All-trans-retinoic acid reduces BACE1 expression under inflammatory conditions via modulation of nuclear factor κB (NFκB) signaling

Journal of Biological Chemistry

Volumn 290, Issue 37, 2015, Pages 22532-22542

  Wang, Ruishan ^a   Chen, Shaoya ^a   Liu, Yingchun ^a   Diao, Shiyong ^a   Xue, Yueqiang ^a   You, Xiaoqing ^b   Park, Edwards A ^a,c   Liao, Francesca Fang ^a  

^a UNIVERSITY OF TENNESSEE HEALTH SCIENCE CENTER   (United States)

^b VETERANS AFFAIRS MEDICAL CENTER   (United States)

^c FUJIAN MEDICAL UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

GLYCOPROTEINS; MAMMALS; MODULATION; PROTEINS;

ALL-TRANS RETINOIC ACIDS; AMYLOID PRECURSOR PROTEINS; CARDIO-VASCULAR DISEASE; INFLAMMATORY CONDITIONS; INFLAMMATORY RESPONSE; NUCLEAR FACTOR KAPPAB; NUCLEAR TRANSLOCATIONS; THERAPEUTIC POTENTIALS;

GENE EXPRESSION;

AMYLOID BETA PROTEIN; AMYLOID PRECURSOR PROTEIN; BETA SECRETASE 1; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INSULIN; RETINOIC ACID; ASPARTIC PROTEINASE; BACE1 PROTEIN, MOUSE; FAT INTAKE; SECRETASE;

ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ARTICLE; CARDIOVASCULAR DISEASE; CONTROLLED STUDY; DISEASE COURSE; DOWN REGULATION; DRUG EFFICACY; ENZYME ACTIVITY; GENE EXPRESSION; GENE MUTATION; GENETIC TRANSCRIPTION; INFLAMMATION; INSULIN RESISTANCE; LIPID DIET; MODULATION; MOUSE; NERVOUS SYSTEM INFLAMMATION; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN CLEAVAGE; PROTEIN EXPRESSION; RISK FACTOR; UPREGULATION; ANIMAL; BIOSYNTHESIS; BRAIN; DISEASE MODEL; DRUG EFFECTS; FAT INTAKE; GENE EXPRESSION REGULATION; GENETICS; METABOLISM; PATHOLOGY; PHARMACOLOGY; SIGNAL TRANSDUCTION; TRANSGENIC MOUSE;

ALZHEIMER DISEASE; AMYLOID PRECURSOR PROTEIN SECRETASES; ANIMALS; ASPARTIC ACID ENDOPEPTIDASES; BRAIN; DIETARY FATS; DISEASE MODELS, ANIMAL; GENE EXPRESSION REGULATION, ENZYMOLOGIC; MICE; MICE, TRANSGENIC; NF-KAPPA B; SIGNAL TRANSDUCTION; TRETINOIN;

EID: 84941552694     PISSN: 00219258     EISSN: 1083351X     Source Type: Journal    
DOI: 10.1074/jbc.M115.662908     Document Type: Article

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