-
1
-
-
0028151819
-
Negative selection of lymphocytes
-
Nossal GJV. Negative selection of lymphocytes. Cell. 76:1994;229-239.
-
(1994)
Cell
, vol.76
, pp. 229-239
-
-
Nossal, G.J.V.1
-
2
-
-
0028032155
-
Positive selection of lymphocytes
-
Von Boehmer H. Positive selection of lymphocytes. Cell. 76:1994;219-228.
-
(1994)
Cell
, vol.76
, pp. 219-228
-
-
Von Boehmer, H.1
-
3
-
-
0028201339
-
Selective events in T cell development
-
Robey E, Fowlkes BJ. Selective events in T cell development. Annu Rev Immunol. 12:1994;675-705.
-
(1994)
Annu Rev Immunol
, vol.12
, pp. 675-705
-
-
Robey, E.1
Fowlkes, B.J.2
-
4
-
-
0029961719
-
Die and let live: Eliminating dangerous lymphocytes
-
Abbas AK. Die and let live: eliminating dangerous lymphocytes. Cell. 84:1996;655-657.
-
(1996)
Cell
, vol.84
, pp. 655-657
-
-
Abbas, A.K.1
-
5
-
-
0028956550
-
Regulators of cell death
-
Korsmeyer SJ. Regulators of cell death. Trends Genet. 11:1995;101-105.
-
(1995)
Trends Genet
, vol.11
, pp. 101-105
-
-
Korsmeyer, S.J.1
-
6
-
-
0028915454
-
Regulation of lymphocyte survival by the bcl-2 gene family
-
Cory S. Regulation of lymphocyte survival by the bcl-2 gene family. Annu Rev Immunol. 13:1995;513-543.
-
(1995)
Annu Rev Immunol
, vol.13
, pp. 513-543
-
-
Cory, S.1
-
7
-
-
0030584079
-
Apoptosis meets signal transduction: Elimination of a BAD influence
-
Gajewski TF, Thompson CB. Apoptosis meets signal transduction: elimination of a BAD influence. Cell. 87:1996;589-592.
-
(1996)
Cell
, vol.87
, pp. 589-592
-
-
Gajewski, T.F.1
Thompson, C.B.2
-
8
-
-
0028927607
-
The Fas death factor
-
Nagata S, Golstein P. The Fas death factor. Science. 267:1995;1449-1455.
-
(1995)
Science
, vol.267
, pp. 1449-1455
-
-
Nagata, S.1
Golstein, P.2
-
9
-
-
0028050656
-
Functional Myc-Max heterodimer is required for activation-induced apoptosis in T cell hybridomas
-
Bissonnette RP, McGahon A, Mahboubi A, Green DR. Functional Myc-Max heterodimer is required for activation-induced apoptosis in T cell hybridomas. J Exp Med. 180:1994;2413-2418.
-
(1994)
J Exp Med
, vol.180
, pp. 2413-2418
-
-
Bissonnette, R.P.1
McGahon, A.2
Mahboubi, A.3
Green, D.R.4
-
10
-
-
0027976193
-
Apoptotic signals delivered through the T-cell receptor of a T-cell hybrid require the immediate-early gene nur77
-
Liu Z-G, Smith SW, McLaughlin KA, Schwartz LM, Osborne B. Apoptotic signals delivered through the T-cell receptor of a T-cell hybrid require the immediate-early gene nur77. Nature. 367:1994;281-284.
-
(1994)
Nature
, vol.367
, pp. 281-284
-
-
Liu, Z.-G.1
Smith, S.W.2
McLaughlin, K.A.3
Schwartz, L.M.4
Osborne, B.5
-
11
-
-
0028008513
-
Requirement for the ophan steroid receptor Nur77 in apoptosis of T-cell hybridomas
-
Woronicz JD, Calnan B, Ngo V, Winoto A. Requirement for the ophan steroid receptor Nur77 in apoptosis of T-cell hybridomas. Nature. 367:1994;277-281.
-
(1994)
Nature
, vol.367
, pp. 277-281
-
-
Woronicz, J.D.1
Calnan, B.2
Ngo, V.3
Winoto, A.4
-
12
-
-
0030176271
-
A novel gene product that couples TCR signaling to Fas(CD95) expression in activation-induced cell death
-
of special interest. This paper reports the cloning and identification of TDAG51 as a protein which regulates Fas transcription in response to TCR signals.
-
Park C-G, Lee S-Y, Kandala G, Lee SY, Choi Y. A novel gene product that couples TCR signaling to Fas(CD95) expression in activation-induced cell death. of special interest Immunity. 4:1996;583-591 This paper reports the cloning and identification of TDAG51 as a protein which regulates Fas transcription in response to TCR signals.
