-
1
-
-
4844223738
-
Does the Leishmania major paradigm of pathogenesis and protection hold for New World cutaneous leishmaniases or the visceral disease?
-
McMahon-Pratt D., and Alexander J. Does the Leishmania major paradigm of pathogenesis and protection hold for New World cutaneous leishmaniases or the visceral disease?. Immunol. Rev. 201 (2004) 206-224
-
(2004)
Immunol. Rev.
, vol.201
, pp. 206-224
-
-
McMahon-Pratt, D.1
Alexander, J.2
-
2
-
-
0031112271
-
IL-10 is required to prevent immune hyperactivity during infection with Trypanosoma cruzi
-
Hunter C.A., et al. IL-10 is required to prevent immune hyperactivity during infection with Trypanosoma cruzi. J. Immunol. 158 (1997) 3311-3316
-
(1997)
J. Immunol.
, vol.158
, pp. 3311-3316
-
-
Hunter, C.A.1
-
3
-
-
28444480206
-
Type 1 chemokine receptor expression in Chagas' disease correlates with morbidity in cardiac patients
-
Gomes J.A., et al. Type 1 chemokine receptor expression in Chagas' disease correlates with morbidity in cardiac patients. Infect. Immun. 73 (2005) 7960-7966
-
(2005)
Infect. Immun.
, vol.73
, pp. 7960-7966
-
-
Gomes, J.A.1
-
4
-
-
0035478642
-
Apoptotic deletion of Th cells specific for the 19-kDa carboxyl-terminal fragment of merozoite surface protein 1 during malaria infection
-
Wipasa J., et al. Apoptotic deletion of Th cells specific for the 19-kDa carboxyl-terminal fragment of merozoite surface protein 1 during malaria infection. J. Immunol. 167 (2001) 3903-3909
-
(2001)
J. Immunol.
, vol.167
, pp. 3903-3909
-
-
Wipasa, J.1
-
5
-
-
0028816515
-
+ T cell death by apoptosis in experimental Chagas' disease
-
+ T cell death by apoptosis in experimental Chagas' disease. J. Immunol. 154 (1995) 744-752
-
(1995)
J. Immunol.
, vol.154
, pp. 744-752
-
-
Lopes, M.F.1
-
6
-
-
31344438663
-
Atrophy of mesenteric lymph nodes in experimental Chagas' disease: differential role of Fas/Fas-L and TNFRI/TNF pathways
-
de Meis J., et al. Atrophy of mesenteric lymph nodes in experimental Chagas' disease: differential role of Fas/Fas-L and TNFRI/TNF pathways. Microbes Infect. 8 (2006) 221-231
-
(2006)
Microbes Infect.
, vol.8
, pp. 221-231
-
-
de Meis, J.1
-
7
-
-
14644420959
-
Myocardiocyte apoptosis in heart failure in chronic Chagas' disease
-
Tostes Jr. S., et al. Myocardiocyte apoptosis in heart failure in chronic Chagas' disease. Int. J. Cardiol. 99 (2005) 233-237
-
(2005)
Int. J. Cardiol.
, vol.99
, pp. 233-237
-
-
Tostes Jr., S.1
-
8
-
-
0034016495
-
Trypanosoma cruzi-induced immunosuppression: B cells undergo spontaneous apoptosis and lipopolysaccharide (LPS) arrests their proliferation during acute infection
-
Zuniga E., et al. Trypanosoma cruzi-induced immunosuppression: B cells undergo spontaneous apoptosis and lipopolysaccharide (LPS) arrests their proliferation during acute infection. Clin. Exp. Immunol. 119 (2000) 507-515
-
(2000)
Clin. Exp. Immunol.
, vol.119
, pp. 507-515
-
-
Zuniga, E.1
-
9
-
-
0035479324
-
Intracellular protozoan parasites and apoptosis: diverse strategies to modulate parasite-host interactions
-
Luder C.G., et al. Intracellular protozoan parasites and apoptosis: diverse strategies to modulate parasite-host interactions. Trends Parasitol. 17 (2001) 480-486
-
(2001)
Trends Parasitol.
, vol.17
, pp. 480-486
-
-
Luder, C.G.1
-
10
-
-
2442560408
-
Manipulation of apoptosis in the host-parasite interaction
-
James E.R., and Green D.R. Manipulation of apoptosis in the host-parasite interaction. Trends Parasitol. 20 (2004) 280-287
-
(2004)
Trends Parasitol.
