Challenging the dogma of mitochondrial reactive oxygen species overproduction in diabetic kidney disease

Kidney International

Volumn 90, Issue 2, 2016, Pages 272-279

  Coughlan, Melinda T ^a,b   Sharma, Kumar ^c,d  

^a BAKER IDI HEART AND DIABETES INSTITUTE   (Australia)

^b MONASH UNIVERSITY   (Australia)

^c UNIVERSITY OF CALIFORNIA   (United States)

^d VA SAN DIEGO HEALTHCARE SYSTEM   (United States)

Author keywords

diabetic nephropathy; mitochondria; oxidative stress

Indexed keywords

REACTIVE OXYGEN METABOLITE;

DIABETIC NEPHROPATHY; DISEASE COURSE; DISORDERS OF MITOCHONDRIAL FUNCTIONS; ENDOPLASMIC RETICULUM; HUMAN; INFLAMMATION; INTRACELLULAR SIGNALING; KIDNEY FIBROSIS; MITOCHONDRIAL BIOGENESIS; MITOCHONDRION; NONHUMAN; OXIDATIVE PHOSPHORYLATION; PATHOGENESIS; PRIORITY JOURNAL; REVIEW; STEADY STATE; SYNTHESIS; ANIMAL; COMPLICATION; FIBROSIS; HYPERGLYCEMIA; KIDNEY; METABOLISM; OXIDATIVE STRESS; PATHOLOGY; SIGNAL TRANSDUCTION;

ANIMALS; DIABETIC NEPHROPATHIES; FIBROSIS; HUMANS; HYPERGLYCEMIA; KIDNEY; MITOCHONDRIA; OXIDATIVE STRESS; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION;

EID: 84992489196     PISSN: 00852538     EISSN: 15231755     Source Type: Journal    
DOI: 10.1016/j.kint.2016.02.043     Document Type: Review

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