-
1
-
-
84940795715
-
Update of mortality attributable to diabetes for the IDF diabetes atlas: Estimates for the year 2013
-
IDF Diabetes Atlas Group, "Update of mortality attributable to diabetes for the IDF Diabetes Atlas: estimates for the year 2013," Diabetes Research and Clinical Practice, vol. 109, no. 3, pp. 461-465, 2015.
-
(2015)
Diabetes Research and Clinical Practice
, vol.109
, Issue.3
, pp. 461-465
-
-
-
2
-
-
33845992169
-
Global and regional mortality from ischaemic heart disease and stroke attributable to higher-than-optimum blood glucose concentration: Comparative risk assessment
-
G. Danaei, C. M. Lawes, S. Vander Hoorn, C. J. Murray, and M. Ezzati, "Global and regional mortality from ischaemic heart disease and stroke attributable to higher-than-optimum blood glucose concentration: comparative risk assessment," The Lancet, vol. 368, no. 9548, pp. 1651-1659, 2006.
-
(2006)
The Lancet
, vol.368
, Issue.9548
, pp. 1651-1659
-
-
Danaei, G.1
Lawes, C.M.2
Vander Hoorn, S.3
Murray, C.J.4
Ezzati, M.5
-
3
-
-
34347352169
-
Diabetic cardiomyopathy revisited
-
S. Boudina and E.D. Abel, "Diabetic cardiomyopathy revisited," Circulation, vol. 115, no. 25, pp. 3213-3223, 2007.
-
(2007)
Circulation
, vol.115
, Issue.25
, pp. 3213-3223
-
-
Boudina, S.1
Abel, E.D.2
-
4
-
-
0026321248
-
Echocardiographic evidence for the existence of a distinct diabetic cardiomyopathy (The framingham heart study)
-
M. Galderisi, K. M. Anderson, P. W. F. Wilson, and D. Levy, "Echocardiographic evidence for the existence of a distinct diabetic cardiomyopathy (The Framingham Heart Study)," The American Journal of Cardiology, vol. 68, no. 1, pp. 85-89, 1991.
-
(1991)
The American Journal of Cardiology
, vol.68
, Issue.1
, pp. 85-89
-
-
Galderisi, M.1
Anderson, K.M.2
Wilson, P.W.F.3
Levy, D.4
-
5
-
-
79959251277
-
Potential role of nuclear factor κb in diabetic cardiomyopathy
-
O. Lorenzo, B. Picatoste, S. Ares-Carrasco, E. Ramírez, J. Egido, and J. Tuñón, "Potential role of nuclear factor κb in diabetic cardiomyopathy," Mediators of Inflammation, vol. 2011, Article ID 652097, 9 pages, 2011.
-
(2011)
Mediators of Inflammation
, vol.2011
-
-
Lorenzo, O.1
Picatoste, B.2
Ares-Carrasco, S.3
Ramírez, E.4
Egido, J.5
Tuñón, J.6
-
6
-
-
84953747754
-
Resveratrol ameliorates myocardial fibrosis by inhibiting ROS/ERK/TGF-β/periostin pathway in STZ-induced diabetic mice
-
H. Wu, G.-N. Li, J. Xie et al., "Resveratrol ameliorates myocardial fibrosis by inhibiting ROS/ERK/TGF-β/periostin pathway in STZ-induced diabetic mice," BMC Cardiovascular Disorders, vol. 16, no. 1, article 5, 2016.
-
(2016)
BMC Cardiovascular Disorders
, vol.16
, Issue.1
-
-
Wu, H.1
Li, G.-N.2
Xie, J.3
-
7
-
-
84894315676
-
Chronic resveratrol administration improves diabetic cardiomyopathy in part by reducing oxidative stress
-
M. Mohammadshahi, F. Haidari, and F. G. Soufi, "Chronic resveratrol administration improves diabetic cardiomyopathy in part by reducing oxidative stress," Cardiology Journal, vol. 21, no. 1, pp. 39-46, 2014.
