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Volumn 277, Issue 38, 2002, Pages 35635-35641

STAT3 activation by type I interferons is dependent on specific tyrosines located in the cytoplasmic domain of interferon receptor chain 2c: Activation of multiple STATs proceeds through the redundant usage of two tyrosine residues

(5) Velichko, Sharlene a,b Charis Wagner, T a,b Turkson, James a,b Jove, Richard a,b Croze, Ed a,b

a BAYER AG (Germany)

b H LEE MOFFITT CANCER CENTER AND RESEARCH INSTITUTE (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ACTIVATION ANALYSIS; CARCINOGENS; CELLS; TRANSDUCERS; TUMORS;

PHOSPHORYLATION;

BIOCHEMISTRY;

INTERFERON; INTERFERON RECEPTOR; STAT1 PROTEIN; STAT2 PROTEIN; STAT3 PROTEIN; TYROSINE;

ARTICLE; CANCER; CONTROLLED STUDY; CYTOPLASM; FIBROBLAST; FIBROSARCOMA; GROWTH REGULATION; HUMAN; HUMAN CELL; IMMUNOMODULATION; ONCOGENE; PRIORITY JOURNAL; PROTEIN FUNCTION; SIGNAL TRANSDUCTION;

BASE SEQUENCE; DNA PRIMERS; DNA-BINDING PROTEINS; ELECTROPHORETIC MOBILITY SHIFT ASSAY; HUMANS; INTERFERON TYPE I; MEMBRANE PROTEINS; RECEPTOR, INTERFERON ALPHA-BETA; RECEPTORS, INTERFERON; STAT3 TRANSCRIPTION FACTOR; TRANS-ACTIVATORS; TUMOR CELLS, CULTURED; TYROSINE;

EID: 0037144598 PISSN: 00219258 EISSN: None Source Type: Journal
DOI: 10.1074/jbc.M204578200 Document Type: Article

Times cited : (33)

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