Divergent biophysical defects caused by mutant sodium channels in dilated cardiomyopathy with arrhythmia

Circulation Research

Volumn 102, Issue 3, 2008, Pages 364-371

  Nguyen, Thao P ^a,d   Wang, Dao W ^b   Rhodes, Thomas H ^b   George Jr , Alfred L ^b,c  

^a DEPARTMENT OF PATHOLOGY   (United States)

^b VANDERBILT UNIVERSITY   (United States)

^c VANDERBILT UNIVERSITY SCHOOL OF MEDICINE   (United States)

^d UCLA MEDICAL CENTER   (United States)

Author keywords

Arrhythmia; Cardiomyopathy; Genetics; Ion channels

Indexed keywords

ION CHANNEL; SODIUM CHANNEL; VOLTAGE GATED SODIUM CHANNEL;

ARTICLE; CARDIOMYOPATHY; CONGESTIVE CARDIOMYOPATHY; CONTROLLED STUDY; DATA ANALYSIS; ELECTROPHYSIOLOGY; GENE MUTATION; HEART ARRHYTHMIA; HOMEOSTASIS; HUMAN; HUMAN CELL; HYPERPOLARIZATION; MUSCLE CELL; MUTAGENESIS; PRIORITY JOURNAL; SCN5A GENE; SINUS BRADYCARDIA; STEADY STATE;

AMINO ACID SUBSTITUTION; ARRHYTHMIAS, CARDIAC; CALCIUM; CARDIOMYOPATHY, DILATED; CELL LINE; GENETIC DISEASES, INBORN; HOMEOSTASIS; HUMANS; ION TRANSPORT; MUSCLE PROTEINS; MUTATION, MISSENSE; MYOCYTES, CARDIAC; PATCH-CLAMP TECHNIQUES; SODIUM; SODIUM CHANNELS;

EID: 39449138242     PISSN: 00097330     EISSN: None     Source Type: Journal    
DOI: 10.1161/CIRCRESAHA.107.164673     Document Type: Article

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