Genistein ameliorates β-amyloid peptide (25-35)-induced hippocampal neuronal apoptosis

Free Radical Biology and Medicine

Volumn 36, Issue 2, 2004, Pages 180-188

  Zeng, Haiyan ^a   Chen, Qi ^b   Zhao, Baolu ^a  

^a INSTITUTE OF BIOPHYSICS   (China)

^b SALK INSTITUTE FOR BIOLOGICAL STUDIES   (United States)

Author keywords

Antioxidation; Apoptosis; Caspase 3; Estrogen receptor; Free radicals; Genistein; Hippocampal neuronal cells; Intracellular free Ca2+; Amyloid protein

Indexed keywords

AMYLOID BETA PROTEIN; ANTIOXIDANT; CALCIUM ION; CASPASE 3; DNA FRAGMENT; ESTROGEN RECEPTOR; GENISTEIN; PEPTIDE; REACTIVE OXYGEN METABOLITE;

ANIMAL CELL; ANIMAL TISSUE; ANTIOXIDANT ACTIVITY; APOPTOSIS; ARTICLE; ASSOCIATION; BRAIN NERVE CELL; CALCIUM CELL LEVEL; CELL CULTURE; CELL DAMAGE; CELL VIABILITY; CONCENTRATION RESPONSE; CONTROLLED STUDY; DRUG EFFECT; ENZYME ACTIVATION; HIPPOCAMPUS; NEUROPROTECTION; NEWBORN; NONHUMAN; PHENOTYPE; POLYMERIZATION; PRIORITY JOURNAL; PROTEIN LOCALIZATION; SIGNAL TRANSDUCTION;

EID: 1642464725     PISSN: 08915849     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.freeradbiomed.2003.10.018     Document Type: Article

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