Site and mechanism of leptin action in a rodent form of congenital lipodystrophy

Journal of Clinical Investigation

Volumn 113, Issue 3, 2004, Pages 414-424

  Asilmaz, Esra ^a   Cohen, Paul ^a   Miyazaki, Makoto ^b   Dobrzyn, Pawel ^b   Ueki, Kohjiro ^c   Fayzikhodjaeva, Gulnorakhon ^a   Soukas, Alexander A ^a   Kahn, C Ronald ^c   Ntambi, James M ^b   Socci, Nicholas D ^d   Friedman, Jeffrey M ^a  

^a ROCKEFELLER UNIVERSITY   (United States)

^b UNIVERSITY OF WISCONSIN MADISON   (United States)

^c HARVARD MEDICAL SCHOOL   (United States)

^d ALBERT EINSTEIN COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ACYL COENZYME A DESATURASE; INSULIN RECEPTOR SUBSTRATE 2; LEPTIN; TRANSCRIPTION FACTOR;

ADIPOSE TISSUE; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; ENERGY BALANCE; ENZYME ACTIVITY; FATTY LIVER; GENE DELETION; HYPERGLYCEMIA; HYPERINSULINEMIA; HYPOTHALAMUS HYPOPHYSIS SYSTEM; INSULIN RESISTANCE; LIPOATROPHY; LIPODYSTROPHY; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; RAT; SIGNAL TRANSDUCTION; STEATOSIS; ANIMAL; C57BL MOUSE; DISEASE MODEL; FEMALE; GENE EXPRESSION PROFILING; LIVER; MALE; METABOLISM; MOUSE; PATHOLOGY; PHYLOGENY; SKELETAL MUSCLE;

ANIMALS; DISEASE MODELS, ANIMAL; FEMALE; GENE EXPRESSION PROFILING; LEPTIN; LIPODYSTROPHY; LIVER; MALE; MICE; MICE, INBRED C57BL; MUSCLE, SKELETAL; PHYLOGENY; STEAROYL-COA DESATURASE;

EID: 11144357287     PISSN: 00219738     EISSN: None     Source Type: Journal    
DOI: 10.1172/JCI200419511     Document Type: Article

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