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Archives of Biochemistry and Biophysics

Volumn 508, Issue 2, 2011, Pages 198-203

Ubiquitin proteasomal pathway mediated degradation of p53 in melanoma

(3) Anwar, Adil a,c Norris, David A a,b Fujita, Mayumi a,b

a UNIVERSITY OF COLORADO SCHOOL OF MEDICINE (United States)

b VETERANS AFFAIRS MEDICAL CENTER (United States)

c UNIVERSITY OF COLORADO (United States)

Author keywords

p53; Proteasomal degradation; Proteasomal inhibitor MG132; Skin Cancer; Tumor suppressor; Ubiquitination

Indexed keywords

BENZYLOXYCARBONYLLEUCYLLEUCYLLEUCINAL; CYCLOHEXIMIDE; LACTACYSTIN BETA LACTONE; PROTEASOME; PROTEASOME INHIBITOR; PROTEIN P53; UBIQUITIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTINEOPLASTIC ACTIVITY; ARTICLE; CANCER SIZE; CELL CYCLE ARREST; CELL CYCLE G1 PHASE; CELL GROWTH; CONTROLLED STUDY; HALF LIFE TIME; MELANOMA; MELANOMA CELL; MOLECULAR WEIGHT; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN MODIFICATION; PROTEIN PHOSPHORYLATION; PROTEIN STABILITY; PROTEIN SYNTHESIS INHIBITION; TRANSGENIC MOUSE; UBIQUITINATION;

ANIMALS; CELL LINE, TUMOR; ETOPOSIDE; G1 PHASE; GENE DELETION; GENES, P16; HALF-LIFE; HUMANS; LACTONES; LEUPEPTINS; MELANOCYTES; MELANOMA; MICE; MICE, TRANSGENIC; PROTEASE INHIBITORS; PROTEASOME ENDOPEPTIDASE COMPLEX; PROTEIN STABILITY; TUMOR SUPPRESSOR PROTEIN P53; UBIQUITIN;

EID: 79952651012 PISSN: 00039861 EISSN: 10960384 Source Type: Journal
DOI: 10.1016/j.abb.2010.12.012 Document Type: Article

Times cited : (10)

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