-
(2014)
Cardiology Journal
, vol.21
, Issue.1
, pp. 39-46
-
-
Mohammadshahi, M.1
Haidari, F.2
Soufi, F.G.3
-
8
-
-
84902345161
-
Impairments in mitochondrial palmitoyl-CoA respiratory kinetics that precede development of diabetic cardiomyopathy are prevented by resveratrol in ZDF rats
-
M.-S. Beaudoin, C. G. R. Perry, A. M. Arkell et al., "Impairments in mitochondrial palmitoyl-CoA respiratory kinetics that precede development of diabetic cardiomyopathy are prevented by resveratrol in ZDF rats," Journal of Physiology, vol. 592, no. 12, pp. 2519-2533, 2014.
-
(2014)
Journal of Physiology
, vol.592
, Issue.12
, pp. 2519-2533
-
-
Beaudoin, M.-S.1
Perry, C.G.R.2
Arkell, A.M.3
-
9
-
-
77749254621
-
Resveratrol, an activator of SIRT1, upregulates sarcoplasmic calcium ATPase and improves cardiac function in diabetic cardiomyopathy
-
M. Sulaiman, M. J. Matta, N. R. Sunderesan, M. P. Gupta, M. Periasamy, and M. Gupta, "Resveratrol, an activator of SIRT1, upregulates sarcoplasmic calcium ATPase and improves cardiac function in diabetic cardiomyopathy," American Journal of Physiology-Heart and Circulatory Physiology, vol. 298, no. 3, pp. H833-H843, 2010.
-
(2010)
American Journal of Physiology-Heart and Circulatory Physiology
, vol.298
, Issue.3
, pp. H833-H843
-
-
Sulaiman, M.1
Matta, M.J.2
Sunderesan, N.R.3
Gupta, M.P.4
Periasamy, M.5
Gupta, M.6
-
10
-
-
84863085663
-
Resveratrol treatment reduces cardiac progenitor cell dysfunction and prevents morpho-functional ventricular remodeling in type-1 diabetic rats
-
F. Delucchi, R. Berni, C. Frati et al., "Resveratrol treatment reduces cardiac progenitor cell dysfunction and prevents morpho-functional ventricular remodeling in type-1 diabetic rats," PLoS ONE, vol. 7, no. 6, Article ID e39836, 2012.
-
(2012)
PLoS ONE
, vol.7
, Issue.6
-
-
Delucchi, F.1
Berni, R.2
Frati, C.3
-
11
-
-
74449089862
-
Targeting HMGB1 in inflammation
-
H. Yang and K. J. Tracey, "Targeting HMGB1 in inflammation," Biochimica et Biophysica Acta-Gene Regulatory Mechanisms, vol. 1799, no. 1-2, pp. 149-156, 2010.
-
(2010)
Biochimica et Biophysica Acta-Gene Regulatory Mechanisms
, vol.1799
, Issue.1-2
, pp. 149-156
-
-
Yang, H.1
Tracey, K.J.2
-
12
-
-
78649333667
-
HMGB1: The missing link between diabetes mellitus and heart failure
-
H. C. Volz, C. Seidel, D. Laohachewin et al., "HMGB1: the missing link between diabetes mellitus and heart failure," Basic Research in Cardiology, vol. 105, no. 6, pp. 805-820, 2010.
-
(2010)
Basic Research in Cardiology
, vol.105
, Issue.6
, pp. 805-820
-
-
Volz, H.C.1
Seidel, C.2
Laohachewin, D.3
-
13
-
-
77449142258
-
Hyperglycemia-induced reactive oxygen species increase expression of the receptor for advanced glycation end products (RAGE) and RAGE ligands
-
D. Yao and M. Brownlee, "Hyperglycemia-induced reactive oxygen species increase expression of the receptor for advanced glycation end products (RAGE) and RAGE ligands," Diabetes, vol. 59, no. 1, pp. 249-255, 2010.
-
(2010)
Diabetes
, vol.59
, Issue.1
, pp. 249-255
-
-
Yao, D.1
Brownlee, M.2
-
14
-
-
80051589315
-
Niaspan reduces high-mobility group box 1/receptor for advanced glycation endproducts after stroke in type-1 diabetic rats
-
X. Ye, M. Chopp, X. Liu et al., "Niaspan reduces high-mobility group box 1/receptor for advanced glycation endproducts after stroke in type-1 diabetic rats," Neuroscience, vol. 190, pp. 339-345, 2011.