-
(1996)
Immunity
, vol.4
, pp. 583-591
-
-
Park, C.-G.1
Lee, S.-Y.2
Kandala, G.3
Lee, S.Y.4
Choi, Y.5
-
13
-
-
0029671219
-
Interfering with apoptosis: Ca(2+)-binding protein ALG-2 and Alzheimer's disease gene ALG-3
-
Vito P, Lacana E, D'Adamio L. Interfering with apoptosis: Ca(2+)-binding protein ALG-2 and Alzheimer's disease gene ALG-3. Science. 271:1996;521-525.
-
(1996)
Science
, vol.271
, pp. 521-525
-
-
Vito, P.1
Lacana, E.2
D'Adamio, L.3
-
14
-
-
0027364326
-
The c-myc protein induces cell cycle progression and apoptosis through dimerization with Max
-
Amati B, Littlewood TD, Evan GI, Land H. The c-myc protein induces cell cycle progression and apoptosis through dimerization with Max. EMBO J. 12:1993;5083-5087.
-
(1993)
EMBO J
, vol.12
, pp. 5083-5087
-
-
Amati, B.1
Littlewood, T.D.2
Evan, G.I.3
Land, H.4
-
15
-
-
0029779280
-
Cdc25 cell-cycle phosphatase as a target of c-myc
-
1/S transition, is a physiological target for c-myc.
-
1/S transition, is a physiological target for c-myc.
-
(1996)
Nature
, vol.382
, pp. 511-517
-
-
Galaktionov, K.1
Chen, X.2
Beach, D.3
-
17
-
-
0024368696
-
Cyclosporin A inhibits activation-induced cell death in T-cells and thymocytes
-
Shi YF, Sahai BM, Green DR. Cyclosporin A inhibits activation-induced cell death in T-cells and thymocytes. Nature. 339:1989;625-626.
-
(1989)
Nature
, vol.339
, pp. 625-626
-
-
Shi, Y.F.1
Sahai, B.M.2
Green, D.R.3
-
18
-
-
0028842908
-
Regulation of the Nur77 orphan steroid receptor in activation-induced apoptosis
-
of special interest. This paper reports an extensive characterization of Nur77 in T cell hybridomas during TCR-mediated apoptosis. It also shows that the MEF2 family of transcription factors mediates the calcium responsiveness of the Nur77 promoter.
-
Woronicz JD, Lina A, Calnan BJ, Szychowski S, Cheng L, Winoto A. Regulation of the Nur77 orphan steroid receptor in activation-induced apoptosis. of special interest Mol Cell Biol. 15:1995;6364-6376 This paper reports an extensive characterization of Nur77 in T cell hybridomas during TCR-mediated apoptosis. It also shows that the MEF2 family of transcription factors mediates the calcium responsiveness of the Nur77 promoter.
-
(1995)
Mol Cell Biol
, vol.15
, pp. 6364-6376
-
-
Woronicz, J.D.1
Lina, A.2
Calnan, B.J.3
Szychowski, S.4
Cheng, L.5
Winoto, A.6
-
19
-
-
0028606279
-
Molecular cloning of a novel thyroid/steroid receptor superfamily gene from cultured rat neuronal cells
-
Ohkura N, Hijikuro M, Yamamoto A, Miki K. Molecular cloning of a novel thyroid/steroid receptor superfamily gene from cultured rat neuronal cells. Biochem Biophys Res Commun. 205:1994;1959-1965.
-
(1994)
Biochem Biophys Res Commun
, vol.205
, pp. 1959-1965
-
-
Ohkura, N.1
Hijikuro, M.2
Yamamoto, A.3
Miki, K.4
-
20
-
-
0027074637
-
Identification of a new brain-specific transcription factor, NURR1
-
Law SW, Conneely OM, DeMayo FJ, O'Malley BW. Identification of a new brain-specific transcription factor, NURR1. Mol Endocrinol. 6:1992;2129-2135.
-
(1992)
Mol Endocrinol
, vol.6
, pp. 2129-2135
-
-
Law, S.W.1
Conneely, O.M.2
DeMayo, F.J.3
O'Malley, B.W.4
-
21
-
-
0030977267
-
Functional redundancy of the Nur77 and Nor-1 orphan steroid receptors in T cell apoptosis
-
of outstanding interest. These authors identify Nor-1, a member of the Nur77 family, as a protein with similar function to Nur77. Constitutive Nor-1 expression leads to massive thymocyte cell death. In addition, this paper shows that the expression of Nur77 can rescue the lymphoproliferative disease of gld/gld (FasL mutant) mice, demonstrating a Fas-independent nature of Nur77-initiated apoptosis.
