RelB modulation of IκBα stability as a mechanism of transcription suppression of interleukin-1α (IL-1α), IL-1β, and tumor necrosis factor alpha in fibroblasts

Molecular and Cellular Biology

Volumn 19, Issue 11, 1999, Pages 7688-7696

  Xia, Yiyang ^a   Chen, Shizhong ^a   Wang, Yibin ^b   Mackman, Nigel ^a   Ku, George ^c   Lo, David ^a   Feng, Lili ^a  

^a SCRIPPS RESEARCH INSTITUTE   (United States)

^b UNIVERSITY OF CALIFORNIA SAN DIEGO   (United States)

^c VERTEX PHARMACEUTICALS   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CHEMOKINE; GENE PRODUCT; I KAPPA B ALPHA; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INTERLEUKIN 1ALPHA; INTERLEUKIN 1BETA; NUCLEAR PROTEIN; PHOSPHOTRANSFERASE; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR RELB; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG; CHUK PROTEIN, MOUSE; DNA BINDING PROTEIN; I KAPPA B; I KAPPA B KINASE; IKBKB PROTEIN, MOUSE; IKBKE PROTEIN, MOUSE; INTERLEUKIN 1; LIPOPOLYSACCHARIDE; MESSENGER RNA; NF KAPPAB INHIBITOR ALPHA; NF-KAPPAB INHIBITOR ALPHA; ONCOPROTEIN; PROTEIN SERINE THREONINE KINASE; RELB PROTEIN, MOUSE;

ANIMAL CELL; ARTICLE; CONTROLLED STUDY; CYTOKINE PRODUCTION; FIBROBLAST; IMMUNE RESPONSE; INFLAMMATION; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN FAMILY; PROTEIN MODIFICATION; PROTEIN STABILITY; ANIMAL; BIOSYNTHESIS; CELL CULTURE; CELL NUCLEUS; CYTOLOGY; GENE EXPRESSION REGULATION; GENETIC TRANSCRIPTION; GENETICS; IMMUNITY; KIDNEY; METABOLISM; MOUSE MUTANT; PHOSPHORYLATION; PHYSIOLOGY;

ANIMALIA;

ANIMALS; CELL NUCLEUS; CELLS, CULTURED; DNA-BINDING PROTEINS; FIBROBLASTS; GENE EXPRESSION REGULATION; I-KAPPA B KINASE; I-KAPPA B PROTEINS; IMMUNITY; INFLAMMATION; INTERLEUKIN-1; KIDNEY; LIPOPOLYSACCHARIDES; MICE; MICE, MUTANT STRAINS; PHOSPHORYLATION; PROTEIN-SERINE-THREONINE KINASES; PROTO-ONCOGENE PROTEINS; RNA, MESSENGER; TRANSCRIPTION FACTOR RELB; TRANSCRIPTION FACTORS; TRANSCRIPTION, GENETIC; TUMOR NECROSIS FACTOR-ALPHA;

EID: 0032589473     PISSN: 02707306     EISSN: None     Source Type: Journal    
DOI: 10.1128/mcb.19.11.7688     Document Type: Article

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