Receptor interacting protein 3-mediated necroptosis promotes lipopolysaccharide-induced inflammation and acute respiratory distress syndrome in mice

PLoS ONE

Volumn 11, Issue 5, 2016, Pages

  Wang, Linlin ^a,b   Wang, Tingting ^a   Li, Haobo ^c   Liu, Qing ^a   Zhang, Zhongjun ^b   Xie, Wanli ^a   Feng, Yinglu ^a   Socorburam, Tumenjavkhlan ^a   Wu, Gui ^d   Xia, Zhengyuan ^c   Wu, Qingping ^a  

^a HUAZHONG UNIVERSITY OF SCIENCE AND TECHNOLOGY   (China)

^b SHENZHEN PEOPLE'S HOSPITAL   (China)

^c UNIVERSITY OF HONG KONG   (Hong Kong)

^d WUHAN GENERAL HOSPITAL OF GUANGZHOU COMMAND   (China)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE 3; CASPASE 8; INTERLEUKIN 10; INTERLEUKIN 1ALPHA; INTERLEUKIN 1BETA; INTERLEUKIN 6; LIPOPOLYSACCHARIDE; MIXED LINEAGE KINASE DOMAIN LIKE PROTEIN; MYELOPEROXIDASE; PHOSPHOTRANSFERASE; RECEPTOR INTERACTING PROTEIN 3; RECEPTOR PROTEIN; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG; X LINKED INHIBITOR OF APOPTOSIS; BIRC4 PROTEIN, MOUSE; INHIBITOR OF APOPTOSIS PROTEIN; PEROXIDASE; RECEPTOR INTERACTING PROTEIN SERINE THREONINE KINASE; RIPK3 PROTEIN, MOUSE;

ADULT RESPIRATORY DISTRESS SYNDROME; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; CYTOKINE RELEASE; DISEASE SEVERITY; ENZYME ACTIVITY; HYPOTHERMIA; LUNG PARENCHYMA; MALE; MOUSE; NECROPTOSIS; NEUTROPHIL COUNT; NONHUMAN; PROTEIN BLOOD LEVEL; PROTEIN CLEAVAGE; PROTEIN FUNCTION; SURVIVAL RATE; ANIMAL; BRONCHOALVEOLAR LAVAGE FLUID; C57BL MOUSE; CHEMICALLY INDUCED; DISEASE MODEL; DRUG EFFECTS; GENETICS; INFLAMMATION; KNOCKOUT MOUSE; LUNG; LUNG INJURY; METABOLISM; NECROSIS; NEUTROPHIL;

ANIMALS; APOPTOSIS; BRONCHOALVEOLAR LAVAGE FLUID; DISEASE MODELS, ANIMAL; INFLAMMATION; INHIBITOR OF APOPTOSIS PROTEINS; LIPOPOLYSACCHARIDES; LUNG; LUNG INJURY; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NECROSIS; NEUTROPHILS; PEROXIDASE; RECEPTOR-INTERACTING PROTEIN SERINE-THREONINE KINASES; RESPIRATORY DISTRESS SYNDROME, ADULT;

EID: 84973621787     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0155723     Document Type: Article

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