-
(2011)
Neuroscience
, vol.190
, pp. 339-345
-
-
Ye, X.1
Chopp, M.2
Liu, X.3
-
15
-
-
79953141815
-
Increased serum high-mobility group box-1 and cleaved receptor for advanced glycation endproducts levels and decreased endogenous secretory receptor for advanced glycation endproducts levels in diabetic and non-diabetic patients with heart failure
-
L. J. Wang, L. Lu, F. R. Zhang, Q. J. Chen, R. De Caterina, and W. F. Shen, "Increased serum high-mobility group box-1 and cleaved receptor for advanced glycation endproducts levels and decreased endogenous secretory receptor for advanced glycation endproducts levels in diabetic and non-diabetic patients with heart failure," European Journal of Heart Failure, vol. 13, no. 4, pp. 440-449, 2011.
-
(2011)
European Journal of Heart Failure
, vol.13
, Issue.4
, pp. 440-449
-
-
Wang, L.J.1
Lu, L.2
Zhang, F.R.3
Chen, Q.J.4
De Caterina, R.5
Shen, W.F.6
-
16
-
-
84894101125
-
Inhibition of highmobility group box 1 improves myocardial fibrosis and dysfunction in diabetic cardiomyopathy
-
W.-K. Wang, B. Wang, Q.-H. Lu et al., "Inhibition of highmobility group box 1 improves myocardial fibrosis and dysfunction in diabetic cardiomyopathy," International Journal of Cardiology, vol. 172, no. 1, pp. 202-212, 2014.
-
(2014)
International Journal of Cardiology
, vol.172
, Issue.1
, pp. 202-212
-
-
Wang, W.-K.1
Wang, B.2
Lu, Q.-H.3
-
17
-
-
37149008763
-
The role of the receptor for advanced glycation end-products in lung fibrosis
-
M. He, H. Kubo, K. Ishizawa et al., "The role of the receptor for advanced glycation end-products in lung fibrosis," American Journal of Physiology-Lung Cellular and Molecular Physiology, vol. 293, no. 6, pp. L1427-L1436, 2007.
-
(2007)
American Journal of Physiology-Lung Cellular and Molecular Physiology
, vol.293
, Issue.6
, pp. L1427-L1436
-
-
He, M.1
Kubo, H.2
Ishizawa, K.3
-
18
-
-
84864125510
-
Protective effects of curcumin against hepatic fibrosis induced by carbon tetrachloride: Modulation of high-mobility group box 1, toll-like receptor 4 and 2 expression
-
C.-T. Tu, Q.-Y. Yao, B.-L. Xu, J.-Y. Wang, C.-H. Zhou, and S.-C. Zhang, "Protective effects of curcumin against hepatic fibrosis induced by carbon tetrachloride: modulation of high-mobility group box 1, Toll-like receptor 4 and 2 expression," Food and Chemical Toxicology, vol. 50, no. 9, pp. 3343-3351, 2012.
-
(2012)
Food and Chemical Toxicology
, vol.50
, Issue.9
, pp. 3343-3351
-
-
Tu, C.-T.1
Yao, Q.-Y.2
Xu, B.-L.3
Wang, J.-Y.4
Zhou, C.-H.5
Zhang, S.-C.6
-
19
-
-
84865631414
-
Inhibition of highmobility group box 1 protein (HMGB1) enhances bacterial clearance and protects against pseudomonas aeruginosa pneumonia in cystic fibrosis
-
M. Entezari, D. J. Weiss, R. Sitapara et al., "Inhibition of highmobility group box 1 protein (HMGB1) enhances bacterial clearance and protects against Pseudomonas aeruginosa pneumonia in cystic fibrosis," Molecular Medicine, vol. 18, no. 1, pp. 477-485, 2012.
