Histone deacetylase inhibitor sodium butyrate enhances cellular radiosensitivity by inhibiting both DNA nonhomologous end joining and homologous recombination

DNA Repair

Volumn 10, Issue 9, 2011, Pages 970-977

  Koprinarova, Miglena ^a   Botev, Peter ^a   Russev, George ^a  

^a INSTITUTE OF MOLECULAR BIOLOGY   (Bulgaria)

Author keywords

Irradiation; DNA nonhomologous end joining; Histone acetylation; Homologous recombination; Sodium butyrate

Indexed keywords

BUTYRIC ACID; DNA; HISTONE DEACETYLASE INHIBITOR; HISTONE H2AX; RAD51 PROTEIN; RADIOSENSITIZING AGENT;

AGAR GEL ELECTROPHORESIS; ARTICLE; CELL CYCLE ARREST; CELL CYCLE G1 PHASE; CELL CYCLE G2 PHASE; CHROMATIN; CHROMATIN STRUCTURE; CONTROLLED STUDY; DRUG EFFECT; DYNAMICS; FLUORESCENCE ACTIVATED CELL SORTING; GAMMA RADIATION; HELA CELL; HOST CELL; HUMAN; HUMAN CELL; MONITORING; PLASMID; PRIORITY JOURNAL; RADIOSENSITIVITY;

BUTYRATES; CELL CYCLE; DNA BREAKS, DOUBLE-STRANDED; DNA REPAIR; GAMMA RAYS; HELA CELLS; HISTONE DEACETYLASE INHIBITORS; HISTONES; HOMOLOGOUS RECOMBINATION; HUMANS; RADIATION TOLERANCE; RECOMBINATION, GENETIC;

EID: 80051818192     PISSN: 15687864     EISSN: 15687856     Source Type: Journal    
DOI: 10.1016/j.dnarep.2011.07.003     Document Type: Article

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