The free radical antioxidant vitamin E protects cortical synaptosomal membranes from amyloid β-peptide(25-35) toxicity but not from hydroxynonenal toxicity: Relevance to the free radical hypothesis of Alzheimer's disease

Neurochemical Research

Volumn 23, Issue 11, 1998, Pages 1403-1410

  Subramaniam, Ram ^a   Koppal, Tanuja ^a   Green, Michael ^a   Yatin, Servet ^a   Jordan, Brad ^a   Drake, Jennifer ^a   Butterfield, D Allan ^a  

^a UNIVERSITY OF KENTUCKY   (United States)

Author keywords

Alzheimer's; Amyloid; Free radicals; HNE; Lipid peroxidation; Protein oxidation; Vitamin E

Indexed keywords

ALPHA TOCOPHEROL; AMYLOID BETA PROTEIN;

ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ARTICLE; DRUG EFFICACY; GERBIL; LIPID PEROXIDATION; MALE; NEUROFIBRILLARY TANGLE; NEUROTOXICITY; NONHUMAN; PATHOPHYSIOLOGY; PRIORITY JOURNAL;

ALDEHYDES; ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; ANIMALS; ANTIOXIDANTS; BIOLOGICAL MARKERS; CELLS, CULTURED; CEREBRAL CORTEX; ELECTRON SPIN RESONANCE SPECTROSCOPY; FREE RADICAL SCAVENGERS; NEUROPROTECTIVE AGENTS; OXIDATION-REDUCTION; PEPTIDE FRAGMENTS; RATS; RATS, SPRAGUE-DAWLEY; SYNAPTOSOMES; TETRAZOLIUM SALTS; THIAZOLES; VITAMIN E;

EID: 0031741578     PISSN: 03643190     EISSN: None     Source Type: Journal    
DOI: 10.1023/A:1020754807671     Document Type: Article

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