-
(2012)
Molecular Medicine
, vol.18
, Issue.1
, pp. 477-485
-
-
Entezari, M.1
Weiss, D.J.2
Sitapara, R.3
-
20
-
-
77954268849
-
2+/calmodulin-dependent protein kinase II activity in diabetic mouse retina
-
2+/calmodulin-dependent protein kinase II activity in diabetic mouse retina," Diabetes, vol. 59, no. 7, pp. 1825-1835, 2010.
-
(2010)
Diabetes
, vol.59
, Issue.7
, pp. 1825-1835
-
-
Kim, Y.-H.1
Kim, Y.-S.2
Kang, S.-S.3
Cho, G.-J.4
Choi, W.-S.5
-
21
-
-
77958026411
-
Resveratrol improves left ventricular diastolic relaxation in type 2 diabetes by inhibiting oxidative/nitrative stress: In vivo demonstration with magnetic resonance imaging
-
H. Zhang, B. Morgan, B. J. Potter et al., "Resveratrol improves left ventricular diastolic relaxation in type 2 diabetes by inhibiting oxidative/nitrative stress: in vivo demonstration with magnetic resonance imaging," American Journal of Physiology-Heart and Circulatory Physiology, vol. 299, no. 4, pp. H985-H994, 2010.
-
(2010)
American Journal of Physiology-Heart and Circulatory Physiology
, vol.299
, Issue.4
, pp. H985-H994
-
-
Zhang, H.1
Morgan, B.2
Potter, B.J.3
-
22
-
-
84954304927
-
Diabetes-induced oxidative stress in endothelial progenitor cells may be sustained by a positive feedback loop involving high mobility group box-1
-
H. Wu, R. Li, Z.-H. Wei et al., "Diabetes-induced oxidative stress in endothelial progenitor cells may be sustained by a positive feedback loop involving high mobility group box-1," Oxidative Medicine and Cellular Longevity, vol. 2016, Article ID 1943918, 9 pages, 2016.
-
(2016)
Oxidative Medicine and Cellular Longevity
, vol.2016
-
-
Wu, H.1
Li, R.2
Wei, Z.-H.3
-
23
-
-
79956218866
-
The role of highmobility group box-1 protein in the development of diabetic nephropathy
-
J. Kim, E. Sohn, C.-S. Kim, K. Jo, and J. S. Kim, "The role of highmobility group box-1 protein in the development of diabetic nephropathy," American Journal of Nephrology, vol. 33, no. 6, pp. 524-529, 2011.
-
(2011)
American Journal of Nephrology
, vol.33
, Issue.6
, pp. 524-529
-
-
Kim, J.1
Sohn, E.2
Kim, C.-S.3
Jo, K.4
Kim, J.S.5
-
24
-
-
52749084063
-
Receptor for advanced glycation end products (RAGE) deficiency attenuates the development of atherosclerosis in diabetes
-
A. Soro-Paavonen, A. M. D. Watson, J. Li et al., "Receptor for advanced glycation end products (RAGE) deficiency attenuates the development of atherosclerosis in diabetes," Diabetes, vol. 57, no. 9, pp. 2461-2469, 2008.
-
(2008)
Diabetes
, vol.57
, Issue.9
, pp. 2461-2469
-
-
Soro-Paavonen, A.1
Watson, A.M.D.2
Li, J.3
-
25
-
-
84899596065
-
Resveratrol reduces the proinflammatory effects and lipopolysaccharide-induced expression of HMGB1 and TLR4 in RAW264.7 cells
-
Y. Yang, S. Li, Q. Yang et al., "Resveratrol reduces the proinflammatory effects and lipopolysaccharide-induced expression of HMGB1 and TLR4 in RAW264.7 cells," Cellular Physiology and Biochemistry, vol. 33, no. 5, pp. 1283-1292, 2014.