-
Cheng LE, Chan FK, Cado D, Winoto A. Functional redundancy of the Nur77 and Nor-1 orphan steroid receptors in T cell apoptosis. of outstanding interest EMBO J. 16:1997;1865-1875 These authors identify Nor-1, a member of the Nur77 family, as a protein with similar function to Nur77. Constitutive Nor-1 expression leads to massive thymocyte cell death. In addition, this paper shows that the expression of Nur77 can rescue the lymphoproliferative disease of gld/gld (FasL mutant) mice, demonstrating a Fas-independent nature of Nur77-initiated apoptosis.
-
(1997)
EMBO J
, vol.16
, pp. 1865-1875
-
-
Cheng, L.E.1
Chan, F.K.2
Cado, D.3
Winoto, A.4
-
22
-
-
0028807837
-
A role of the orphan steroid receptor Nur77 in antigen-induced negative selection
-
of outstanding interest. This paper, along with [23], demonstrates that expression of a dominant negative Nur77 protein partially inhibits apoptosis accompanying negative selection. These data suggest that members of the Nur77 family are crucial for thymocytes apoptosis. In addition, this paper, along with [25], shows that constitutive expression of Nur77 leads to massive thymocyte apoptosis.
-
Calnan B, Szychowski S, Chan FKM, Cado D, Winoto A. A role of the orphan steroid receptor Nur77 in antigen-induced negative selection. of outstanding interest Immunity. 3:1995;273-282 This paper, along with [23], demonstrates that expression of a dominant negative Nur77 protein partially inhibits apoptosis accompanying negative selection. These data suggest that members of the Nur77 family are crucial for thymocytes apoptosis. In addition, this paper, along with [25], shows that constitutive expression of Nur77 leads to massive thymocyte apoptosis.
-
(1995)
Immunity
, vol.3
, pp. 273-282
-
-
Calnan, B.1
Szychowski, S.2
Chan, F.K.M.3
Cado, D.4
Winoto, A.5
-
23
-
-
0029914914
-
Inhibition of Nur77/Nurr1 leads to inefficient clonal deletion of self-reactive T cells
-
of outstanding interest. See annotation [22].
-
Zhou T, Cheng J, Yang P, Wang Z, Liu C, Su X, Bluethmann H, Mountz JD. Inhibition of Nur77/Nurr1 leads to inefficient clonal deletion of self-reactive T cells. of outstanding interest J Exp Med. 183:1996;1879-1892 See annotation [22].
-
(1996)
J Exp Med
, vol.183
, pp. 1879-1892
-
-
Zhou, T.1
Cheng, J.2
Yang, P.3
Wang, Z.4
Liu, C.5
Su, X.6
Bluethmann, H.7
Mountz, J.D.8
-
24
-
-
0029056318
-
Unimpaired thymic and peripheral T cell death in mice lacking the nuclear receptor NGFI-B (Nur77)
-
Lee SL, Wesselschmidt RL, Linette GP, Kanagawa O, Russell JH, Milbrandt J. Unimpaired thymic and peripheral T cell death in mice lacking the nuclear receptor NGFI-B (Nur77). Science. 269:1995;532-535.
-
(1995)
Science
, vol.269
, pp. 532-535
-
-
Lee, S.L.1
Wesselschmidt, R.L.2
Linette, G.P.3
Kanagawa, O.4
Russell, J.H.5
Milbrandt, J.6
-
25
-
-
0029952319
-
Apoptosis of nur77/N10-transgenic thymocytes involves the Fas/Fas ligand pathway
-
of outstanding interest. This paper, along with [22], shows that constitutive expression of Nur77 in thymocytes leads to apoptosis. These authors further found that FasL transcription is upregulated in Nur77 transgenic thymocytes. Mutation at the FasL gene partially rescues thymocytes from Nur77-mediated apoptosis, suggesting that FasL is one of the genes regulated by Nur77.
-
Weih F, Ryseck R-P, Chen L, Bravo R. Apoptosis of nur77/N10-transgenic thymocytes involves the Fas/Fas ligand pathway. of outstanding interest Proc Natl Acad Sci USA. 93:1996;5533-5538 This paper, along with [22], shows that constitutive expression of Nur77 in thymocytes leads to apoptosis. These authors further found that FasL transcription is upregulated in Nur77 transgenic thymocytes. Mutation at the FasL gene partially rescues thymocytes from Nur77-mediated apoptosis, suggesting that FasL is one of the genes regulated by Nur77.
