How do Bax and Bak lead to permeabilization of the outer mitochondrial membrane?

Current Opinion in Cell Biology

Volumn 18, Issue 6, 2006, Pages 685-689

  Antignani, Antonella ^a   Youle, Richard J ^a  

^a NATIONAL INSTITUTE OF NEUROLOGICAL DISORDERS AND STROKE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DIPHTHERIA TOXIN; PROTEIN BAK; PROTEIN BAX; PROTEIN BCL 2;

APOPTOSIS; CELL DEATH; CELL PERMEABILIZATION; CELLULAR DISTRIBUTION; HUMAN; LIPID BILAYER; MITOCHONDRIAL MEMBRANE; MORPHOGENESIS; NONHUMAN; OUTER MEMBRANE; PRIORITY JOURNAL; PROTEIN FUNCTION; REVIEW; WORM;

ANIMALS; APOPTOSIS; BCL-2 HOMOLOGOUS ANTAGONIST-KILLER PROTEIN; BCL-2-ASSOCIATED X PROTEIN; CAENORHABDITIS ELEGANS PROTEINS; CELL MEMBRANE PERMEABILITY; DROSOPHILA PROTEINS; HUMANS; MITOCHONDRIA; MITOCHONDRIAL MEMBRANES; MODELS, BIOLOGICAL;

MAMMALIA;

EID: 33750619845     PISSN: 09550674     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.ceb.2006.10.004     Document Type: Review

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