Amyloid precursor protein gene disruption attenuates degeneration of substantia nigra compacta neurons following axotomy

Brain Research

Volumn 938, Issue 1-2, 2002, Pages 38-44

  DeGiorgio, Lorraine A ^a   Shimizu, Yoshinori ^a   Chun, Hong S ^a   Kim, Yoon Seong ^a   Sugama, Shuei ^a   Son, Jin H ^a   Joh, Tong H ^a   Volpe, Bruce T ^a  

^a WEILL CORNELL MEDICAL COLLEGE   (United States)

Author keywords

Amyloid precursor protein; Delayed degeneration; Substantia nigra compacta

Indexed keywords

AMYLOID BETA PROTEIN; AMYLOID PRECURSOR PROTEIN; EPITOPE; TYROSINE 3 MONOOXYGENASE;

AMINO TERMINAL SEQUENCE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CARBOXY TERMINAL SEQUENCE; CELL LOSS; CELL NUCLEUS; CONTROLLED STUDY; CORRELATION ANALYSIS; CYTOSOL; DOPAMINERGIC NERVE CELL; GENE DISRUPTION; IMMUNOREACTIVITY; MEDIAL FOREBRAIN BUNDLE; MORPHOMETRICS; MOUSE; NERVE CELL DEGENERATION; NERVE FIBER TRANSECTION; NEUROPROTECTION; NEUROTOXICITY; NONHUMAN; PRIORITY JOURNAL; PROTEIN DOMAIN; PROTEIN LOCALIZATION; PROTEIN TRANSPORT; SUBSTANTIA NIGRA; VENTRAL TEGMENTUM; WESTERN BLOTTING; WILD TYPE;

AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; AXOTOMY; BLOTTING, WESTERN; CELL NUCLEUS; CYTOSOL; FLUORESCENT ANTIBODY TECHNIQUE; MEDIAL FOREBRAIN BUNDLE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NERVE DEGENERATION; SUBSTANTIA NIGRA; TYROSINE 3-MONOOXYGENASE;

EID: 0037205154     PISSN: 00068993     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0006-8993(02)02483-6     Document Type: Article

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