Regulation of gap junctions by nitric oxide influences the generation of arrhythmias resulting from acute ischemia and reperfusion in vivo

Frontiers in Pharmacology

Volumn 4 JUN, Issue , 2013, Pages

  Végh, Ágnes ^a   Gönczi, Márton ^a   Miskolczi, Gottfried ^a   Kovács, Mária ^a  

^a UNIVERSITY OF SZEGED   (Hungary)

Author keywords

Arrhythmias; Gap junction; Ischemia reperfusion; Nitric oxide

Indexed keywords

ANTIARRHYTHMIC PEPTIDE; ANTIARRHYTHMIC PEPTIDE 10; CARBENOXOLONE; CONNEXIN 43; CYCLIC AMP DEPENDENT PROTEIN KINASE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; NITRITE; NITROPRUSSIDE SODIUM; PROTEIN KINASE C; ROTIGAPTIDE; SODIUM NITRITE; UNCLASSIFIED DRUG;

ARRHYTHMOGENESIS; ARTICLE; CARDIAC GAP JUNCTION; DOG; DRUG ACTIVITY; DRUG EFFICACY; DRUG POTENCY; GAP JUNCTION; HEART INFARCTION; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; HEART PROTECTION; HEART VENTRICLE ARRHYTHMIA; IMPEDANCE; IN VIVO STUDY; ISCHEMIC PRECONDITIONING; LEFT CORONARY ARTERY; MEMBRANE PERMEABILITY; NITROSYLATION; NONHUMAN; PROTEIN PHOSPHORYLATION;

EID: 84881588838     PISSN: None     EISSN: 16639812     Source Type: Journal    
DOI: 10.3389/fphar.2013.00076     Document Type: Article

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