-
(1996)
Proc Natl Acad Sci USA
, vol.93
, pp. 5533-5538
-
-
Weih, F.1
Ryseck, R.-P.2
Chen, L.3
Bravo, R.4
-
26
-
-
0029976817
-
An essential role for NF-κB in preventing TNF-α-induced cell death
-
of special interest. This paper, along with [27-29], demonstrates that the absence of NF-κB renders cells more susceptible to TNF-induced apoptosis.
-
Beg AA, Baltimore D. An essential role for NF-κB in preventing TNF-α-induced cell death. of special interest Science. 274:1996;782-784 This paper, along with [27-29], demonstrates that the absence of NF-κB renders cells more susceptible to TNF-induced apoptosis.
-
(1996)
Science
, vol.274
, pp. 782-784
-
-
Beg, A.A.1
Baltimore, D.2
-
27
-
-
0029992609
-
Suppression of TNF-α-induced apoptosis by NF-κB
-
of special interest. See annotation [26].
-
Antwerp DJ, Martin SJ, Kafri T, Green DR, Verma IM. Suppression of TNF-α-induced apoptosis by NF-κB. of special interest Science. 274:1996;787-789 See annotation [26].
-
(1996)
Science
, vol.274
, pp. 787-789
-
-
Antwerp, D.J.1
Martin, S.J.2
Kafri, T.3
Green, D.R.4
Verma, I.M.5
-
28
-
-
0030298294
-
Dissection of TNF receptor 1 effector functions: JNK activation is not linked to apoptosis while NF-κB activation prevents cell death
-
of special interest. See annotation [26].
-
Liu Z-G, Hsu H, Goeddel DV, Karin M. Dissection of TNF receptor 1 effector functions: JNK activation is not linked to apoptosis while NF-κB activation prevents cell death. of special interest Cell. 87:1996;565-576 See annotation [26].
-
(1996)
Cell
, vol.87
, pp. 565-576
-
-
Liu, Z.-G.1
Hsu, H.2
Goeddel, D.V.3
Karin, M.4
-
29
-
-
0029858387
-
TNF-and cancer therapy-induced apoptosis: Potentiation by inhibition of NF-κB
-
of special interest. See annotation [26].
-
Wang C-Y, Mayo MW, Baldwin ASJ. TNF-and cancer therapy-induced apoptosis: potentiation by inhibition of NF-κB. of special interest Science. 274:1996;784-787 See annotation [26].
-
(1996)
Science
, vol.274
, pp. 784-787
-
-
Wang, C.-Y.1
Mayo, M.W.2
Baldwin, A.S.J.3
-
30
-
-
0029977751
-
E2F and cell proliferation: A world turned upside down
-
Weinberg RA. E2F and cell proliferation: a world turned upside down. Cell. 85:1996;457-459.
-
(1996)
Cell
, vol.85
, pp. 457-459
-
-
Weinberg, R.A.1
-
31
-
-
0029949784
-
E2F-1 functions in mice to promote apoptosis and suppress proliferation
-
Field SJ, Tsai F-Y, Kuo F, Zubiaga AM, Kaelin WG, Livingston DM, Orkin SH, Greenberg ME. E2F-1 functions in mice to promote apoptosis and suppress proliferation. Cell. 85:1996;549-561.
-
(1996)
Cell
, vol.85
, pp. 549-561
-
-
Field, S.J.1
Tsai, F.-Y.2
Kuo, F.3
Zubiaga, A.M.4
Kaelin, W.G.5
Livingston, D.M.6
Orkin, S.H.7
Greenberg, M.E.8
-
32
-
-
13244300652
-
Impaired negative selection of T cells in Hodgkin's disease antigen CD30-deficient mice
-
of outstanding interest. These authors generate CD30-deficient mice and show that antigen negative selection is partially defective in these mice.
-
Amakawa R, Hakem A, Kundig TM, Matsuyama T, Simard JJL, Timms E, Wakeman A, Mittruecker H-W, Griesser H, Takimoto H, et al. Impaired negative selection of T cells in Hodgkin's disease antigen CD30-deficient mice. of outstanding interest Cell. 84:1996;551-562 These authors generate CD30-deficient mice and show that antigen negative selection is partially defective in these mice.
-
(1996)
Cell
, vol.84
, pp. 551-562
-
-
Amakawa, R.1
Hakem, A.2
Kundig, T.M.3
Matsuyama, T.4
Simard, J.J.L.5
Timms, E.6
Wakeman, A.7
Mittruecker, H.-W.8
Griesser, H.9
Takimoto, H.10
-
33
-
-
0030050618
-
T cell receptor-dependent cell death of T cell hybridomas mediated by the CD30 cytoplasmic domain in association with tumor necrosis factor receptor-associated factors
-
Lee SY, Park CG, Choi Y. T cell receptor-dependent cell death of T cell hybridomas mediated by the CD30 cytoplasmic domain in association with tumor necrosis factor receptor-associated factors. J Exp Med. 183:1996;669-674.
