Regulated necrotic cell death: The passive aggressive side of bax and bak

Circulation Research

Volumn 116, Issue 11, 2015, Pages 1800-1809

  Karch, Jason ^a   Molkentin, Jeffery D ^a,b  

^a UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

^b CINCINNATI CHILDREN'S HOSPITAL MEDICAL CENTER   (United States)

Author keywords

apoptosis; calcium; mitochondria; necrosis

Indexed keywords

ADENOSINE TRIPHOSPHATE; CASPASE; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE; NUCLEASE; PROTEIN BAK; PROTEIN BAX; REACTIVE OXYGEN METABOLITE; BAK1 PROTEIN, HUMAN; BAX PROTEIN, HUMAN; CARRIER PROTEIN; MITOCHONDRIAL PROTEIN;

APOPTOSIS; ENZYME ACTIVATION; MITOCHONDRIAL MEMBRANE; NONHUMAN; OLIGOMERIZATION; PRIORITY JOURNAL; PROTEIN FAMILY; REGULATORY MECHANISM; REVIEW; BIOLOGICAL MODEL; HUMAN; METABOLISM; MITOCHONDRION; NECROSIS;

BCL-2 HOMOLOGOUS ANTAGONIST-KILLER PROTEIN; BCL-2-ASSOCIATED X PROTEIN; HUMANS; MITOCHONDRIA; MITOCHONDRIAL MEMBRANE TRANSPORT PROTEINS; MITOCHONDRIAL MEMBRANES; MITOCHONDRIAL PROTEINS; MODELS, BIOLOGICAL; NECROSIS; REACTIVE OXYGEN SPECIES;

EID: 84936815629     PISSN: 00097330     EISSN: 15244571     Source Type: Journal    
DOI: 10.1161/CIRCRESAHA.116.305421     Document Type: Review

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