C/EBP homologous protein (CHOP) contributes to hepatocyte death via the promotion of ERO1α signalling in acute liver failure

Biochemical Journal

Volumn 466, Issue , 2015, Pages 369-378

  Rao, Jianhua ^a   Zhang, Chuangyong ^a   Wang, Ping ^a   Lu, Ling ^a   Qian, Xiaofeng ^a   Qin, Jianjie ^a   Pan, Xiongxiong ^a   Li, Guoqiang ^a   Wang, Xuehao ^a   Zhang, Feng ^a  

^a NANJING MEDICAL UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

ACTIVATING TRANSCRIPTION FACTOR 4; CCAAT ENHANCER BINDING PROTEIN HOMOLOGOUS PROTEIN; ENDOPLASMIC RETICULUM OXIDOREDUCTIN 1 ALPHA; ENZYME; GALACTOSAMINE; INOSITOL REQUIRING ENZYME 1; LIPOPOLYSACCHARIDE; MESSENGER RNA; PROTEIN; PROTEIN KINASE R; REACTIVE OXYGEN METABOLITE; RNA; UNCLASSIFIED DRUG; DDIT3 PROTEIN, HUMAN; DDIT3 PROTEIN, MOUSE; ERO1L PROTEIN, HUMAN; ERO1L PROTEIN, MOUSE; GLYCOPROTEIN; GROWTH ARREST AND DNA DAMAGE INDUCIBLE PROTEIN 153; MEMBRANE PROTEIN; OXIDOREDUCTASE; RECOMBINANT PROTEIN;

ACUTE LIVER FAILURE; ADULT; ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; CELL DEATH; CELL SURVIVAL; CONTROLLED STUDY; EDEMA; ENDOPLASMIC RETICULUM STRESS; FEMALE; HUMAN; HUMAN CELL; IN VITRO STUDY; IN VIVO STUDY; LIVER CELL; LIVER CELL DAMAGE; LIVER INJURY; MALE; MOUSE; MOUSE MODEL; NONHUMAN; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN DEPLETION; PROTEIN EXPRESSION; PROTEIN FUNCTION; SIGNAL TRANSDUCTION; UNFOLDED PROTEIN RESPONSE; UPREGULATION; ANIMAL; ANTAGONISTS AND INHIBITORS; C57BL MOUSE; CELL CULTURE; CHEMISTRY; DISEASE MODEL; ENZYMOLOGY; GENE SILENCING; GENETICS; KNOCKOUT MOUSE; METABOLISM; MIDDLE AGED; NECROSIS; PATHOLOGY; SURVIVAL;

ANIMALIA; MUS;

ADULT; ANIMALS; APOPTOSIS; CELLS, CULTURED; DISEASE MODELS, ANIMAL; FEMALE; GENE SILENCING; GLYCOPROTEINS; HEPATOCYTES; HUMANS; LIVER FAILURE, ACUTE; MALE; MAP KINASE SIGNALING SYSTEM; MEMBRANE GLYCOPROTEINS; MICE, INBRED C57BL; MICE, KNOCKOUT; MIDDLE AGED; NECROSIS; OXIDOREDUCTASES; RECOMBINANT PROTEINS; SURVIVAL ANALYSIS; TRANSCRIPTION FACTOR CHOP; UP-REGULATION;

EID: 84923233769     PISSN: 02646021     EISSN: 14708728     Source Type: Journal    
DOI: 10.1042/BJ20140412     Document Type: Article

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