SLC26 Cl-/HCO3- exchangers in the kidney: Roles in health and disease

Kidney International

Volumn 84, Issue 4, 2013, Pages 657-666

  Soleimani, Manoocher ^a,b,c  

^a UNIVERSITY OF CINCINNATI MEDICAL CENTER   (United States)

^b VETERANS AFFAIRS MEDICAL CENTER   (United States)

^c UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

Author keywords

hypertension; oxalate stone; renal tubular acidosis; salt excretion; volume depletion

Indexed keywords

ALDOSTERONE; BICARBONATE CHLORIDE ANTIPORTER; CARRIER PROTEIN; CHLORIDE CHANNEL; CYSTIC FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR; CYTOKERATIN AE1; DEOXYCORTICOSTERONE ACETATE; DESMOPRESSIN; EPITHELIAL SODIUM CHANNEL; FORMIC ACID; FRUCTOSE; GLUCOSE TRANSPORTER 5; HYDROCHLOROTHIAZIDE; HYPERTONIC SOLUTION; OXALIC ACID; PENDRIN; POTASSIUM; PRESTIN; PROTON TRANSPORTING ADENOSINE TRIPHOSPHATASE; SODIUM; SODIUM CHLORIDE; SODIUM PROTON EXCHANGE PROTEIN 3; SOLUTE LINKED CARRIER 26; SOLUTE LINKED CARRIER 26A11; SOLUTE LINKED CARRIER 26A3; SOLUTE LINKED CARRIER 26A6; SOLUTE LINKED CARRIER 26A7; SOLUTE LINKED CARRIER 26A9; UNCLASSIFIED DRUG; VANILLOID RECEPTOR 5; ANTIPORTER; CHLORIDE; OXALIC ACID DERIVATIVE; SLC26A4 PROTEIN, HUMAN; SLC26A7 PROTEIN, HUMAN; SLC26A9 PROTEIN, HUMAN;

ACID BASE BALANCE; BASOLATERAL MEMBRANE; BLOOD PRESSURE REGULATION; CELL MEMBRANE; CELLULAR DISTRIBUTION; CHLORIDE DIARRHEA; CHLORIDE LOSING DIARRHEA; CHLORIDE TRANSPORT; CHONDRODYSPLASIA; CONGENITAL CHLORIDE DIARRHEA; CYSTIC FIBROSIS; DISEASE ASSOCIATION; DISORDERS OF ACID BASE BALANCE; DIURESIS; DOWN REGULATION; ELECTROLYTE DISTURBANCE; ENZYME ACTIVITY; FOOD INTAKE; HORMONAL REGULATION; HUMAN; HYPERCALCIURIA; HYPERLIPIDEMIA; HYPEROXALURIA; HYPERTENSION; HYPOTENSION; INSULIN RESISTANCE; INTESTINE; KIDNEY; KIDNEY COLLECTING TUBULE; KIDNEY FUNCTION; KIDNEY INTERSTITIUM; KIDNEY PROXIMAL TUBULE; METABOLIC ALKALOSIS; METABOLIC SYNDROME X; MOLECULAR CLONING; NEPHROLITHIASIS; NEPHRON; NONHUMAN; NUCLEOTIDE SEQUENCE; OSMOLALITY; OUTER MEDULLARY COLLECTING DUCT; PATHOPHYSIOLOGY; PENDRED SYNDROME; PHENOTYPE; POTASSIUM DEPLETION; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; PROTEIN PROTEIN INTERACTION; PROTEIN SECRETION; REVIEW; SIDE EFFECT; SODIUM ABSORPTION; SODIUM EXCRETION; SODIUM RESTRICTION; STOMACH ACID SECRETION; TISSUE DISTRIBUTION; UPREGULATION; URINARY EXCRETION; ANIMAL; DISEASE MODEL; HOMEOSTASIS; KIDNEY DISEASE; METABOLISM; MOUSE; PHYSIOLOGY;

ACID-BASE EQUILIBRIUM; ANIMALS; ANTIPORTERS; CHLORIDE-BICARBONATE ANTIPORTERS; CHLORIDES; DISEASE MODELS, ANIMAL; HOMEOSTASIS; HUMANS; KIDNEY; KIDNEY DISEASES; MEMBRANE TRANSPORT PROTEINS; MICE; OXALATES;

EID: 84885308385     PISSN: 00852538     EISSN: 15231755     Source Type: Journal    
DOI: 10.1038/ki.2013.138     Document Type: Review

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