The v-Jun oncoprotein replaces p39 c-Jun as the predominant AP-1 constituent in ASV17-transformed fibroblasts: Implications for SAPK/JNK-mediated signal transduction

Oncogene

Volumn 12, Issue 11, 1996, Pages 2409-2418

  Kilbey, A ^a   Black, E J ^a   Unlu, M ^a   Gillespie, D A F ^a  

^a BEATSON INSTITUTE FOR CANCER RESEARCH   (United Kingdom)

Author keywords

AP 1; SAPK JNK; Transformation; v Jun

Indexed keywords

ONCOPROTEIN; PROTEIN KINASE; TRANSCRIPTION FACTOR AP 1;

ANIMAL CELL; ARTICLE; CARCINOGENESIS; CELL TRANSFORMATION; CHICKEN; CONTROLLED STUDY; DNA TRANSCRIPTION; EMBRYO; FIBROBLAST; GENE EXPRESSION; MOUSE; NONHUMAN; ONCOGENE C JUN; PRIORITY JOURNAL; PROTEIN DNA BINDING; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; SIGNAL TRANSDUCTION; TRANSCRIPTION REGULATION;

3T3 CELLS; AMINO ACID SEQUENCE; ANIMALS; CA(2+)-CALMODULIN DEPENDENT PROTEIN KINASE; CELL TRANSFORMATION, NEOPLASTIC; CHICK EMBRYO; CHLORAMPHENICOL O-ACETYLTRANSFERASE; DNA; JNK MITOGEN-ACTIVATED PROTEIN KINASES; MICE; MITOGEN-ACTIVATED PROTEIN KINASES; MOLECULAR SEQUENCE DATA; ONCOGENE PROTEIN P65(GAG-JUN); PHOSPHORYLATION; PROTO-ONCOGENE PROTEINS C-JUN; SIGNAL TRANSDUCTION; TRANSCRIPTION FACTOR AP-1;

ANIMALIA; GALLUS GALLUS;

EID: 0029984823     PISSN: 09509232     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (0)