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Volumn 362, Issue 3, 2002, Pages 561-571

Activation of c-Jun N-terminal kinase promotes survival of cardiac myocytes after oxidative stress

Author keywords

Apoptosis; Caspase; Hypoxia; Ischaemia; Reoxygenation

Indexed keywords

ACTIVATION ANALYSIS; CELLS;

EID: 0037086678     PISSN: 02646021     EISSN: None     Source Type: Journal    
DOI: 10.1042/0264-6021:3620561     Document Type: Article
Times cited : (152)

References (67)
  • 1
    • 0344734113 scopus 로고    scopus 로고
    • Modulation of the myocardial response to ischemia
    • (1998) Lab. Invest. , vol.78 , pp. 1345-1373
    • Buja, L.M.1
  • 24
    • 0032571270 scopus 로고    scopus 로고
    • Stimulation of stress-regulated mitogen-activated protein kinases (stress-activated protein kinases/c-Jun-N-terminal kinases and p38-mitogen-activated protein kinases) in perfused rat hearts by oxidative and other stresses
    • (1998) J. Biol. Chem. , vol.273 , pp. 7228-7234
    • Clerk, A.1    Fuller, S.J.2    Michael, A.3    Sugden, P.H.4
  • 60
    • 0034714334 scopus 로고    scopus 로고
    • p38 mitogen-activated protein kinase mediates Bid cleavage, mitochondrial dysfunction, and caspase-3 activation during apoptosis induced by singlet oxygen but not by hydrogen peroxide
    • (2000) J. Biol. Chem. , vol.275 , pp. 25939-25948
    • Zhuang, S.1    Demirs, J.2    Kochevar, I.3


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.