Ras induces chromosome instability and abrogation of the DNA damage response

Cancer Research

Volumn 66, Issue 21, 2006, Pages 10505-10512

  Abulaiti, Adili ^a   Fikaris, Aphrothiti J ^a   Tsygankova, Oxana M ^a   Meinkoth, Judy L ^a  

^a UNIVERSITY OF PENNSYLVANIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ATR PROTEIN; CHECKPOINT KINASE 1; ETOPOSIDE; HISTONE H2AX; HORMONE; MARKER; MUTANT PROTEIN; PROTEIN P53; RAS PROTEIN;

ANCHORAGE INDEPENDENT GROWTH; ANIMAL CELL; APOPTOSIS; ARTICLE; CELL DIFFERENTIATION; CELL DIVISION; CELL POPULATION; CELL PROLIFERATION; CELL STRUCTURE; CELL SURVIVAL; CELL TRANSFORMATION; CHROMOSOMAL INSTABILITY; CHROMOSOME DAMAGE; CHROMOSOME SEGREGATION; CONTROLLED STUDY; DNA DAMAGE; DOSE RESPONSE; ENDOCRINE DISEASE; ENZYME ACTIVITY; MICRONUCLEUS; MITOGENESIS; MITOSIS; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; RAT; THYROID CELL; THYROID TUMOR;

ANIMALS; APOPTOSIS; CELL CYCLE; CELL CYCLE PROTEINS; CELL TRANSFORMATION, NEOPLASTIC; CHROMOSOMAL INSTABILITY; DNA DAMAGE; DNA REPAIR; GENES, RAS; HISTONES; PROTEIN-SERINE-THREONINE KINASES; RATS; RATS, WISTAR; THYROID NEOPLASMS;

EID: 33751278260     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: 10.1158/0008-5472.CAN-06-2351     Document Type: Article

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