Severe disease, unaltered leukocyte migration, and reduced IFN-γ production in CXCR3-/- mice with experimental autoimmune encephalomyelitis

Journal of Immunology

Volumn 176, Issue 7, 2006, Pages 4399-4409

  Liu, Liping ^a   Huang, DeRen ^a   Matsui, Masaru ^a,d   He, Toby T ^a   Hu, Taofang ^a   DeMartino, Julie ^b   Lu, Bao ^c   Gerard, Craig ^c   Ransohoff, Richard M ^a  

^a LERNER RESEARCH INSTITUTE   (United States)

^b MERCK RESEARCH LABORATORIES   (United States)

^c HARVARD MEDICAL SCHOOL   (United States)

^d KANAZAWA MEDICAL UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

CHEMOKINE; CHEMOKINE RECEPTOR CXCR3; CXCL9 CHEMOKINE; GAMMA INTERFERON; GAMMA INTERFERON INDUCIBLE PROTEIN 10; GAMMA INTERFERON INDUCIBLE PROTEIN 11; LIGAND; MYELIN OLIGODENDROCYTE GLYCOPROTEIN; UNCLASSIFIED DRUG; VON WILLEBRAND FACTOR; ANTIBODY; CHEMOKINE RECEPTOR; CXC CHEMOKINE RECEPTOR 3; GLYCOPROTEIN; INDUCIBLE NITRIC OXIDE SYNTHASE; MYELIN OLIGODENDROCYTE GLYCOPROTEIN (35 55); MYELIN OLIGODENDROCYTE GLYCOPROTEIN (35-55); PEPTIDE FRAGMENT; PROTEOLIPID PROTEIN; PROTEOLIPID PROTEIN 139 151; PROTEOLIPID PROTEIN 139-151;

ALLERGIC ENCEPHALOMYELITIS; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BLOOD BRAIN BARRIER; CELL ACTIVATION; CELL INFILTRATION; CONTROLLED STUDY; CYTOKINE PRODUCTION; DISEASE SEVERITY; LEUKOCYTE MIGRATION; LYMPH NODE CELL; MOUSE; MULTIPLE SCLEROSIS; MYELITIS; NONHUMAN; PRIORITY JOURNAL; QUALITATIVE ANALYSIS; QUANTITATIVE ANALYSIS; TH1 CELL; ANIMAL; BIOSYNTHESIS; C57BL MOUSE; CELL PROLIFERATION; CYTOLOGY; DISEASE COURSE; DRUG EFFECT; GENETICS; IMMUNOLOGY; METABOLISM; MOUSE MUTANT; NEUTROPHIL CHEMOTAXIS; PATHOLOGY; PERMEABILITY; T LYMPHOCYTE;

ANIMALS; ANTIBODIES; BLOOD-BRAIN BARRIER; CELL PROLIFERATION; CHEMOTAXIS, LEUKOCYTE; DISEASE PROGRESSION; ENCEPHALOMYELITIS, AUTOIMMUNE, EXPERIMENTAL; GLYCOPROTEINS; INTERFERON TYPE II; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MYELIN PROTEOLIPID PROTEIN; NITRIC OXIDE SYNTHASE TYPE II; PEPTIDE FRAGMENTS; PERMEABILITY; RECEPTORS, CHEMOKINE; T-LYMPHOCYTES;

EID: 33646029748     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.176.7.4399     Document Type: Article

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