Exacerbation of endothelial dysfunction during the progression of diabetes: Role of oxidative stress

American Journal of Physiology - Regulatory Integrative and Comparative Physiology

Volumn 302, Issue 6, 2012, Pages 674-681

  Huang, An ^a   Yang, Yang Ming ^a   Feher, Attila ^a   Bagi, Zsolt ^a   Kaley, Gabor ^a   Sun, Dong ^a,b  

^a NEW YORK MEDICAL COLLEGE   (United States)

^b XUZHOU MEDICAL UNIVERSITY   (China)

Author keywords

Diabetes; Endothelium; Nitric oxide; Nitrotyrosine; Shear stress

Indexed keywords

3 NITROTYROSINE; ENDOTHELIAL NITRIC OXIDE SYNTHASE; INDUCIBLE NITRIC OXIDE SYNTHASE; NITRIC OXIDE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE; SUPEROXIDE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTERY DILATATION; ARTICLE; BIOAVAILABILITY; CONTROLLED STUDY; DIABETES MELLITUS; DISEASE COURSE; DISEASE EXACERBATION; ENDOTHELIAL DYSFUNCTION; ENZYME INHIBITION; ENZYME PHOSPHORYLATION; IMMUNOPRECIPITATION; MALE; MESENTERIC ARTERY; MOUSE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; SHEAR STRESS; WILD TYPE;

ANIMALS; DIABETES MELLITUS, TYPE 2; DISEASE MODELS, ANIMAL; DISEASE PROGRESSION; ENDOTHELIUM, VASCULAR; HYPERGLYCEMIA; HYPERINSULINISM; MALE; MICE; MICE, INBRED C57BL; MICE, MUTANT STRAINS; NITRIC OXIDE; NITRIC OXIDE SYNTHASE TYPE II; NITRIC OXIDE SYNTHASE TYPE III; OXIDATIVE STRESS; SUPEROXIDES; TIME FACTORS; TYROSINE;

EID: 84863266319     PISSN: 03636119     EISSN: 15221490     Source Type: Journal    
DOI: 10.1152/ajpregu.00699.2011     Document Type: Article

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