, vol.20
, pp. 280-287
-
-
James, E.R.1
Green, D.R.2
-
11
-
-
33749367964
-
Lymphocyte apoptosis as an immune subversion strategy of microbial pathogens
-
Carrero J.A., and Unanue E.R. Lymphocyte apoptosis as an immune subversion strategy of microbial pathogens. Trends Immunol. 27 (2006) 497-503
-
(2006)
Trends Immunol.
, vol.27
, pp. 497-503
-
-
Carrero, J.A.1
Unanue, E.R.2
-
12
-
-
17444393621
-
The importance of aberrant T-cell responses in Chagas disease
-
DosReis G.A., et al. The importance of aberrant T-cell responses in Chagas disease. Trends Parasitol. 21 (2005) 237-243
-
(2005)
Trends Parasitol.
, vol.21
, pp. 237-243
-
-
DosReis, G.A.1
-
13
-
-
0031670153
-
Nitric oxide-induced apoptotic cell death in the acute phase of Trypanosoma cruzi infection in mice
-
Martins G.A., et al. Nitric oxide-induced apoptotic cell death in the acute phase of Trypanosoma cruzi infection in mice. Immunol. Lett. 63 (1998) 113-120
-
(1998)
Immunol. Lett.
, vol.63
, pp. 113-120
-
-
Martins, G.A.1
-
14
-
-
0035012573
-
Fas-FasL interaction modulates nitric oxide production in Trypanosoma cruzi-infected mice
-
Martins G.A., et al. Fas-FasL interaction modulates nitric oxide production in Trypanosoma cruzi-infected mice. Immunology 103 (2001) 122-129
-
(2001)
Immunology
, vol.103
, pp. 122-129
-
-
Martins, G.A.1
-
15
-
-
0029935407
-
Trypanosoma cruzi-induced immunosuppression: selective triggering of CD4+ T-cell death by the T-cell receptor-CD3 pathway and not by the CD69 or Ly-6 activation pathway
-
Lopes M.F., and DosReis G.A. Trypanosoma cruzi-induced immunosuppression: selective triggering of CD4+ T-cell death by the T-cell receptor-CD3 pathway and not by the CD69 or Ly-6 activation pathway. Infect. Immun. 64 (1996) 1559-1564
-
(1996)
Infect. Immun.
, vol.64
, pp. 1559-1564
-
-
Lopes, M.F.1
DosReis, G.A.2
-
16
-
-
0033782882
-
The macrophage haunted by cell ghosts: a pathogen grows
-
Lopes M.F., et al. The macrophage haunted by cell ghosts: a pathogen grows. Immunol. Today 21 (2000) 489-494
-
(2000)
Immunol. Today
, vol.21
, pp. 489-494
-
-
Lopes, M.F.1
-
17
-
-
0031454570
-
Immunosuppressive effects of apoptotic cells
-
Voll R.E., et al. Immunosuppressive effects of apoptotic cells. Nature 390 (1997) 350-351
-
(1997)
Nature
, vol.390
, pp. 350-351
-
-
Voll, R.E.1
-
18
-
-
0032519925
-
Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-β, PGE2, and PAF
-
Fadok V.A., et al. Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-β, PGE2, and PAF. J. Clin. Invest. 101 (1998) 890-898
-
(1998)
J. Clin. Invest.
, vol.101
, pp. 890-898
-
-
Fadok, V.A.1
-
19
-
-
0031939406
-
+ T lymphocytes from infected hosts
-
+ T lymphocytes from infected hosts. J. Immunol. 160 (1998) 1313-1319
-
(1998)
J. Immunol.
, vol.160
, pp. 1313-1319
-
-
Nunes, M.P.1
-
20
-
-
0034642522
-
Uptake of apoptotic cells drives the growth of a pathogenic trypanosome in macrophages
-
Freire-de-Lima C.G., et al. Uptake of apoptotic cells drives the growth of a pathogenic trypanosome in macrophages. Nature 403 (2000) 199-203
-
(2000)
Nature
, vol.403
, pp. 199-203
-
-
Freire-de-Lima, C.G.1
-
21
-
-
34250323002
-
Unraveling the lethal synergism between Trypanosoma cruzi infection and LPS: a role for increased macrophage reactivity
-
Paiva C.N., et al. Unraveling the lethal synergism between Trypanosoma cruzi infection and LPS: a role for increased macrophage reactivity. Eur. J. Immunol. 37 (2007) 1355-1364
-
(2007)
Eur. J. Immunol.
