Single-chromosome Gains Commonly Function as Tumor Suppressors

Cancer Cell

Volumn 31, Issue 2, 2017, Pages 240-255

  Sheltzer, Jason M ^a,b   Ko, Julie H ^b   Replogle, John M ^b   Habibe Burgos, Nicole C ^b   Chung, Erica S ^c   Meehl, Colleen M ^b   Sayles, Nicole M ^a   Passerini, Verena ^d   Storchova, Zuzana ^d,e   Amon, Angelika ^b  

^a Simons Center for Quantitative Biology   (United States)

^b MASSACHUSETTS INSTITUTE OF TECHNOLOGY   (United States)

^c ALBERT EINSTEIN COLLEGE OF MEDICINE   (United States)

^d MAX PLANCK INSTITUTE OF BIOCHEMISTRY   (Germany)

^e University of Koblenz Landau   (Germany)

Author keywords

aneuploidy; chromosomal instability; genome dosage imbalance; genomic instability; transformation

Indexed keywords

ANEUPLOIDY; ARTICLE; CANCER INHIBITION; CARCINOGENESIS; CARCINOGENICITY; CELL ENGINEERING; CELL GROWTH; CELL PROLIFERATION; COLORECTAL CANCER CELL LINE; HUMAN; IN VITRO STUDY; KARYOTYPE EVOLUTION; M PHASE CELL CYCLE CHECKPOINT; NONHUMAN; ONCOGENE; PHENOTYPE; PRIORITY JOURNAL; SUPERNUMERARY CHROMOSOME; TRISOMIC CELL LINE; TUMOR XENOGRAFT; ANIMAL; CELL TRANSFORMATION; CHROMOSOME ABERRATION; GENETICS; GENOMIC INSTABILITY; HCT 116 CELL LINE; NEOPLASMS; ONCOGENE RAS;

ANEUPLOIDY; ANIMALS; CELL PROLIFERATION; CELL TRANSFORMATION, NEOPLASTIC; CHROMOSOME ABERRATIONS; GENES, RAS; GENOMIC INSTABILITY; HCT116 CELLS; HUMANS; NEOPLASMS; ONCOGENES;

EID: 85009785424     PISSN: 15356108     EISSN: 18783686     Source Type: Journal    
DOI: 10.1016/j.ccell.2016.12.004     Document Type: Article

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