-
(2014)
Cellular Physiology and Biochemistry
, vol.33
, Issue.5
, pp. 1283-1292
-
-
Yang, Y.1
Li, S.2
Yang, Q.3
-
26
-
-
84864563524
-
Different gene expression in human heart tissue and progenitor cells from control and diabetic subjects: Relevance to the pathogenesis of human diabetic cardiomyopathy
-
E. de Cillis, A. Leonardini, L. Laviola, F. Giorgino, L. D. L. Tupputi Schinosa, and A. S. Bortone, "Different gene expression in human heart tissue and progenitor cells from control and diabetic subjects: relevance to the pathogenesis of human diabetic cardiomyopathy," Surgical Technology International, vol. 19, pp. 165-174, 2010.
-
(2010)
Surgical Technology International
, vol.19
, pp. 165-174
-
-
De Cillis, E.1
Leonardini, A.2
Laviola, L.3
Giorgino, F.4
Tupputi Schinosa, L.D.L.5
Bortone, A.S.6
-
27
-
-
33847775295
-
Hydrogen peroxide stimulates macrophages and monocytes to actively release HMGB1
-
D. Tang, Y. Shi, R. Kang et al., "Hydrogen peroxide stimulates macrophages and monocytes to actively release HMGB1," Journal of Leukocyte Biology, vol. 81, no. 3, pp. 741-747, 2007.
-
(2007)
Journal of Leukocyte Biology
, vol.81
, Issue.3
, pp. 741-747
-
-
Tang, D.1
Shi, Y.2
Kang, R.3
-
28
-
-
34248228729
-
Hemorrhagic shock induces NAD(P)H oxidase activation in neutrophils: Role of HMGB1-TLR4 signaling
-
J. Fan, Y. Li, R. M. Levy et al., "Hemorrhagic shock induces NAD(P)H oxidase activation in neutrophils: role of HMGB1-TLR4 signaling," Journal of Immunology, vol. 178, no. 10, pp. 6573-6580, 2007.
-
(2007)
Journal of Immunology
, vol.178
, Issue.10
, pp. 6573-6580
-
-
Fan, J.1
Li, Y.2
Levy, R.M.3
-
29
-
-
84866396383
-
HMGB1 in renal ischemic injury
-
M. M. Rabadi, T. Ghaly, M. S. Goligorksy, and B. B. Ratliff, "HMGB1 in renal ischemic injury," American Journal of Physiology-Renal Physiology, vol. 303, no. 6, pp. F873-F885, 2012.
-
(2012)
American Journal of Physiology-Renal Physiology
, vol.303
, Issue.6
, pp. F873-F885
-
-
Rabadi, M.M.1
Ghaly, T.2
Goligorksy, M.S.3
Ratliff, B.B.4
-
30
-
-
84869121923
-
Quercetin treatment ameliorates inflammation and fibrosis in mice with nonalcoholic steatohepatitis
-
E. Marcolin, B. San-Miguel, D. Vallejo et al., "Quercetin treatment ameliorates inflammation and fibrosis in mice with nonalcoholic steatohepatitis," Journal of Nutrition, vol. 142, no. 10, pp. 1821-1828, 2012.
-
(2012)
Journal of Nutrition
, vol.142
, Issue.10
, pp. 1821-1828
-
-
Marcolin, E.1
San-Miguel, B.2
Vallejo, D.3
-
31
-
-
84898846870
-
Calcitriol modulates receptor for advanced glycation end products (RAGE) in diabetic hearts
-
T.-W. Lee, Y.-H. Kao, T.-I. Lee, C.-J. Chang, G.-S. Lien, and Y.-J. Chen, "Calcitriol modulates receptor for advanced glycation end products (RAGE) in diabetic hearts," International Journal of Cardiology, vol. 173, no. 2, pp. 236-241, 2014.
-
(2014)
International Journal of Cardiology
, vol.173
, Issue.2
, pp. 236-241
-
-
Lee, T.-W.1
Kao, Y.-H.2
Lee, T.-I.3
Chang, C.-J.4
Lien, G.-S.5
Chen, Y.-J.6
-
32
-
-
84863116202
-
Cannabinoid 1 receptor promotes cardiac dysfunction, oxidative stress, inflammation, and fibrosis in diabetic cardiomyopathy
-
M. Rajesh, S. Bátkai, M. Kechrid et al., "Cannabinoid 1 receptor promotes cardiac dysfunction, oxidative stress, inflammation, and fibrosis in diabetic cardiomyopathy," Diabetes, vol. 61, no. 3, pp. 716-727, 2012.