, vol.37
, pp. 1355-1364
-
-
Paiva, C.N.1
-
22
-
-
0034770842
-
Trypanosoma cruzi: peripheral blood monocytes and heart macrophages in the resistance to acute experimental infection in rats
-
Melo R.C., and Machado C.R. Trypanosoma cruzi: peripheral blood monocytes and heart macrophages in the resistance to acute experimental infection in rats. Exp. Parasitol. 97 (2001) 15-23
-
(2001)
Exp. Parasitol.
, vol.97
, pp. 15-23
-
-
Melo, R.C.1
Machado, C.R.2
-
23
-
-
12144285915
-
Macrophage interactions with neutrophils regulate Leishmania major infection
-
Ribeiro-Gomes F.L., et al. Macrophage interactions with neutrophils regulate Leishmania major infection. J. Immunol. 172 (2004) 4454-4462
-
(2004)
J. Immunol.
, vol.172
, pp. 4454-4462
-
-
Ribeiro-Gomes, F.L.1
-
24
-
-
0038702580
-
Reciprocal expression of TRAIL and CD95L in Th1 and Th2 cells: role of apoptosis in T helper subset differentiation
-
Zhang X.R., et al. Reciprocal expression of TRAIL and CD95L in Th1 and Th2 cells: role of apoptosis in T helper subset differentiation. Cell Death Differ. 10 (2003) 203-210
-
(2003)
Cell Death Differ.
, vol.10
, pp. 203-210
-
-
Zhang, X.R.1
-
25
-
-
14544288632
-
+ T-cell memory via TRAIL-mediated activation-induced cell death
-
+ T-cell memory via TRAIL-mediated activation-induced cell death. Nature 434 (2005) 88-93
-
(2005)
Nature
, vol.434
, pp. 88-93
-
-
Janssen, E.M.1
-
26
-
-
0029112253
-
Induction of apoptosis in mature T cells by tumour necrosis factor
-
Zheng L., et al. Induction of apoptosis in mature T cells by tumour necrosis factor. Nature 377 (1995) 348-351
-
(1995)
Nature
, vol.377
, pp. 348-351
-
-
Zheng, L.1
-
27
-
-
0036069819
-
Activated T cell death in vivo mediated by proapoptotic bcl-2 family member bim
-
Hildeman D.A., et al. Activated T cell death in vivo mediated by proapoptotic bcl-2 family member bim. Immunity 16 (2002) 759-767
-
(2002)
Immunity
, vol.16
, pp. 759-767
-
-
Hildeman, D.A.1
-
28
-
-
33845636846
-
Withdrawal symptoms on display: Bcl-2 members under investigation
-
Alves N.L., et al. Withdrawal symptoms on display: Bcl-2 members under investigation. Trends Immunol. 28 (2007) 26-32
-
(2007)
Trends Immunol.
, vol.28
, pp. 26-32
-
-
Alves, N.L.1
-
29
-
-
0032921484
-
Increased susceptibility of Fas ligand-deficient gld mice to Trypanosoma cruzi infection due to a Th2-biased host immune response
-
Lopes M.F., et al. Increased susceptibility of Fas ligand-deficient gld mice to Trypanosoma cruzi infection due to a Th2-biased host immune response. Eur. J. Immunol. 29 (1999) 81-89
-
(1999)
Eur. J. Immunol.
