The multiple roles of pendrin in the kidney

Nephrology Dialysis Transplantation

Volumn 30, Issue 8, 2015, Pages 1257-1266

  Soleimani, Manoocher ^a,b,c  

^a UNIVERSITY OF CINCINNATI   (United States)

^b VETERANS AFFAIRS MEDICAL CENTER   (United States)

^c UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

Author keywords

hypertension; oxalate stone; renal tubular acidosis; salt excretion; volume depletion

Indexed keywords

BICARBONATE; PENDRIN; POTASSIUM; SODIUM CHLORIDE COTRANSPORTER; ANION TRANSPORT PROTEIN; CARRIER PROTEIN; SLC12A3 PROTEIN, HUMAN; SLC12A3 PROTEIN, MOUSE; SLC26A4 PROTEIN, HUMAN; SLC26A4 PROTEIN, MOUSE;

ACID BASE BALANCE; BLOOD PRESSURE REGULATION; CALCIUM EXCRETION; CYSTIC FIBROSIS; ELECTROLYTE DISTURBANCE; GENE MUTATION; HUMAN; HYPERCALCIURIA; HYPERTENSION; KIDNEY; NEPHROLITHIASIS; NONHUMAN; PENDRED SYNDROME; PRIORITY JOURNAL; REVIEW; SIGNAL TRANSDUCTION; SLC26A4 GENE; SODIUM ABSORPTION; SODIUM EXCRETION; STRUCTURE ACTIVITY RELATION; ANIMAL; ELECTROLYTE BALANCE; KIDNEY DISEASE; METABOLISM; MOUSE; PATHOPHYSIOLOGY; PHYSIOLOGY;

ACID-BASE EQUILIBRIUM; ANIMALS; ANION TRANSPORT PROTEINS; HUMANS; KIDNEY DISEASES; MEMBRANE TRANSPORT PROTEINS; MICE; SOLUTE CARRIER FAMILY 12, MEMBER 3; WATER-ELECTROLYTE BALANCE;

EID: 84939603015     PISSN: 09310509     EISSN: 14602385     Source Type: Journal    
DOI: 10.1093/ndt/gfu307     Document Type: Review

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