Protein kinase C iota regulates pancreatic acinar-to-ductal metaplasia

PLoS ONE

Volumn 7, Issue 2, 2012, Pages

  Scotti, Michele L ^a   Smith, Kristin E ^a   Butler, Amanda M ^a   Calcagno, Shelly R ^a   Crawford, Howard C ^b   Leitges, Michael ^c   Fields, Alan P ^a   Murray, Nicole R ^a  

^a MAYO CLINIC   (United States)

^b STONY BROOK UNIVERSITY   (United States)

^c UNIVERSITY OF OSLO   (Norway)

Author keywords

[No Author keywords available]

Indexed keywords

CYTOKERATIN 19; MATRILYSIN; NESTIN; NOTCH RECEPTOR; PROTEIN KINASE C IOTA; TRANSFORMING GROWTH FACTOR ALPHA; TUMOR MARKER; ISOENZYME; PROTEIN KINASE C; PROTEIN KINASE C LAMBDA; PROTEIN P21;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL DIFFERENTIATION; CONTROLLED STUDY; DISEASE MARKER; DOWN REGULATION; GENE REPRESSION; HUMAN; IN VITRO STUDY; IN VIVO STUDY; MALIGNANT TRANSFORMATION; MOUSE; NONHUMAN; ONCOGENE K RAS; PANCREAS CANCER; PROTEIN DNA INTERACTION; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; PROTEIN SYNTHESIS INHIBITION; SIGNAL TRANSDUCTION; UPREGULATION; ACINAR CELL; ANIMAL; CELL CULTURE; CHEMICALLY INDUCED DISORDER; METABOLISM; METAPLASIA; PANCREAS DUCT; PATHOLOGY; PHYSIOLOGY;

ACINAR CELLS; ANIMALS; CELLS, CULTURED; ISOENZYMES; MATRIX METALLOPROTEINASE 7; METAPLASIA; MICE; PANCREATIC DUCTS; PROTEIN KINASE C; PROTO-ONCOGENE PROTEINS P21(RAS); RECEPTORS, NOTCH; TRANSFORMING GROWTH FACTOR ALPHA;

EID: 84863408964     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0030509     Document Type: Article

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