, vol.29
, pp. 81-89
-
-
Lopes, M.F.1
-
30
-
-
33947675823
-
The Fas death pathway controls coordinated expansions of type 1 CD8 and type 2 CD4 T cells in Trypanosoma cruzi infection
-
Guillermo L.V., et al. The Fas death pathway controls coordinated expansions of type 1 CD8 and type 2 CD4 T cells in Trypanosoma cruzi infection. J. Leukoc. Biol. 81 (2007) 942-951
-
(2007)
J. Leukoc. Biol.
, vol.81
, pp. 942-951
-
-
Guillermo, L.V.1
-
31
-
-
2942746419
-
Caspase activation: revisiting the induced proximity model
-
Shi Y. Caspase activation: revisiting the induced proximity model. Cell 117 (2004) 855-858
-
(2004)
Cell
, vol.117
, pp. 855-858
-
-
Shi, Y.1
-
32
-
-
20844446810
-
Caspase-8 activity prevents type 2 cytokine responses and is required for protective T cell-mediated immunity against Trypanosoma cruzi infection
-
Silva E.M., et al. Caspase-8 activity prevents type 2 cytokine responses and is required for protective T cell-mediated immunity against Trypanosoma cruzi infection. J. Immunol. 174 (2005) 6314-6321
-
(2005)
J. Immunol.
, vol.174
, pp. 6314-6321
-
-
Silva, E.M.1
-
33
-
-
34147115539
-
Caspase inhibition reduces lymphocyte apoptosis and improves host immune responses to Trypanosoma cruzi infection
-
Silva E.M., et al. Caspase inhibition reduces lymphocyte apoptosis and improves host immune responses to Trypanosoma cruzi infection. Eur. J. Immunol. 37 (2007) 738-746
-
(2007)
Eur. J. Immunol.
, vol.37
, pp. 738-746
-
-
Silva, E.M.1
-
34
-
-
0032791044
-
Gamma interferon modulates CD95 (Fas) and CD95 ligand (Fas-L) expression and nitric oxide-induced apoptosis during the acute phase of Trypanosoma cruzi infection: a possible role in immune response control
-
Martins G.A., et al. Gamma interferon modulates CD95 (Fas) and CD95 ligand (Fas-L) expression and nitric oxide-induced apoptosis during the acute phase of Trypanosoma cruzi infection: a possible role in immune response control. Infect. Immun. 67 (1999) 3864-3871
-
(1999)
Infect. Immun.
, vol.67
, pp. 3864-3871
-
-
Martins, G.A.1
-
35
-
-
0028807425
-
Tumor necrosis factor alpha mediates resistance to Trypanosoma cruzi infection in mice by inducing nitric oxide production in infected gamma interferon-activated macrophages
-
Silva J.S., et al. Tumor necrosis factor alpha mediates resistance to Trypanosoma cruzi infection in mice by inducing nitric oxide production in infected gamma interferon-activated macrophages. Infect. Immun. 63 (1995) 4862-4867
-
(1995)
Infect. Immun.
, vol.63
, pp. 4862-4867
-
-
Silva, J.S.1
-
36
-
-
0037093846
-
Costimulation of host T lymphocytes by a trypanosomal trans-sialidase: involvement of CD43 signaling
-
Todeschini A.R., et al. Costimulation of host T lymphocytes by a trypanosomal trans-sialidase: involvement of CD43 signaling. J. Immunol. 168 (2002) 5192-5198
-
(2002)
J. Immunol.
, vol.168
, pp. 5192-5198
-
-
Todeschini, A.R.1
-
37
-
-
0033394463
-
Trans-sialidase from Trypanosoma cruzi induces apoptosis in cells from the immune system in vivo
-
Leguizamon M.S., et al. Trans-sialidase from Trypanosoma cruzi induces apoptosis in cells from the immune system in vivo. J. Infect. Dis. 180 (1999) 1398-1402
-
(1999)
J. Infect. Dis.
, vol.180
, pp. 1398-1402
-
-
Leguizamon, M.S.1
-
38
-
-
0037133708
-
Thymocyte depletion in Trypanosoma cruzi infection is mediated by trans-sialidase-induced apoptosis on nurse cells complex
-
Mucci J., et al. Thymocyte depletion in Trypanosoma cruzi infection is mediated by trans-sialidase-induced apoptosis on nurse cells complex. Proc. Natl. Acad. Sci. U. S. A. 99 (2002) 3896-3901
-
(2002)
Proc. Natl. Acad. Sci. U. S. A.
, vol.99
, pp. 3896-3901
-
-
Mucci, J.1
-
39
-
-
26444434342
-
Pharmacological manipulation of cell death: clinical applications in sight?