-
(2012)
Diabetes
, vol.61
, Issue.3
, pp. 716-727
-
-
Rajesh, M.1
Bátkai, S.2
Kechrid, M.3
-
33
-
-
78650043221
-
Prevention of hyperglycemiainduced myocardial apoptosis by gene silencing of toll-like receptor-4
-
Y. Zhang, T. Peng, H. Zhu et al., "Prevention of hyperglycemiainduced myocardial apoptosis by gene silencing of Toll-like receptor-4," Journal of Translational Medicine, vol. 8, article 133, 2010.
-
(2010)
Journal of Translational Medicine
, vol.8
-
-
Zhang, Y.1
Peng, T.2
Zhu, H.3
-
34
-
-
84938490692
-
Matrine pretreatment improves cardiac function in rats with diabetic cardiomyopathy via suppressing ROS/TLR-4 signaling pathway
-
Z.-W. Liu, J.-K. Wang, C. Qiu et al., "Matrine Pretreatment improves cardiac function in rats with diabetic cardiomyopathy via suppressing ROS/TLR-4 signaling pathway," Acta Pharmacologica Sinica, vol. 36, no. 3, pp. 323-333, 2015.
-
(2015)
Acta Pharmacologica Sinica
, vol.36
, Issue.3
, pp. 323-333
-
-
Liu, Z.-W.1
Wang, J.-K.2
Qiu, C.3
-
35
-
-
77952375084
-
Resveratrol ameliorates vasculopathy in STZ-induced diabetic rats: Role of AGE-RAGE signalling
-
Y.-H. Jing, K.-H. Chen, S.-H. Yang, P.-C. Kuo, and J.-K. Chen, "Resveratrol ameliorates vasculopathy in STZ-induced diabetic rats: role of AGE-RAGE signalling," Diabetes/Metabolism Research and Reviews, vol. 26, no. 3, pp. 212-222, 2010.
-
(2010)
Diabetes/Metabolism Research and Reviews
, vol.26
, Issue.3
, pp. 212-222
-
-
Jing, Y.-H.1
Chen, K.-H.2
Yang, S.-H.3
Kuo, P.-C.4
Chen, J.-K.5
-
36
-
-
23844478190
-
Specific inhibition of MyD88-independent signaling pathways of TLR3 and TLR4 by resveratrol: Molecular targets are TBK1 and RIP1 in TRIF complex
-
H. S. Youn, J. Y. Lee, K. A. Fitzgerald, H. A. Young, S. Akira, and D. H. Hwang, "Specific inhibition of MyD88-independent signaling pathways of TLR3 and TLR4 by resveratrol: molecular targets are TBK1 and RIP1 in TRIF complex," The Journal of Immunology, vol. 175, no. 5, pp. 3339-3346, 2005.
-
(2005)
The Journal of Immunology
, vol.175
, Issue.5
, pp. 3339-3346
-
-
Youn, H.S.1
Lee, J.Y.2
Fitzgerald, K.A.3
Young, H.A.4
Akira, S.5
Hwang, D.H.6
-
37
-
-
84900428277
-
High glucose induces smad activation via the transcriptional coregulator p300 and contributes to cardiac fibrosis and hypertrophy
-
A. Bugyei-Twum, A. Advani, S. L. Advani et al., "High glucose induces Smad activation via the transcriptional coregulator p300 and contributes to cardiac fibrosis and hypertrophy," Cardiovascular Diabetology, vol. 13, article 89, 2014.
-
(2014)
Cardiovascular Diabetology
, vol.13
-
-
Bugyei-Twum, A.1
Advani, A.2
Advani, S.L.3
-
38
-
-
84908233904
-
Up-regulated HMGB1 in EAM directly led to collagen deposition by a PKCβ/Erk1/2-dependent pathway: Cardiac fibroblast/myofibroblast might be another source of HMGB1
-
Z. Su, J. Yin, T. Wang et al., "Up-regulated HMGB1 in EAM directly led to collagen deposition by a PKCβ/Erk1/2-dependent pathway: cardiac fibroblast/myofibroblast might be another source of HMGB1," Journal of Cellular and Molecular Medicine, vol. 18, no. 9, pp. 1740-1751, 2014.
