Cardiolipin remodeling by TAZ/tafazzin is selectively required for the initiation of mitophagy

Autophagy

Volumn 11, Issue 4, 2015, Pages 643-652

  Hsu, Paul ^a   Liu, Xiaolei ^a   Zhang, Jun ^a   Wang, Hong Gang ^a   Ye, Ji Ming ^b   Shi, Yuguang ^a  

^a PENN STATE COLLEGE OF MEDICINE   (United States)

^b RMIT UNIVERSITY   (Australia)

Author keywords

Autophagy; Barth syndrome; Cardiolipin; Mitochondrial dysfunction; Mitophagosome; Mitophagy; Tafazzin

Indexed keywords

CARDIOLIPIN; PEPTIDES AND PROTEINS; TAFAZZIN; UNCLASSIFIED DRUG; TAZ PROTEIN, HUMAN; TAZ PROTEIN, MOUSE; TRANSCRIPTION FACTOR;

ANIMAL CELL; ARTICLE; BIOGENESIS; CONTROLLED STUDY; EMBRYO; MITOCHONDRION; MITOPHAGY; MOUSE; NONHUMAN; OXIDATIVE PHOSPHORYLATION; OXIDATIVE STRESS; QUALITY CONTROL; ANIMAL; AUTOPHAGY; CONGESTIVE CARDIOMYOPATHY; DEFICIENCY; GENETICS; HUMAN; METABOLISM; MUTATION; PATHOLOGY; PHYSIOLOGY;

ANIMALS; AUTOPHAGY; CARDIOLIPINS; CARDIOMYOPATHY, DILATED; HUMANS; MICE; MITOCHONDRIA; MITOCHONDRIAL DEGRADATION; MUTATION; TRANSCRIPTION FACTORS;

EID: 84942573346     PISSN: 15548627     EISSN: 15548635     Source Type: Journal    
DOI: 10.1080/15548627.2015.1023984     Document Type: Article

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