Aberrant sodium influx causes cardiomyopathy and atrial fibrillation in mice

Journal of Clinical Investigation

Volumn 126, Issue 1, 2016, Pages 112-122

  Wan, Elaine ^a   Abrams, Jeffrey ^a   Weinberg, Richard L ^a,b   Katchman, Alexander N ^a   Bayne, Joseph ^a   Zakharov, Sergey I ^a   Yang, Lin ^a   Morrow, John P ^a   Garan, Hasan ^a   Marx, Steven O ^a  

^a New York Presbyterian Columbia University Medical Center   (United States)

^b UNIVERSITY OF MICHIGAN MEDICAL SCHOOL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

SODIUM CALCIUM EXCHANGE PROTEIN; SODIUM CHANNEL NAV1.5; CALCIUM; SCN5A PROTEIN, MOUSE; SODIUM;

ACTION POTENTIAL DURATION; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ATRIAL FIBRILLATION; BINDING SITE; CARDIOMYOPATHY; CELL DAMAGE; CONTROLLED STUDY; ECHOCARDIOGRAPHY; FOURIER TRANSFORMATION; GENE MUTATION; HEART ATRIUM ARRHYTHMIA; HEART LEFT VENTRICLE EJECTION FRACTION; HEART MUSCLE FIBROSIS; HEART VENTRICLE ARRHYTHMIA; MOUSE; MUSCLE FIBRIL; NONHUMAN; OPERATOR GENE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PROCESSING; SODIUM CURRENT; SODIUM TRANSPORT; ANIMAL; CARDIOMYOPATHIES; ELECTROCARDIOGRAPHY; METABOLISM; PHYSIOLOGY;

ANIMALS; ATRIAL FIBRILLATION; CALCIUM; CARDIOMYOPATHIES; ELECTROCARDIOGRAPHY; MICE; NAV1.5 VOLTAGE-GATED SODIUM CHANNEL; SODIUM;

EID: 84956668087     PISSN: 00219738     EISSN: 15588238     Source Type: Journal    
DOI: 10.1172/JCI84669     Document Type: Article

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