-
Green D.R., and Kroemer G. Pharmacological manipulation of cell death: clinical applications in sight?. J. Clin. Invest. 115 (2005) 2610-2617
-
(2005)
J. Clin. Invest.
, vol.115
, pp. 2610-2617
-
-
Green, D.R.1
Kroemer, G.2
-
40
-
-
26444560960
-
Caspases: pharmacological manipulation of cell death
-
Lavrik I.N., et al. Caspases: pharmacological manipulation of cell death. J. Clin. Invest. 115 (2005) 2665-2672
-
(2005)
J. Clin. Invest.
, vol.115
, pp. 2665-2672
-
-
Lavrik, I.N.1
-
41
-
-
0033431682
-
Prevention of lymphocyte cell death in sepsis improves survival in mice
-
Hotchkiss R.S., et al. Prevention of lymphocyte cell death in sepsis improves survival in mice. Proc. Natl. Acad. Sci. U. S. A. 96 (1999) 14541-14546
-
(1999)
Proc. Natl. Acad. Sci. U. S. A.
, vol.96
, pp. 14541-14546
-
-
Hotchkiss, R.S.1
-
42
-
-
5944251623
-
Caspase inhibitors improve survival in sepsis: a critical role of the lymphocyte
-
Hotchkiss R.S., et al. Caspase inhibitors improve survival in sepsis: a critical role of the lymphocyte. Nat. Immunol. 1 (2000) 496-501
-
(2000)
Nat. Immunol.
, vol.1
, pp. 496-501
-
-
Hotchkiss, R.S.1
-
43
-
-
33646882912
-
Suppression of alveolar macrophage apoptosis prolongs survival of rats and mice with pneumocystis pneumonia
-
Lasbury M.E., et al. Suppression of alveolar macrophage apoptosis prolongs survival of rats and mice with pneumocystis pneumonia. J. Immunol. 176 (2006) 6443-6453
-
(2006)
J. Immunol.
, vol.176
, pp. 6443-6453
-
-
Lasbury, M.E.1
-
44
-
-
0038312126
-
Adoptive transfer of apoptotic splenocytes worsens survival, whereas adoptive transfer of necrotic splenocytes improves survival in sepsis
-
Hotchkiss R.S., et al. Adoptive transfer of apoptotic splenocytes worsens survival, whereas adoptive transfer of necrotic splenocytes improves survival in sepsis. Proc. Natl. Acad. Sci. U. S. A. 100 (2003) 6724-6729
-
(2003)
Proc. Natl. Acad. Sci. U. S. A.
, vol.100
, pp. 6724-6729
-
-
Hotchkiss, R.S.1
-
45
-
-
2442467813
-
+ T cell memory
-
+ T cell memory. J. Immunol. 172 (2004) 6313-6323
-
(2004)
J. Immunol.
, vol.172
, pp. 6313-6323
-
-
Wu, Z.1
-
46
-
-
20044368626
-
Requirement for caspase-8 in NF-κB activation by antigen receptor
-
Su H., et al. Requirement for caspase-8 in NF-κB activation by antigen receptor. Science 307 (2005) 1465-1468
-
(2005)
Science
, vol.307
, pp. 1465-1468
-
-
Su, H.1
-
47
-
-
18544383460
-
Pleiotropic defects in lymphocyte activation caused by caspase-8 mutations lead to human immunodeficiency
-
Chun H.J., et al. Pleiotropic defects in lymphocyte activation caused by caspase-8 mutations lead to human immunodeficiency. Nature 419 (2002) 395-399
-
(2002)
Nature
, vol.419
, pp. 395-399
-
-
Chun, H.J.1
-
48
-
-
0033378016
-
Early activation of caspases during T lymphocyte stimulation results in selective substrate cleavage in nonapoptotic cells
-
Alam A., et al. Early activation of caspases during T lymphocyte stimulation results in selective substrate cleavage in nonapoptotic cells. J. Exp. Med. 190 (1999) 1879-1890
-
(1999)
J. Exp. Med.
, vol.190
, pp. 1879-1890
-
-
Alam, A.1
-
49
-
-
0033386808
-
Caspase activation is required for T cell proliferation
-
Kennedy N.J., et al. Caspase activation is required for T cell proliferation. J. Exp. Med. 190 (1999) 1891-1896
-
(1999)
J. Exp. Med.
, vol.190
, pp. 1891-1896
-
-
Kennedy, N.J.1
-
50
-
-
33947268527
-
Cross-talk between apoptosis and cytokines in the regulation of parasitic infection
-
DosReis G.A., et al. Cross-talk between apoptosis and cytokines in the regulation of parasitic infection. Cytokine Growth Factor Rev. 18 (2007) 97-105
-
(2007)
Cytokine Growth Factor Rev.
