SERCA1 overexpression minimizes skeletal muscle damage in dystrophic mouse models

American Journal of Physiology - Cell Physiology

Volumn 308, Issue 9, 2015, Pages C699-C709

  Mázala, Davi A G ^a   Pratt, Stephen J P ^b   Chen, Dapeng ^a   Molkentin, Jeffery D ^c   Lovering, Richard M ^b   Chin, Eva R ^a  

^a UNIVERSITY OF MARYLAND   (United States)

^b UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE   (United States)

^c UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

Author keywords

Calcium; Duchenne muscular dystrophy; Injury; Mdx; Mdx Utr ; SERCA1

Indexed keywords

CALCIUM ION; SARCOPLASMIC RETICULUM CALCIUM TRANSPORTING ADENOSINE TRIPHOSPHATASE; SARCOPLASMIC RETICULUM CALCIUM TRANSPORTING ADENOSINE TRIPHOSPHATASE 1; UNCLASSIFIED DRUG; BIOLOGICAL MARKER; CREATINE KINASE MM; UTROPHIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BODY MASS; CALCIUM CELL LEVEL; CONTROLLED STUDY; DIAPHRAGM; DISEASE PREDISPOSITION; DUCHENNE MUSCULAR DYSTROPHY; ECCENTRIC MUSCLE CONTRACTION; GASTROCNEMIUS MUSCLE; GENE OVEREXPRESSION; GENOTYPE; MOUSE; MUSCLE CELL; MUSCLE INJURY; MUSCLE MASS; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; QUADRICEPS FEMORIS MUSCLE; SOLEUS MUSCLE; TORQUE; ANIMAL; BIOMECHANICS; BLOOD; CALCIUM SIGNALING; DEFICIENCY; DISEASE MODEL; ENZYMOLOGY; GENETICS; HYPERTROPHY; METABOLISM; MUSCLE CONTRACTION; MUSCLE STRENGTH; NECROSIS; ORGAN SIZE; PATHOLOGY; PATHOPHYSIOLOGY; PHENOTYPE; SEVERITY OF ILLNESS INDEX; TRANSGENIC MOUSE; UPREGULATION; X CHROMOSOME LINKED MUSCULAR DYSTROPHIC MOUSE;

MUS;

ANIMALS; BIOMARKERS; BIOMECHANICAL PHENOMENA; CALCIUM SIGNALING; CREATINE KINASE, MM FORM; DISEASE MODELS, ANIMAL; GENOTYPE; HYPERTROPHY; MICE, INBRED MDX; MICE, TRANSGENIC; MUSCLE CONTRACTION; MUSCLE STRENGTH; MUSCULAR DYSTROPHY, DUCHENNE; NECROSIS; ORGAN SIZE; PHENOTYPE; QUADRICEPS MUSCLE; SARCOPLASMIC RETICULUM CALCIUM-TRANSPORTING ATPASES; SEVERITY OF ILLNESS INDEX; TORQUE; UP-REGULATION; UTROPHIN;

EID: 84930836359     PISSN: 03636143     EISSN: 15221563     Source Type: Journal    
DOI: 10.1152/ajpcell.00341.2014     Document Type: Article

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