JNK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen species

Genes and Development

Volumn 18, Issue 23, 2004, Pages 2905-2915

  Ventura, Juan Jose ^a   Cogswell, Patricia ^b   Flavell, Richard A ^c   Baldwin Jr , Albert S ^b   Davis, Roger J ^a  

^a UNIVERSITY OF MASSACHUSETTS MEDICAL SCHOOL   (United States)

^b UNIVERSITY OF NORTH CAROLINA   (United States)

^c YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Apoptosis; JNK; Necrosis; Reactive oxygen species; TNF

Indexed keywords

REACTIVE OXYGEN METABOLITE; STRESS ACTIVATED PROTEIN KINASE; TUMOR NECROSIS FACTOR;

ANIMAL CELL; ARTICLE; CELL DEATH; CELL ISOLATION; CELL SURVIVAL; CONTROLLED STUDY; EMBRYO; FIBROBLAST; GROWTH INHIBITION; MOUSE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN FUNCTION; WILD TYPE;

ANIMALS; CELL DEATH; JNK MITOGEN-ACTIVATED PROTEIN KINASES; MAP KINASE KINASE 4; MICE; MITOGEN-ACTIVATED PROTEIN KINASE KINASES; REACTIVE OXYGEN SPECIES; TUMOR NECROSIS FACTOR-ALPHA;

ANIMALIA;

EID: 10044265209     PISSN: 08909369     EISSN: None     Source Type: Journal    
DOI: 10.1101/gad.1223004     Document Type: Article

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