Invariant NKT cells produce IL-17 through IL-23-dependent and -independent pathways with potential modulation of Th17 response in collagen-induced arthritis

International Journal of Molecular Medicine

Volumn 22, Issue 3, 2008, Pages 369-374

  Yoshiga, Y ^a   Goto, D ^a   Segawa, D ^a   Ohnishi, Y ^b   Matsumoto, I ^a   Ito, S ^a   Tsutsumi, A ^a   Taniguchi, M ^c   Sumida, Takayuki ^a  

^a UNIVERSITY OF TSUKUBA   (Japan)

^b MITSUBISHI CHEMICAL CORPORATION   (Japan)

^c RIKEN Center for Integrative Medical Sciences   (Japan)

Author keywords

Collagen induced arthritis; Interleukin 17; Interleukin 23; Natural killer T cells; T helper 17 cells

Indexed keywords

ALPHA GALACTOSYLCERAMIDE; COLLAGEN TYPE 2; INTERLEUKIN 17; INTERLEUKIN 23;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTHRITIS; ARTICLE; CELL SELECTION; CELL STIMULATION; CONTROLLED STUDY; CYTOKINE PRODUCTION; ENZYME LINKED IMMUNOSORBENT ASSAY; HELPER CELL; IMMUNOHISTOCHEMISTRY; IMMUNOMODULATION; IMMUNOPATHOGENESIS; LYMPHOCYTE COUNT; LYMPHOCYTE FUNCTION; MOUSE; NATURAL KILLER T CELL; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; SPLEEN CELL; ANIMAL; BIOSYNTHESIS; C57BL MOUSE; IMMUNOLOGY; METABOLISM; REGULATORY T LYMPHOCYTE;

ANIMALS; ARTHRITIS, EXPERIMENTAL; INTERLEUKIN-17; INTERLEUKIN-23; MICE; MICE, INBRED C57BL; SIGNAL TRANSDUCTION; T-LYMPHOCYTES, HELPER-INDUCER; T-LYMPHOCYTES, REGULATORY;

EID: 53049086275     PISSN: 11073756     EISSN: None     Source Type: Journal    
DOI: 10.3892/ijmm_00000032     Document Type: Article

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