-
(2014)
Journal of Cellular and Molecular Medicine
, vol.18
, Issue.9
, pp. 1740-1751
-
-
Su, Z.1
Yin, J.2
Wang, T.3
-
39
-
-
85001122938
-
Necrotic myocardial cells release damage-associatedmolecular patterns that provoke fibroblast activation in vitro and trigger myocardial inflammation and fibrosis in vivo
-
W. Zhang, K. J. Lavine, S. Epelman et al., "Necrotic myocardial cells release damage-associatedmolecular patterns that provoke fibroblast activation in vitro and trigger myocardial inflammation and fibrosis in vivo," Journal of the American Heart Association, vol. 4, no. 6, Article ID e001993, 2015.
-
(2015)
Journal of the American Heart Association
, vol.4
, Issue.6
-
-
Zhang, W.1
Lavine, K.J.2
Epelman, S.3
-
40
-
-
51649111202
-
High-mobility group box-1 in ischemia-reperfusion injury of the heart
-
M. Andrassy, H. C. Volz, J. C. Igwe et al., "High-mobility group box-1 in ischemia-reperfusion injury of the heart," Circulation, vol. 117, no. 25, pp. 3216-3226, 2008.
-
(2008)
Circulation
, vol.117
, Issue.25
, pp. 3216-3226
-
-
Andrassy, M.1
Volz, H.C.2
Igwe, J.C.3
-
41
-
-
84881463486
-
-/- mice by downregulating the HMGB1-RAGE axis
-
-/- mice by downregulating the HMGB1-RAGE axis," Acta Pharmacologica Sinica, vol. 34, no. 6, pp. 830-836, 2013.
-
(2013)
Acta Pharmacologica Sinica
, vol.34
, Issue.6
, pp. 830-836
-
-
Liu, M.1
Yu, Y.2
Jiang, H.3
-
42
-
-
84255163024
-
Receptor for AGE (RAGE): Signaling mechanisms in the pathogenesis of diabetes and its complications
-
R. Ramasamy, S. F. Yan, and A. M. Schmidt, "Receptor for AGE (RAGE): signaling mechanisms in the pathogenesis of diabetes and its complications," Annals of the New York Academy of Sciences, vol. 1243, no. 1, pp. 88-102, 2011.
-
(2011)
Annals of the New York Academy of Sciences
, vol.1243
, Issue.1
, pp. 88-102
-
-
Ramasamy, R.1
Yan, S.F.2
Schmidt, A.M.3
-
43
-
-
77951545792
-
The role of HMGB1/RAGE in inflammatory cardiomyopathy
-
H. C. Volz, Z. Kaya, H. A. Katus, and M. Andrassy, "The role of HMGB1/RAGE in inflammatory cardiomyopathy," Seminars in Thrombosis and Hemostasis, vol. 36, no. 2, pp. 185-194, 2010.
-
(2010)
Seminars in Thrombosis and Hemostasis
, vol.36
, Issue.2
, pp. 185-194
-
-
Volz, H.C.1
Kaya, Z.2
Katus, H.A.3
Andrassy, M.4
-
44
-
-
84928784665
-
Dioscin ameliorates cerebral ischemia/reperfusion injury through the downregulation of TLR4 signaling via HMGB-1 inhibition
-
X. Tao, X. Sun, L. Yin et al., "Dioscin ameliorates cerebral ischemia/reperfusion injury through the downregulation of TLR4 signaling via HMGB-1 inhibition," Free Radical Biology and Medicine, vol. 84, Article ID 12343, pp. 103-115, 2015.
