p50-NF-κB complexes partially compensate for the absence of RelB: Severely increased pathology in p50(-/-)relB(-/-)double-knockout mice

Journal of Experimental Medicine

Volumn 185, Issue 7, 1997, Pages 1359-1370

  Weih, Falk ^a   Durham, Stephen K ^a   Barton, Debra S ^a   Sha, William C ^b,c   Baltimore, David ^b   Bravo, Rodrigo ^a  

^a BRISTOL MYERS SQUIBB   (United States)

^b MASSACHUSETTS INSTITUTE OF TECHNOLOGY   (United States)

^c UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; PROTEIN SUBUNIT; ONCOPROTEIN; RELB PROTEIN, MOUSE; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR RELB;

ANIMAL MODEL; ANIMAL TISSUE; ANTIGEN BINDING; ARTICLE; HISTOPATHOLOGY; HUMORAL IMMUNE DEFICIENCY; HYPERPLASIA; INFLAMMATORY DISEASE; INFLAMMATORY INFILTRATE; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; THYMUS ATROPHY; ANIMAL; BONE MARROW; COMPARATIVE STUDY; DIGESTIVE SYSTEM; GENE EXPRESSION REGULATION; GENETICS; HEART MUSCLE; INFLAMMATION; LUNG; LYMPHOCYTE; LYMPHOID TISSUE; MACROPHAGE; MORTALITY; MOUSE MUTANT; MULTIPLE MALFORMATION SYNDROME; PATHOLOGY; PROTEIN BINDING;

ABNORMALITIES, MULTIPLE; ANIMALS; BONE MARROW; DIGESTIVE SYSTEM; GENE EXPRESSION REGULATION; INFLAMMATION; LUNG; LYMPHOCYTES; LYMPHOID TISSUE; MACROPHAGES; MICE; MICE, KNOCKOUT; MYOCARDIUM; NF-KAPPA B; NF-KAPPA B P50 SUBUNIT; PHENOTYPE; PROTEIN BINDING; PROTO-ONCOGENE PROTEINS; TRANSCRIPTION FACTOR RELB; TRANSCRIPTION FACTORS;

EID: 0030615259     PISSN: 00221007     EISSN: None     Source Type: Journal    
DOI: 10.1084/jem.185.7.1359     Document Type: Article

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