Inflammation causes a long-term hyperexcitability in the nociceptive sensory neurons of Aplysia

Learning and Memory

Volumn 6, Issue 3, 1999, Pages 331-340

  Farr, Maryjane ^a   Mathews, Jenny ^a   Zhu, De Fen ^a   Ambron, Richard T ^a  

^a Och Spine at New York Presbyterian Hospitals   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

INTERLEUKIN 6; PROTEIN KINASE C; RECOMBINANT TRANSFORMING GROWTH FACTOR BETA; SEROTONIN; TRANSFORMING GROWTH FACTOR BETA1;

ACTION POTENTIAL; ANIMAL CELL; APLYSIA; ARTICLE; BLOOD CELL; EXCITABILITY; FOREIGN BODY REACTION; HYPERALGESIA; IMMUNOBLOTTING; IMMUNOCYTOCHEMISTRY; INFLAMMATION; NERVE INJURY; NERVOUS SYSTEM; NOCICEPTION; NONHUMAN; PRIORITY JOURNAL; SENSORY NERVE CELL; TISSUE INJURY;

ANIMALS; APLYSIA; AXONAL TRANSPORT; CELLS, CULTURED; ELECTROPHYSIOLOGY; HEMOCYTES; HUMANS; IMMUNOHISTOCHEMISTRY; INFLAMMATION; NEURONS, AFFERENT; NOCICEPTORS; PEPTIDES; PROTEIN KINASE C; RECOMBINANT PROTEINS; TRANSFORMING GROWTH FACTOR BETA;

EID: 0032877007     PISSN: 10720502     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (2)

1. Ambron, R.T. and E.T. Walters. 1996. Priming events and retrograde injury signals: A new perspective on the cellular and molecular biology of nerve regeneration. Mol. Neurobiol. 13: 61-79.
2. Ambron, R.T., X.P. Zhang, J.D. Gunstream, M. Povelones, and E.T. Walters. 1996. Intrinsic injury signals enhance growth, survival and excitability of Aplysia neurons. J. Neurosci. 16: 7469-7477.