β-Amyloid-induced calcium influx induces apoptosis in culture by oxidative stress rather than tau phosphorylation

Molecular Brain Research

Volumn 76, Issue 2, 2000, Pages 389-395

  Ekinci, Fatma J ^a   Linsley, Maria Dawn ^a   Shea, Thomas B ^a  

^a University of Massachusetts Lowell   (United States)

Author keywords

Amyloid; Alzheimer's disease; Apoptosis; Calcium influx; Neurodegeneration; Oxidative stress; Tau phosphorylation

Indexed keywords

ALPHA TOCOPHEROL; AMYLOID BETA PROTEIN; CALCIUM; N (6 AMINOHEXYL) 5 CHLORO 1 NAPHTHALENESULFONAMIDE; REACTIVE OXYGEN METABOLITE; TAU PROTEIN;

ALZHEIMER DISEASE; APOPTOSIS; ARTICLE; CALCIUM TRANSPORT; CELL CULTURE; DENSITOMETRY; IMMUNOFLUORESCENCE; NERVE DEGENERATION; NEUROBLASTOMA CELL; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION;

AMYLOID BETA-PROTEIN; ANTIOXIDANTS; APOPTOSIS; CALCIUM; CHELATING AGENTS; CULTURE MEDIA, SERUM-FREE; EGTAZIC ACID; ENZYME INHIBITORS; HUMANS; KINETICS; NEUROBLASTOMA; OXIDATIVE STRESS; PEPTIDE FRAGMENTS; PHOSPHORYLATION; REACTIVE OXYGEN SPECIES; SULFONAMIDES; TAU PROTEINS; TUMOR CELLS, CULTURED; VITAMIN E;

EID: 0034728670     PISSN: 0169328X     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0169-328X(00)00025-5     Document Type: Article

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