, vol.18
, pp. 97-105
-
-
DosReis, G.A.1
-
51
-
-
14844342614
-
A role for caspases in controlling IL-4 expression in T cells
-
Sehra S., et al. A role for caspases in controlling IL-4 expression in T cells. J. Immunol. 174 (2005) 3440-3446
-
(2005)
J. Immunol.
, vol.174
, pp. 3440-3446
-
-
Sehra, S.1
-
52
-
-
26444523510
-
Apoptosis modulates protective immunity to the pathogenic fungus Histoplasma capsulatum
-
Allen H.L., and Deepe Jr. G.S. Apoptosis modulates protective immunity to the pathogenic fungus Histoplasma capsulatum. J. Clin. Invest. 115 (2005) 2875-2885
-
(2005)
J. Clin. Invest.
, vol.115
, pp. 2875-2885
-
-
Allen, H.L.1
Deepe Jr., G.S.2
-
53
-
-
0028172588
-
Differential ability of Th1 and Th2 T cells to express Fas ligand and to undergo activation-induced cell death
-
Ramsdell F., et al. Differential ability of Th1 and Th2 T cells to express Fas ligand and to undergo activation-induced cell death. Int. Immunol. 6 (1994) 1545-1553
-
(1994)
Int. Immunol.
, vol.6
, pp. 1545-1553
-
-
Ramsdell, F.1
-
54
-
-
0030970526
-
Unequal death in T helper cell (Th)1 and Th2 effectors: Th1, but not Th2, effectors undergo rapid Fas/FasL-mediated apoptosis
-
Zhang X., et al. Unequal death in T helper cell (Th)1 and Th2 effectors: Th1, but not Th2, effectors undergo rapid Fas/FasL-mediated apoptosis. J. Exp. Med. 185 (1997) 1837-1849
-
(1997)
J. Exp. Med.
, vol.185
, pp. 1837-1849
-
-
Zhang, X.1
-
55
-
-
0031563740
-
Activation-induced T cell death: resistance or susceptibility correlate with cell surface fas ligand expression and T helper phenotype
-
Oberg H.H., et al. Activation-induced T cell death: resistance or susceptibility correlate with cell surface fas ligand expression and T helper phenotype. Cell. Immunol. 181 (1997) 93-100
-
(1997)
Cell. Immunol.
, vol.181
, pp. 93-100
-
-
Oberg, H.H.1
-
56
-
-
0032711440
-
Selective up-regulation of phosphatidylinositol 3′-kinase activity in Th2 cells inhibits caspase-8 cleavage at the death-inducing complex: a mechanism for Th2 resistance from Fas-mediated apoptosis
-
Varadhachary A.S., et al. Selective up-regulation of phosphatidylinositol 3′-kinase activity in Th2 cells inhibits caspase-8 cleavage at the death-inducing complex: a mechanism for Th2 resistance from Fas-mediated apoptosis. J. Immunol. 163 (1999) 4772-4779
-
(1999)
J. Immunol.
, vol.163
, pp. 4772-4779
-
-
Varadhachary, A.S.1
-
57
-
-
33747144028
-
Granzyme B is critical for T cell receptor-induced cell death of type 2 helper T cells
-
Devadas S., et al. Granzyme B is critical for T cell receptor-induced cell death of type 2 helper T cells. Immunity 25 (2006) 237-247
-
(2006)
Immunity
, vol.25
, pp. 237-247
-
-
Devadas, S.1
-
58
-
-
0035057934
-
CD95 is required for the early control of parasite burden in the liver of Leishmania donovani-infected mice
-
Alexander C.E., et al. CD95 is required for the early control of parasite burden in the liver of Leishmania donovani-infected mice. Eur. J. Immunol. 31 (2001) 1199-1210
-
(2001)
Eur. J. Immunol.
