Impairment of glucose and glutamate transport and induction of mitochondrial oxidative stress and dysfunction in synaptosomes by amyloid β- peptide: Role of the lipid peroxidation product 4-hydroxynonenal

Journal of Neurochemistry

Volumn 69, Issue 1, 1997, Pages 273-284

  Keller, Jeffrey N ^a,b   Pang, Zheng ^a   Geddes, James W ^a   Begley, James G ^a   Germeyer, Ariane ^a   Waeg, Georg ^c   Mattson, Mark P ^a,b  

^a UNIVERSITY OF KENTUCKY   (United States)

^b UNIVERSITY OF KENTUCKY   (United States)

^c UNIVERSITY OF GRAZ   (Austria)

Author keywords

Alzheimer's disease; ATP; Cerebral cortex; Excitotoxicity; Malondialdehyde; Mitochondrial respiration; Superoxide anion radical; Synaptic metabolism

Indexed keywords

4 HYDROXYNONENAL; AMYLOID BETA PROTEIN; GLUCOSE; GLUTAMIC ACID;

ALZHEIMER DISEASE; AMINO ACID TRANSPORT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN CORTEX; BRAIN MITOCHONDRION; CONTROLLED STUDY; FEMALE; GLUCOSE TRANSPORT; LIPID PEROXIDATION; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT; SYNAPTOSOME;

ANIMALIA;

EID: 0030966546     PISSN: 00223042     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1471-4159.1997.69010273.x     Document Type: Article

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