-
(1996)
J Exp Med
, vol.183
, pp. 669-674
-
-
Lee, S.Y.1
Park, C.G.2
Choi, Y.3
-
34
-
-
0030061149
-
Structure and expression of murine CD30 and its role in cytokine production
-
Bowen MA, Lee RK, Miragliotta G, Nam SY, Podack ER. Structure and expression of murine CD30 and its role in cytokine production. J Immunol. 156:1996;442-449.
-
(1996)
J Immunol
, vol.156
, pp. 442-449
-
-
Bowen, M.A.1
Lee, R.K.2
Miragliotta, G.3
Nam, S.Y.4
Podack, E.R.5
-
35
-
-
0028877057
-
An essential role for gp39, the ligand for CD40, in thymic selection
-
Foy TM, Page DM, Waldschmidt TJ, Schoneveld A, Laman JD, Masters SR, Tygrett L, Ledbetter JA, Aruffo A, Claasen E, et al. An essential role for gp39, the ligand for CD40, in thymic selection. J Exp Med. 182:1995;1377-1388.
-
(1995)
J Exp Med
, vol.182
, pp. 1377-1388
-
-
Foy, T.M.1
Page, D.M.2
Waldschmidt, T.J.3
Schoneveld, A.4
Laman, J.D.5
Masters, S.R.6
Tygrett, L.7
Ledbetter, J.A.8
Aruffo, A.9
Claasen, E.10
-
36
-
-
0030175603
-
Role of Fas-mediated cell death in the regulation of immune responses
-
Van Parijs L, Abbas AK. Role of Fas-mediated cell death in the regulation of immune responses. Curr Opin Immunol. 8:1996;355-361.
-
(1996)
Curr Opin Immunol
, vol.8
, pp. 355-361
-
-
Van Parijs, L.1
Abbas, A.K.2
-
37
-
-
0030200829
-
The roles of Fas/APO-1 (CD95) and TNF in antigen-induced programmed cell death in T cell receptor transgenic mice
-
of special interest. This paper demonstrates that both Fas and TNF pathways play important roles in apoptosis of peripheral T cells but not in negative selection.
-
Sytwu H-K, Liblau RS, McDevitt HO. The roles of Fas/APO-1 (CD95) and TNF in antigen-induced programmed cell death in T cell receptor transgenic mice. of special interest Immunity. 5:1996;17-30 This paper demonstrates that both Fas and TNF pathways play important roles in apoptosis of peripheral T cells but not in negative selection.
-
(1996)
Immunity
, vol.5
, pp. 17-30
-
-
Sytwu, H.-K.1
Liblau, R.S.2
McDevitt, H.O.3
-
38
-
-
0029112253
-
Induction of apoptosis in mature T cells by tumour necrosis factor
-
of outstanding interest. This paper shows that Fas and TNF are crucial players in apoptosis of activated peripheral T cells.
-
Zheng L, Fisher G, Miller RE, Peschon J, Lynch DH, Lenardo MJ. Induction of apoptosis in mature T cells by tumour necrosis factor. of outstanding interest Nature. 377:1995;348-351 This paper shows that Fas and TNF are crucial players in apoptosis of activated peripheral T cells.
-
(1995)
Nature
, vol.377
, pp. 348-351
-
-
Zheng, L.1
Fisher, G.2
Miller, R.E.3
Peschon, J.4
Lynch, D.H.5
Lenardo, M.J.6
-
39
-
-
0030497507
-
Fas modulation of apoptosis during negative selection of thymocytes
-
In contrast to [37] and most other reports, these authors show that Fas can play a role in apoptosis of thymocytes.
-
Castro JE, Listman JA, Jacobson BA, Wang Y, Lopez PA, Ju S, Finn PW, Perkins DL. Fas modulation of apoptosis during negative selection of thymocytes. Immunity. 5:1996;617-627 In contrast to [37] and most other reports, these authors show that Fas can play a role in apoptosis of thymocytes.
-
(1996)
Immunity
, vol.5
, pp. 617-627
-
-
Castro, J.E.1
Listman, J.A.2
Jacobson, B.A.3
Wang, Y.4
Lopez, P.A.5
Ju, S.6
Finn, P.W.7
Perkins, D.L.8
-
40
-
-
10544236916
-
Signal transduction by DR3, a death domain-containing receptor related to TNFR-1 and CD95
-
of outstanding interest. This paper, together with [41,42], reports the identification of a new receptor which contains a death domain. Interestingly, expression of this receptor is restricted to lymphoid cells.
