Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1β production

Nature

Volumn 456, Issue 7219, 2008, Pages 264-268

  Saitoh, Tatsuya ^a,b   Fujita, Naonobu ^b   Jang, Myoung Ho ^a   Uematsu, Satoshi ^a,b   Yang, Bo Gie ^a,b   Satoh, Takashi ^a,b   Omori, Hiroko ^b   Noda, Takeshi ^b   Yamamoto, Naoki ^c   Komatsu, Masaaki ^d,e,f   Tanaka, Keiji ^d   Kawai, Taro ^a,b   Tsujimura, Tohru ^g   Takeuchi, Osamu ^a,b   Yoshimori, Tamotsu ^b,f   Akira, Shizuo ^a,b  

^a OSAKA UNIVERSITY   (Japan)

^b OSAKA UNIVERSITY   (Japan)

^c NATIONAL INSTITUTE OF INFECTIOUS DISEASES   (Japan)

^d TOKYO METROPOLITAN INSTITUTE OF MEDICAL SCIENCE   (Japan)

^e JUNTENDO UNIVERSITY SCHOOL OF MEDICINE   (Japan)

^f JAPAN SCIENCE AND TECHNOLOGY AGENCY   (Japan)

^g HYOGO COLLEGE OF MEDICINE   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

BETA INTERFERON; CELL PROTEIN; DEXTRAN SULFATE; INTERLEUKIN 1 RECEPTOR; INTERLEUKIN 18 ANTIBODY; INTERLEUKIN 18 BINDING PROTEIN; INTERLEUKIN 1BETA; INTERLEUKIN 1BETA ANTIBODY; INTERLEUKIN 1BETA CONVERTING ENZYME; LIPOPOLYSACCHARIDE; MICROTUBULE ASSOCIATED PROTEIN 1; PHOSPHATIDYLETHANOLAMINE; PROTEIN ATG16L1; TOLL LIKE RECEPTOR 4; UNCLASSIFIED DRUG;

CYTOPLASM; DEGRADATION; HISTOLOGY; IMMUNE RESPONSE; PHOSPHOLIPID; POLYMER; PROTEIN; RODENT; SODIUM; TOXIN;

ANIMAL TISSUE; ARTICLE; AUTOPHAGY; CELL STIMULATION; COLITIS; CONJUGATION; CONTROLLED STUDY; CROHN DISEASE; CYTOKINE PRODUCTION; HEMATOPOIETIC CELL; HISTOPATHOLOGY; IMMUNE RESPONSE; INFLAMMATION; LIGHT CHAIN; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEFICIENCY; PROTEIN DEGRADATION; PROTEIN DOMAIN;

ADENINE; ANIMALS; AUTOPHAGY; CHIMERA; COLITIS; DEXTRAN SULFATE; FEMALE; GENE EXPRESSION REGULATION; INTERLEUKIN-1BETA; LIPOPOLYSACCHARIDES; MACROPHAGES; MICE; MICE, INBRED C57BL; MUTATION;

MUS;

EID: 56249090667     PISSN: 00280836     EISSN: 14764679     Source Type: Journal    
DOI: 10.1038/nature07383     Document Type: Article

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