Acetaminophen-induced inhibition of Fas receptor-mediated liver cell apoptosis: Mitochondrial dysfunction versus glutathione depletion

Toxicology and Applied Pharmacology

Volumn 181, Issue 2, 2002, Pages 133-141

  Knight, Tamara R ^a   Jaeschke, Hartmut ^a  

^a UNIVERSITY OF ARKANSAS FOR MEDICAL SCIENCES   (United States)

Author keywords

Acetaminophen; Allopurinol; Apoptosis; Fas receptor; Liver failure; Mitochondria

Indexed keywords

ADENOSINE TRIPHOSPHATE; ALLOPURINOL; CASPASE 3; CELL RECEPTOR; CYTOCHROME C; DNA FRAGMENT; FAS ANTIBODY; FAS RECEPTOR; GLUTATHIONE; GLUTATHIONE DISULFIDE; PARACETAMOL; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL DAMAGE; CONTROLLED STUDY; DRUG INTOXICATION; ENZYME ACTIVATION; ENZYME ACTIVITY; HISTOCHEMISTRY; LIVER INJURY; LIVER MITOCHONDRION; MALE; MITOCHONDRIAL RESPIRATION; MOUSE; NICK END LABELING; NONHUMAN; OXIDATIVE STRESS; PROTEIN PROCESSING; SIGNAL TRANSDUCTION;

ANIMALIA;

EID: 0036607117     PISSN: 0041008X     EISSN: None     Source Type: Journal    
DOI: 10.1006/taap.2002.9407     Document Type: Article

References (37)

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