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Volumn 12, Issue 4, 2007, Pages 1238-1246

Impairment of mitochondrial function by particulate matter (PM) and their toxic components: Implications for PM-induced cardiovascular and lung disease

Author keywords

Apoptosis; Mitochondria; Oxidative stress; Particulate matter (PM); PT pore; Review; ROS

Indexed keywords


EID: 34249783903     PISSN: 27686701     EISSN: 27686698     Source Type: Journal    
DOI: 10.2741/2142     Document Type: Article
Times cited : (119)

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    • Abbreviations: AQ, 9, 10-anthraquinone; ARE, antioxidant response element; DEP, diesel exhaust particles; GSH, glutathione; GSSG, glutathione disulfide; JNK, NH2 terminal Jun kinase; HO-1, heme oxygenase 1; MAPK, mitogen-activated protein kinase; NAC, N-acetylcysteine; NQ, 1,2-naphthaquinone; PQ, 9, 10-phenanthraquinone; O2, Superoxide; "OH, hydroxyl radical; PAH, polycyclic aromatic hydrocarbons; PM, particle matter; PT, permeability transition; ROS, reactive oxygen species; SERCA, Sarco/endoplasmic reticulum Ca2+-ATPase, SOD, Superoxide dismutase; UFP, ultrafine particles; delta-Psim, mitochondrial membrane potential
    • m, mitochondrial membrane potential.


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.