-
Chinnaiyan AM, O'Rourke K, Yu G-L, Lyons RH, Garg M, Duan DR, Xing L, Gentz R, Ni J, Dixit VM. Signal transduction by DR3, a death domain-containing receptor related to TNFR-1 and CD95. of outstanding interest Science. 274:1996;990-992 This paper, together with [41,42], reports the identification of a new receptor which contains a death domain. Interestingly, expression of this receptor is restricted to lymphoid cells.
-
(1996)
Science
, vol.274
, pp. 990-992
-
-
Chinnaiyan, A.M.1
O'Rourke, K.2
Yu, G.-L.3
Lyons, R.H.4
Garg, M.5
Duan, D.R.6
Xing, L.7
Gentz, R.8
Ni, J.9
Dixit, V.M.10
-
41
-
-
0029969717
-
A death-domain-containing receptor that mediates apoptosis
-
of outstanding interest. See annotation [40].
-
Kitson J, Raven T, Jiang Y-P, Goeddel DV, Giles KM, Pun K-T, Grinham CJ, Brown R, Farrow SN. A death-domain-containing receptor that mediates apoptosis. of outstanding interest Nature. 384:1996;372-375 See annotation [40].
-
(1996)
Nature
, vol.384
, pp. 372-375
-
-
Kitson, J.1
Raven, T.2
Jiang, Y.-P.3
Goeddel, D.V.4
Giles, K.M.5
Pun, K.-T.6
Grinham, C.J.7
Brown, R.8
Farrow, S.N.9
-
42
-
-
0030896087
-
TRAMP, a novel apoptosis-mediating receptor with sequence homology to tumor necrosis factor receptor 1 and Fas (Apo-1/CD95)
-
of outstanding interest. See annotation [40].
-
Bodmer J-L, Burns K, Schneider P, Hofmann K, Steiner V, Thome M, Bornand T, Hahne M, Schrster M, Becker K, et al. TRAMP, a novel apoptosis-mediating receptor with sequence homology to tumor necrosis factor receptor 1 and Fas (Apo-1/CD95). of outstanding interest Immunity. 6:1997;79-88 See annotation [40].
-
(1997)
Immunity
, vol.6
, pp. 79-88
-
-
Bodmer, J.-L.1
Burns, K.2
Schneider, P.3
Hofmann, K.4
Steiner, V.5
Thome, M.6
Bornand, T.7
Hahne, M.8
Schrster, M.9
Becker, K.10
-
43
-
-
0030606315
-
CAR1, a TNFR-related protein, is a cellular receptor for cytopathic avian leukosis-sarcoma viruses and mediates apoptosis
-
of special interest. This paper reports identification of chicken CAR1, a membrane protein homologous to the TNF receptor which is apparently distinct from the human DR3/WSL-1/TRAMP reported in [40-42].
-
Brojatsch J, Naughton J, Rolls MM, Zingler K, Young JAT. CAR1, a TNFR-related protein, is a cellular receptor for cytopathic avian leukosis-sarcoma viruses and mediates apoptosis. of special interest Cell. 87:1996;845-855 This paper reports identification of chicken CAR1, a membrane protein homologous to the TNF receptor which is apparently distinct from the human DR3/WSL-1/TRAMP reported in [40-42].
-
(1996)
Cell
, vol.87
, pp. 845-855
-
-
Brojatsch, J.1
Naughton, J.2
Rolls, M.M.3
Zingler, K.4
Young, J.A.T.5
-
44
-
-
0030996297
-
The receptor for the cytotoxic ligand TRAIL
-
of outstanding interest. This paper reports the identification of a new death domain receptor, DR4. The authors also show that the ligand for DR4 is TRAIL/Apo-2 and that DR4 does not mediate apoptosis through Mort1/FADD.
-
Pan G, O'Rourke K, Chinnaiyan AM, Gentz R, Ebner R, Ni J, Dixit VM. The receptor for the cytotoxic ligand TRAIL. of outstanding interest Science. 276:1997;111-113 This paper reports the identification of a new death domain receptor, DR4. The authors also show that the ligand for DR4 is TRAIL/Apo-2 and that DR4 does not mediate apoptosis through Mort1/FADD.
