Activation of C3a receptor is required in cigarette smoke-mediated emphysema

Mucosal Immunology

Volumn 8, Issue 4, 2015, Pages 874-885

  Yuan, X ^a   Shan, M ^a   You, R ^a   Frazier, M V ^a   Hong, M J ^a   Wetsel, R A ^b   Drouin, S ^b   Seryshev, A ^a   Song, L Z ^a   Cornwell, L ^a   Rossen, R D ^a,c   Corry, D B ^a,c   Kheradmand, F ^a,c  

^a BAYLOR COLLEGE OF MEDICINE   (United States)

^b UNIVERSITY OF TEXAS MEDICAL SCHOOL   (United States)

^c Michael E Debakey VA Center for Translational Research on Inflammatory Diseases   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CD11B ANTIGEN; COMPLEMENT COMPONENT C3A; GLYCOPROTEIN P 15095; LEUKOCYTE ELASTASE; COMPLEMENT C3A RECEPTOR; COMPLEMENT COMPONENT C3; COMPLEMENT RECEPTOR; MACROPHAGE ELASTASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; AUTOCRINE EFFECT; CELL INFILTRATION; CLINICAL ARTICLE; CONTROLLED STUDY; DENDRITIC CELL; ENZYME RELEASE; HUMAN; HUMAN CELL; HUMAN TISSUE; LUNG EMPHYSEMA; MOLECULAR PATHOLOGY; MOUSE; NONHUMAN; PARACRINE SIGNALING; PRIORITY JOURNAL; PROTEIN CLEAVAGE; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; PROTEIN SECRETION; RECEPTOR UPREGULATION; SMOKING; WILD TYPE; ADVERSE EFFECTS; ANIMAL; COMPLEMENT ACTIVATION; DEFICIENCY; DISEASE MODEL; EMPHYSEMA; GENE EXPRESSION REGULATION; GENETICS; IMMUNOLOGY; KNOCKOUT MOUSE; LUNG; METABOLISM; NEUTROPHIL CHEMOTAXIS; PATHOLOGY; PROTEIN DEGRADATION; SIGNAL TRANSDUCTION;

ANIMALS; AUTOCRINE COMMUNICATION; CHEMOTAXIS, LEUKOCYTE; COMPLEMENT ACTIVATION; COMPLEMENT C3; COMPLEMENT C3A; DISEASE MODELS, ANIMAL; EMPHYSEMA; GENE EXPRESSION REGULATION; HUMANS; LEUKOCYTE ELASTASE; LUNG; MATRIX METALLOPROTEINASE 12; MICE, KNOCKOUT; PARACRINE COMMUNICATION; PROTEOLYSIS; RECEPTORS, COMPLEMENT; SIGNAL TRANSDUCTION; SMOKING;

EID: 84932610545     PISSN: 19330219     EISSN: 19353456     Source Type: Journal    
DOI: 10.1038/mi.2014.118     Document Type: Article

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