Cyclophilin D deficiency improves mitochondrial function and learning/memory in aging Alzheimer disease mouse model

Neurobiology of Aging

Volumn 32, Issue 3, 2011, Pages 398-406

  Du, Heng ^a   Guo, Lan ^a   Zhang, Wensheng ^a,b   Rydzewska, Monika ^a   Yan, Shidu ^a  

^a College of Physicians and Surgeons of Columbia University ^*  (United States)

^b BEIJING NORMAL UNIVERSITY   (China)

Author keywords

Alzheimer's disease; Amyloid beta; Cyclophilin D; Mitochondrial permeability transition pore

Indexed keywords

AMYLOID BETA PROTEIN; AMYLOID PRECURSOR PROTEIN; CALCIUM; CYCLOPHILIN D; CYTOCHROME C OXIDASE;

AGING; ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN MITOCHONDRION; CALCIUM TRANSPORT; CONTROLLED STUDY; HUMAN; LEARNING; MITOCHONDRIAL PERMEABILITY; MITOCHONDRIAL RESPIRATION; MITOCHONDRION SWELLING; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SPATIAL LEARNING; SPATIAL MEMORY;

AGING; ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN PRECURSOR; ANALYSIS OF VARIANCE; ANIMALS; BRAIN; CALCIUM; CYCLOPHILINS; DISEASE MODELS, ANIMAL; ELECTRON TRANSPORT COMPLEX IV; HUMANS; LEARNING DISORDERS; MAZE LEARNING; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MITOCHONDRIA; MITOCHONDRIAL MEMBRANE TRANSPORT PROTEINS; MUTATION; PROTEIN TRANSPORT;

EID: 79952901937     PISSN: 01974580     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neurobiolaging.2009.03.003     Document Type: Article

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