-
(1997)
Science
, vol.276
, pp. 111-113
-
-
Pan, G.1
O'Rourke, K.2
Chinnaiyan, A.M.3
Gentz, R.4
Ebner, R.5
Ni, J.6
Dixit, V.M.7
-
45
-
-
17544367410
-
Induction of apoptosis by Apo-2 ligand, a new member of the tumor necrosis factor cytokine family
-
Pitti RM, Marsters SA, Ruppert S, Donahue CJ, Moore A, Ashkenazi A. Induction of apoptosis by Apo-2 ligand, a new member of the tumor necrosis factor cytokine family. J Biol Chem. 271:1996;12687-12690.
-
(1996)
J Biol Chem
, vol.271
, pp. 12687-12690
-
-
Pitti, R.M.1
Marsters, S.A.2
Ruppert, S.3
Donahue, C.J.4
Moore, A.5
Ashkenazi, A.6
-
46
-
-
13344285339
-
Identification and characterization of a new member of the TNF family that induces apoptosis
-
Wiley SR, Schooley K, Smolak PJ, Din WS, Huang C-P, Nicholl JK, Sutherland GR, Smith TD, Rauch C, Smith CA, Goodwin RG. Identification and characterization of a new member of the TNF family that induces apoptosis. Immunity. 3:1995;673-682.
-
(1995)
Immunity
, vol.3
, pp. 673-682
-
-
Wiley, S.R.1
Schooley, K.2
Smolak, P.J.3
Din, W.S.4
Huang, C.-P.5
Nicholl, J.K.6
Sutherland, G.R.7
Smith, T.D.8
Rauch, C.9
Smith, C.A.10
Goodwin, R.G.11
-
47
-
-
0030155964
-
Activation of apoptosis by Apo-2 ligand is independent of FADD but blocked by CrmA
-
Marsters SA, Pitti RM, Donahue CJ, Ruppert S, Bauer KD, Ashkenazi A. Activation of apoptosis by Apo-2 ligand is independent of FADD but blocked by CrmA. Curr Biol. 6:1996;750-752.
-
(1996)
Curr Biol
, vol.6
, pp. 750-752
-
-
Marsters, S.A.1
Pitti, R.M.2
Donahue, C.J.3
Ruppert, S.4
Bauer, K.D.5
Ashkenazi, A.6
-
48
-
-
0029664296
-
ICE family protease: Mediators of all apoptotic cell death?
-
Henkart PA. ICE family protease: mediators of all apoptotic cell death? Immunity. 4:1996;195-201.
-
(1996)
Immunity
, vol.4
, pp. 195-201
-
-
Henkart, P.A.1
-
49
-
-
0029995766
-
A license to kill
-
Fraser A, Evan G. A license to kill. Cell. 85:1996;781-784.
-
(1996)
Cell
, vol.85
, pp. 781-784
-
-
Fraser, A.1
Evan, G.2
-
50
-
-
0029782494
-
Cathepsin D protease mediates programmed cell death induced by interferon-gamma, Fas/APO-1 and TNF-alpha
-
Deiss LP, Galinka H, Berissi H, Cohen O, Kimchi A. Cathepsin D protease mediates programmed cell death induced by interferon-gamma, Fas/APO-1 and TNF-alpha. EMBO J. 15:1996;3861-3870.
-
(1996)
EMBO J
, vol.15
, pp. 3861-3870
-
-
Deiss, L.P.1
Galinka, H.2
Berissi, H.3
Cohen, O.4
Kimchi, A.5
-
51
-
-
0029762871
-
Proteasomes play an essential role in thymocyte apoptosis
-
Grimm LM, Goldberg AL, Poirier GG, Schwartz LM, Osborne BA. Proteasomes play an essential role in thymocyte apoptosis. EMBO J. 15:1996;3835-3844.
-
(1996)
EMBO J
, vol.15
, pp. 3835-3844
-
-
Grimm, L.M.1
Goldberg, A.L.2
Poirier, G.G.3
Schwartz, L.M.4
Osborne, B.A.5
-
52
-
-
0028883850
-
Cytotoxicity-dependent APO-1 (Fas/CD95) associated proteins from a death-inducing signaling complex (DISC) with the receptor
-
of outstanding interest. These authors identify several intracellular proteins that associate with the FAS cytoplasmic tail in vivo. One of the Fas-associated proteins is FADD/Mort1. This paper also lays the groundwork for subsequent identification of Fas-associated protease FLICE/MACH-1 as reported in [57]
-
Kischkel FC, Hellbardt S, Behrmann I, Germer M, Pawlita M, Krammer PH, Peter ME. Cytotoxicity-dependent APO-1 (Fas/CD95) associated proteins from a death-inducing signaling complex (DISC) with the receptor. of outstanding interest EMBO J. 14:1995;5579-5588 These authors identify several intracellular proteins that associate with the FAS cytoplasmic tail in vivo. One of the Fas-associated proteins is FADD/Mort1. This paper also lays the groundwork for subsequent identification of Fas-associated protease FLICE/MACH-1 as reported in [57].
