Premature lethality, hyperactivity, and aberrant phosphorylation in transgenic mice expressing a constitutively active form of Fyn

Frontiers in Molecular Neuroscience

Volumn 7, Issue MAY, 2014, Pages

  Xia, Di ^a   Götz, Jürgen ^a  

^a UNIVERSITY OF QUEENSLAND   (Australia)

Author keywords

Alzheimer; Dendrite; Fyn kinase; Palmitoylation; Phosphorylation; Spine; Tau

Indexed keywords

N METHYL DEXTRO ASPARTIC ACID RECEPTOR; PROTEIN KINASE FYN; PROTEIN TYROSINE KINASE; TAU PROTEIN;

ALZHEIMER DISEASE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BRAIN FUNCTION; CELL CULTURE; CELLULAR DISTRIBUTION; CLINICAL ASSESSMENT; CONTROLLED STUDY; DENDRITIC SPINE; HYPERACTIVITY; IMMUNOHISTOCHEMISTRY; LETHALITY; MOUSE; MUTAGENESIS; NONHUMAN; PALMITOYLATION; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; QUANTITATIVE ANALYSIS; SYNAPTOSOME; WESTERN BLOTTING;

EID: 84901367930     PISSN: 16625099     EISSN: None     Source Type: Journal    
DOI: 10.3389/fnmol.2014.00040     Document Type: Article

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