The C-type lectin receptor DCIR is crucial for the development of experimental cerebral malaria

Journal of Immunology

Volumn 191, Issue 5, 2013, Pages 2551-2559

  Maglinao, Maha ^a,b   Klopfleisch, Robert ^b   Seeberger, Peter H ^a,b   Lepenies, Bernd ^a,b  

^a MAX PLANCK INSTITUTE OF COLLOIDS AND INTERFACES   (Germany)

^b FREIE UNIVERSITÄT BERLIN   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

DENDRITIC CELL IMMUNORECEPTOR; GAMMA INTERFERON; INTERLEUKIN 10; INTERLEUKIN 12P40; INTERLEUKIN 12P70; INTERLEUKIN 1BETA; LECTIN RECEPTOR; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN MALARIA; BRAIN TISSUE; CD4+ T LYMPHOCYTE; CD8+ T LYMPHOCYTE; CONTROLLED STUDY; CYTOKINE RELEASE; EXPERIMENTAL STUDY; FEMALE; HISTOPATHOLOGY; IMMUNOPATHOGENESIS; INNATE IMMUNITY; MOUSE; NONHUMAN; PLASMODIUM BERGHEI INFECTION; PRIORITY JOURNAL; PROTECTION; PROTEIN BLOOD LEVEL; PROTEIN DEFICIENCY; PROTEIN FUNCTION; SPLEEN; T LYMPHOCYTE ACTIVATION; WILD TYPE;

ANIMALS; BRAIN; DISEASE MODELS, ANIMAL; FLOW CYTOMETRY; LECTINS, C-TYPE; MALARIA, CEREBRAL; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; PLASMODIUM BERGHEI; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION;

EID: 84883348224     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.1203451     Document Type: Article

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