, vol.31
, pp. 1199-1210
-
-
Alexander, C.E.1
-
59
-
-
0032063360
-
Mice defective in Fas are highly susceptible to Leishmania major infection despite elevated IL-12 synthesis, strong Th1 responses, and enhanced nitric oxide production
-
Huang F.P., et al. Mice defective in Fas are highly susceptible to Leishmania major infection despite elevated IL-12 synthesis, strong Th1 responses, and enhanced nitric oxide production. J. Immunol. 160 (1998) 4143-4147
-
(1998)
J. Immunol.
, vol.160
, pp. 4143-4147
-
-
Huang, F.P.1
-
60
-
-
0030737995
-
Th1 and Th2 subsets equally undergo Fas-dependent and -independent activation-induced cell death
-
Watanabe N., et al. Th1 and Th2 subsets equally undergo Fas-dependent and -independent activation-induced cell death. Eur. J. Immunol. 27 (1997) 1858-1864
-
(1997)
Eur. J. Immunol.
, vol.27
, pp. 1858-1864
-
-
Watanabe, N.1
-
61
-
-
0030745157
-
Cell-mediated immunity in experimental Trypanosoma cruzi infection
-
DosReis G.A. Cell-mediated immunity in experimental Trypanosoma cruzi infection. Parasitol. Today 13 (1997) 335-342
-
(1997)
Parasitol. Today
, vol.13
, pp. 335-342
-
-
DosReis, G.A.1
-
62
-
-
33645513235
-
+ cytotoxic T cells after infection with Trypanosoma cruzi in naive or vaccinated mice
-
+ cytotoxic T cells after infection with Trypanosoma cruzi in naive or vaccinated mice. Infect. Immun. 74 (2006) 2477-2481
-
(2006)
Infect. Immun.
, vol.74
, pp. 2477-2481
-
-
Tzelepis, F.1
-
63
-
-
4844220220
-
+ T cells in Trypanosoma cruzi infection
-
+ T cells in Trypanosoma cruzi infection. Immunol. Rev. 201 (2004) 304-317
-
(2004)
Immunol. Rev.
, vol.201
, pp. 304-317
-
-
Martin, D.1
Tarleton, R.2
-
64
-
-
0028959774
-
Cytokine gene expression during infection of mice lacking CD4 and/or CD8 with Trypanosoma cruzi
-
Rottenberg M.E., et al. Cytokine gene expression during infection of mice lacking CD4 and/or CD8 with Trypanosoma cruzi. Scand. J. Immunol. 41 (1995) 164-170
-
(1995)
Scand. J. Immunol.
, vol.41
, pp. 164-170
-
-
Rottenberg, M.E.1
-
65
-
-
0036896423
-
+ T-cell expansion and promotes resistance to reinfection
-
+ T-cell expansion and promotes resistance to reinfection. Antimicrob. Agents Chemother. 46 (2002) 3790-3796
-
(2002)
Antimicrob. Agents Chemother.
, vol.46
, pp. 3790-3796
-
-
Olivieri, B.P.1
-
66
-
-
28044463623
-
Metacaspases of Trypanosoma cruzi: possible candidates for programmed cell death mediators
-
Kosec G., et al. Metacaspases of Trypanosoma cruzi: possible candidates for programmed cell death mediators. Mol. Biochem. Parasitol. 145 (2006) 18-28
-
(2006)
Mol. Biochem. Parasitol.
, vol.145
, pp. 18-28
-
-
Kosec, G.1
-
67
-
-
2642613665
-
The resolution of lesions induced by Leishmania major in mice requires a functional Fas (APO-1, CD95) pathway of cytotoxicity
-
Conceicao-Silva F., et al. The resolution of lesions induced by Leishmania major in mice requires a functional Fas (APO-1, CD95) pathway of cytotoxicity. Eur. J. Immunol. 28 (1998) 237-245
-
(1998)
Eur. J. Immunol.
, vol.28
, pp. 237-245
-
-
Conceicao-Silva, F.1
-
68
-
-
0037261142
-
Concerted action of perforin and granzymes is critical for the elimination of Trypanosoma cruzi from mouse tissues, but prevention of early host death is in addition dependent on the FasL/Fas pathway
-
Muller U., et al. Concerted action of perforin and granzymes is critical for the elimination of Trypanosoma cruzi from mouse tissues, but prevention of early host death is in addition dependent on the FasL/Fas pathway. Eur. J. Immunol. 33 (2003) 70-78
-
(2003)
Eur. J. Immunol.
, vol.33
, pp. 70-78
-
-
Muller, U.1
|