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Volumn 19, Issue 51, 2000, Pages 5906-5918
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v-Jun sensitizes cells to apoptosis by a mechanism involving mitochondrial cytochrome C release
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Author keywords
Apoptosis; Cytochrome C; p53; v Jun
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Indexed keywords
CYTOCHROME C;
FAS ANTIGEN;
GENE PRODUCT;
GROWTH FACTOR;
PHOSPHOTRANSFERASE;
PROTEIN BCL 2;
PROTEIN P53;
TRANSCRIPTION FACTOR E2F;
UNCLASSIFIED DRUG;
V JUN PROTEIN;
ANIMAL CELL;
APOPTOSIS;
ARTICLE;
CELL SURVIVAL;
CONTROLLED STUDY;
DNA FLANKING REGION;
EMBRYO;
ENZYME RELEASE;
GENE ACTIVATION;
GENE ACTIVITY;
MITOGENESIS;
NONHUMAN;
ONCOGENE MYC;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
REGULATORY MECHANISM;
SENSITIZATION;
SIGNAL TRANSDUCTION;
1-PHOSPHATIDYLINOSITOL 3-KINASE;
ADAPTOR PROTEINS, SIGNAL TRANSDUCING;
AMINO ACID CHLOROMETHYL KETONES;
ANIMALS;
APOPTOSIS;
CARRIER PROTEINS;
CASPASES;
CELLS, CULTURED;
CHICK EMBRYO;
CYSTEINE PROTEINASE INHIBITORS;
CYTOCHROME C GROUP;
FAS-ASSOCIATED DEATH DOMAIN PROTEIN;
FIBROBLASTS;
GENES, JUN;
GROWTH SUBSTANCES;
MITOCHONDRIA;
ONCOGENE PROTEIN P65(GAG-JUN);
ONCOGENE PROTEIN PP60(V-SRC);
PROTO-ONCOGENE PROTEINS C-BCL-2;
TUMOR SUPPRESSOR PROTEIN P53;
ANIMALIA;
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EID: 0034735898
PISSN: 09509232
EISSN: None
Source Type: Journal
DOI: 10.1038/sj.onc.1203954 Document Type: Article |
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Times cited : (8)
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References (48)
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