Pathways that elicit long-term changes in gene expression in nociceptive neurons following nerve injury: Contributions to neuropathic pain

Neurological Research

Volumn 26, Issue 2, 2004, Pages 195-203

  Sung, Ying Ju ^a   Ambron, Richard T ^a  

^a Columbia University ^*  (United States)

Author keywords

CREB; Dorsal root ganglion; Gene expression; Long term hyperexcitability; MAPK; Positive injury signals; Primary nociceptors; Protein kinase G

Indexed keywords

1H 1,2,4 OXADIAZOLO[4,3 A]QUINOXALIN 1 ONE; CYCLIC AMP DEPENDENT PROTEIN KINASE; CYCLIC AMP RESPONSIVE ELEMENT BINDING PROTEIN; CYCLIC GMP DEPENDENT PROTEIN KINASE; CYTOKINE; GUANYLATE CYCLASE INHIBITOR; MESSENGER RNA; MITOGEN ACTIVATED PROTEIN KINASE; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; SEROTONIN;

ANIMAL MODEL; CHRONIC PAIN; GENE EXPRESSION; GENETIC TRANSCRIPTION; INFLAMMATION; NERVE CELL EXCITABILITY; NERVE INJURY; NEUROPATHIC PAIN; NOCICEPTION; NONHUMAN; PAIN; PERIPHERAL NERVE INJURY; RAT; REVIEW; SPINAL GANGLION;

ANIMALS; CHRONIC DISEASE; GENE EXPRESSION REGULATION; HUMANS; LONG-TERM POTENTIATION; NEURONS; PAIN; PERIPHERAL NERVOUS SYSTEM DISEASES; SIGNAL TRANSDUCTION; TIME;

EID: 1842450332     PISSN: 01616412     EISSN: None     Source Type: Journal    
DOI: 10.1179/016164104225013761     Document Type: Review

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