Dopaminergic neurotransmission dysfunction induced by amyloid-β transforms cortical long-term potentiation into long-term depression and produces memory impairment

Neurobiology of Aging

Volumn 41, Issue , 2016, Pages 187-199

  Moreno Castilla, Perla ^a   Rodriguez Duran, Luis F ^b,c   Guzman Ramos, Kioko ^a,c   Barcenas Femat, Alejandro ^a   Escobar, Martha L ^b   Bermudez Rattoni, Federico ^a  

^a UNIVERSIDAD NACIONAL AUTÓNOMA DE MÉXICO   (Mexico)

^b UNIVERSIDAD NACIONAL AUTÓNOMA DE MÉXICO   (Mexico)

^c Metroipolitan Autonomous University Lerma   (Mexico)

Author keywords

Alzheimer's disease; Amyloid beta; Dopamine; Long term depression; Long term potentiation; Recognition memory impairment

Indexed keywords

AMYLOID BETA PROTEIN; DOPAMINE; NORADRENALIN; OLIGOMER;

ALZHEIMER DISEASE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BASOLATERAL AMYGDALA; BRAIN CORTEX; BRAIN DEPTH STIMULATION; CONTROLLED STUDY; DISEASE ACTIVITY; DISEASE ASSOCIATION; DISEASE COURSE; DOPAMINE BRAIN LEVEL; DOPAMINERGIC NEUROTRANSMISSION DYSFUNCTION; DOPAMINERGIC TRANSMISSION; IN VIVO STUDY; INSULA; LONG TERM DEPRESSION; LONG TERM POTENTIATION; MALE; MEMORY DISORDER; MOUSE; NERVE CELL PLASTICITY; NEUROLOGIC DISEASE; NONHUMAN; PATHOPHYSIOLOGY; PRIORITY JOURNAL; SYMPTOM; TRANSGENIC MOUSE; ANIMAL; CHEMICALLY INDUCED; DISEASE MODEL; DOPAMINERGIC NERVE CELL; DRUG EFFECTS; METABOLISM; PHYSIOLOGY; PSYCHOLOGY; SYNAPTIC TRANSMISSION;

ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; ANIMALS; CEREBRAL CORTEX; DISEASE MODELS, ANIMAL; DOPAMINE; DOPAMINERGIC NEURONS; LONG-TERM POTENTIATION; LONG-TERM SYNAPTIC DEPRESSION; MALE; MEMORY DISORDERS; MICE; MICE, TRANSGENIC; NEURONAL PLASTICITY; SYNAPTIC TRANSMISSION;

EID: 84961784169     PISSN: 01974580     EISSN: 15581497     Source Type: Journal    
DOI: 10.1016/j.neurobiolaging.2016.02.021     Document Type: Article

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