Demonstration of the essential role of protein kinase C isoforms in hyperglycemia-induced embryonic malformations

Reproductive Sciences

Volumn 15, Issue 4, 2008, Pages 349-356

  Zhao, Zhiyong ^a   Wu, Ying King ^a   Reece, E Albert ^a  

^a UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE   (United States)

Author keywords

Apoptosis; cPLA2; Diabetic embryopathy; PKC

Indexed keywords

CYTOSOLIC PHOSPHOLIPASE A2; PHOSPHOLIPASE A2; PROTEIN KINASE C; PROTEIN KINASE C ALPHA; PROTEIN KINASE C BETA; PROTEIN KINASE C BETA1; PROTEIN KINASE C BETA2; PROTEIN KINASE C DELTA; PROTEIN KINASE C EPSILON; PROTEIN KINASE C GAMMA; PROTEIN KINASE C ZETA;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELLULAR DISTRIBUTION; CONGENITAL MALFORMATION; CONTROLLED STUDY; EMBRYO; EMBRYOPATHY; ENZYME ACTIVATION; ENZYME INHIBITION; FEMALE; HYPERGLYCEMIA; LIPID PEROXIDATION; MALE; NEURAL TUBE; NONHUMAN; PREGNANCY DIABETES MELLITUS; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; RAT;

ANIMALS; APOPTOSIS; BLOTTING, WESTERN; DIABETES MELLITUS, EXPERIMENTAL; EMBRYO CULTURE TECHNIQUES; EMBRYONIC DEVELOPMENT; FEMALE; FETAL DISEASES; HYPERGLYCEMIA; MALE; NEURAL TUBE; NEURAL TUBE DEFECTS; PHOSPHOLIPASES A2, CYTOSOLIC; PHOSPHORYLATION; PREGNANCY; PROTEIN ISOFORMS; PROTEIN KINASE C; PROTEIN KINASE INHIBITORS; RATS; RATS, SPRAGUE-DAWLEY; SIGNAL TRANSDUCTION; YOLK SAC;

EID: 43849108731     PISSN: 19337191     EISSN: 19337205     Source Type: Journal    
DOI: 10.1177/1933719108316986     Document Type: Article

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