Targeting the FANCJ-BRCA1 interaction promotes a switch from recombination to pol-dependent bypass

Oncogene

Volumn 29, Issue 17, 2010, Pages 2499-2508

  Xie, J ^a   Litman, R ^a   Wang, S ^a   Peng, M ^a   Guillemette, S ^a   Rooney, T ^a   Cantor, S B ^a  

^a UNIVERSITY OF MASSACHUSETTS MEDICAL SCHOOL   (United States)

Author keywords

BRCA1; DNA repair; FANCJ

Indexed keywords

BRCA1 PROTEIN; DNA; FANCJ HELICASE; HELICASE; MISMATCH REPAIR PROTEIN; PROTEIN MLH1; UNCLASSIFIED DRUG;

ARTICLE; CANCER CELL CULTURE; CARCINOGENESIS; CONTROLLED STUDY; DNA DAMAGE; GENOMIC INSTABILITY; HUMAN; HUMAN CELL; PRIORITY JOURNAL; PROTEIN PROTEIN INTERACTION; SIGNAL TRANSDUCTION;

ADAPTOR PROTEINS, SIGNAL TRANSDUCING; BASIC-LEUCINE ZIPPER TRANSCRIPTION FACTORS; BRCA1 PROTEIN; CELL LINE, TUMOR; DNA DAMAGE; DNA REPAIR; DNA-DIRECTED DNA POLYMERASE; DRUG RESISTANCE, NEOPLASM; FANCONI ANEMIA COMPLEMENTATION GROUP PROTEINS; GENOMIC INSTABILITY; HUMANS; MITOMYCIN; NUCLEAR PROTEINS; RECOMBINATION, GENETIC;

EID: 77951878399     PISSN: 09509232     EISSN: 14765594     Source Type: Journal    
DOI: 10.1038/onc.2010.18     Document Type: Article

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