-
(2015)
Free Radical Biology and Medicine
, vol.84
, pp. 103-115
-
-
Tao, X.1
Sun, X.2
Yin, L.3
-
45
-
-
84907833448
-
The role of TLR2 and 4-mediated inflammatory pathways in endothelial cells exposed to high glucose
-
H. Mudaliar, C. Pollock, J. Ma, H. Wu, S. Chadban, and U. Panchapakesan, "The role of TLR2 and 4-mediated inflammatory pathways in endothelial cells exposed to high glucose," PLoS ONE, vol. 9, no. 10, article e108844, 2014.
-
(2014)
PLoS ONE
, vol.9
, Issue.10
-
-
Mudaliar, H.1
Pollock, C.2
Ma, J.3
Wu, H.4
Chadban, S.5
Panchapakesan, U.6
-
46
-
-
84876803947
-
TRAF6 is functional in inhibition of TLR4-mediated NF-κB activation by resveratrol
-
P. B. Jakus, N. Kalman, C. Antus et al., "TRAF6 is functional in inhibition of TLR4-mediated NF-κB activation by resveratrol," Journal of Nutritional Biochemistry, vol. 24, no. 5, pp. 819-823, 2013.
-
(2013)
Journal of Nutritional Biochemistry
, vol.24
, Issue.5
, pp. 819-823
-
-
Jakus, P.B.1
Kalman, N.2
Antus, C.3
-
47
-
-
79955675928
-
A resveratrol and polyphenol preparation suppresses oxidative and inflammatory stress response to a high-fat, high-carbohydrate meal
-
H. Ghanim, C. L. Sia, K. Korzeniewski et al., "A resveratrol and polyphenol preparation suppresses oxidative and inflammatory stress response to a high-fat, high-carbohydrate meal," The Journal of Clinical Endocrinology & Metabolism, vol. 96, no. 5, pp. 1409-1414, 2011.
-
(2011)
The Journal of Clinical Endocrinology & Metabolism
, vol.96
, Issue.5
, pp. 1409-1414
-
-
Ghanim, H.1
Sia, C.L.2
Korzeniewski, K.3
-
48
-
-
84880265121
-
Cerebroprotective effect of resveratrol through antioxidant and anti-inflammatory effects in diabetic rats
-
O. Prabhakar, "Cerebroprotective effect of resveratrol through antioxidant and anti-inflammatory effects in diabetic rats," Naunyn-Schmiedeberg's Archives of Pharmacology, vol. 386, no. 8, pp. 705-710, 2013.
-
(2013)
Naunyn-Schmiedeberg's Archives of Pharmacology
, vol.386
, Issue.8
, pp. 705-710
-
-
Prabhakar, O.1
-
49
-
-
84961119684
-
Resveratrol ameliorates diabetesinduced cardiac dysfunction through AT1R-ERK/p38 MAPK signaling pathway
-
Y. Gao, L. Kang, C. Li et al., "Resveratrol ameliorates diabetesinduced cardiac dysfunction through AT1R-ERK/p38 MAPK signaling pathway," Cardiovascular Toxicology, vol. 16, no. 2, pp. 130-137, 2016.
-
(2016)
Cardiovascular Toxicology
, vol.16
, Issue.2
, pp. 130-137
-
-
Gao, Y.1
Kang, L.2
Li, C.3
-
50
-
-
7744222474
-
Bacterial endotoxin stimulates macrophages to release HMGB1 partly through CD14-and TNF-dependentmechanisms
-
G. Chen, J. Li, M. Ochani et al., "Bacterial endotoxin stimulates macrophages to release HMGB1 partly through CD14-and TNF-dependentmechanisms," Journal of Leukocyte Biology, vol. 76, no. 5, pp. 994-1001, 2004.
-
(2004)
Journal of Leukocyte Biology
, vol.76
, Issue.5
, pp. 994-1001
-
-
Chen, G.1
Li, J.2
Ochani, M.3
-
51
-
-
84908448181
-
HMGB1 in health and disease
-
R. Kang, R. Chen, Q. Zhang et al., "HMGB1 in health and disease," Molecular Aspects of Medicine, vol. 40, pp. 1-116, 1998.
-
(1998)
Molecular Aspects of Medicine
, vol.40
, pp. 1-116
-
-
Kang, R.1
Chen, R.2
Zhang, Q.3
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