; CONTROLLED STUDY; FERROPTOSIS; FLUORESCENCE ACTIVATED CELL SORTING; FLUORESCENCE MICROSCOPY; INDEL MUTATION; LIPID PEROXIDATION; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRION; MORPHOLOGY; MOUSE; NONHUMAN; OXYGEN CONSUMPTION; OXYTOSIS; PLASMID; PROTEIN ANALYSIS; PROTEIN DEPLETION; PROTEIN FUNCTION; SIGNAL TRANSDUCTION; TRANSACTIVATION; ANIMAL; CELL LINE; CRISPR CAS SYSTEM; CYTOLOGY; DRUG EFFECTS; GENE KNOCKOUT; GENETICS; METABOLISM; NERVE CELL; OXIDATIVE STRESS; PHYSIOLOGY;

ANIMALS; BH3 INTERACTING DOMAIN DEATH AGONIST PROTEIN; CELL DEATH; CELL LINE; CRISPR-CAS SYSTEMS; GENE KNOCKOUT TECHNIQUES; LIPID PEROXIDATION; MEMBRANE POTENTIAL, MITOCHONDRIAL; MICE; MITOCHONDRIA; NEURONS; OXIDATIVE STRESS; PIPERAZINES; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION;

EID: 85016771461     PISSN: 22132317     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.redox.2017.03.007     Document Type: Article

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