-
(1995)
EMBO J
, vol.14
, pp. 5579-5588
-
-
Kischkel, F.C.1
Hellbardt, S.2
Behrmann, I.3
Germer, M.4
Pawlita, M.5
Krammer, P.H.6
Peter, M.E.7
-
53
-
-
0029965280
-
FADD/MORT1 is a common mediator of CD95 (Fas/APO-1) and tumor necrosis factor receptor-induced apoptosis
-
of outstanding interest. This paper shows for the first time that expression of a dominant negative FADD protein can block Fas- and TNF-induced cell death.
-
Chinnaiyan AM, Tepper CG, Seldin MF, O'Rourke K, Kischkel FC, Hellbardt S, Krammer PH, Peter ME, Dixit VM. FADD/MORT1 is a common mediator of CD95 (Fas/APO-1) and tumor necrosis factor receptor-induced apoptosis. of outstanding interest J Biol Chem. 271:1996;4961-4965 This paper shows for the first time that expression of a dominant negative FADD protein can block Fas- and TNF-induced cell death.
-
(1996)
J Biol Chem
, vol.271
, pp. 4961-4965
-
-
Chinnaiyan, A.M.1
Tepper, C.G.2
Seldin, M.F.3
O'Rourke, K.4
Kischkel, F.C.5
Hellbardt, S.6
Krammer, P.H.7
Peter, M.E.8
Dixit, V.M.9
-
54
-
-
0030001051
-
A mouse fas-associated protein with homology to the human Mort1/FADD protein is essential for Fas-induced apoptosis
-
of outstanding interest. of special interest. This paper, along with [56], reports the cDNA and genomic sequence of mouse Mort1/FADD. This paper also shows that endogenous FADD-Fas association is dependent on engagement of the Fas receptor. This observation is different from the overexpression studies but supports the results reported in [52].
-
of outstanding interest Zhang J, Winoto A. A mouse fas-associated protein with homology to the human Mort1/FADD protein is essential for Fas-induced apoptosis. of special interest Mol Cell Biol. 16:1996;2756-2763 This paper, along with [56], reports the cDNA and genomic sequence of mouse Mort1/FADD. This paper also shows that endogenous FADD-Fas association is dependent on engagement of the Fas receptor. This observation is different from the overexpression studies but supports the results reported in [52].
-
(1996)
Mol Cell Biol
, vol.16
, pp. 2756-2763
-
-
Zhang, J.1
Winoto, A.2
-
55
-
-
0029760732
-
RIP and FADD: Two "death domain"-containing proteins can induce apoptosis by convergent, but dissociable, pathways
-
Grimm S, Stanger BZ, Leder P. RIP and FADD: two "death domain"-containing proteins can induce apoptosis by convergent, but dissociable, pathways. Proc Natl Acad Sci USA. 93:1996;10923-10927.
-
(1996)
Proc Natl Acad Sci USA
, vol.93
, pp. 10923-10927
-
-
Grimm, S.1
Stanger, B.Z.2
Leder, P.3
-
56
-
-
0030032106
-
TRADD-TRAF2 and TRADD-FADD interactions define two distinct TNF receptor 1 signal transduction pathways
-
of outstanding interest. This paper shows that TRADD mediates TNF-initiated apoptosis through FADD. Expression of a dominant-negative FADD protein can inhibit TNF-mediated cell death.
-
Hsu H, Shu H-B, Pan M-G, Goeddel DV. TRADD-TRAF2 and TRADD-FADD interactions define two distinct TNF receptor 1 signal transduction pathways. of outstanding interest Cell. 84:1996;299-308 This paper shows that TRADD mediates TNF-initiated apoptosis through FADD. Expression of a dominant-negative FADD protein can inhibit TNF-mediated cell death.
-
(1996)
Cell
, vol.84
, pp. 299-308
-
-
Hsu, H.1
Shu, H.-B.2
Pan, M.-G.3
Goeddel, D.V.4
-
57
-
-
15844412409
-
FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death-inducing signaling complex
-
of outstanding interest. This paper, along with [58,59], reports identification of a novel ICE protease that is recruited to the Fas complex through association with Mort1/FADD.
-
Muzio M, Chinnaiyan AM, Kischkel FC, O'Rourke K, Shevchenko A, Ni J, Scaffidi C, Bretz JD, Zhang M, Gentz R, et al. FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death-inducing signaling complex. of outstanding interest Cell. 85:1996;817-827 This paper, along with [58,59], reports identification of a novel ICE protease that is recruited to the Fas complex through association with Mort1/FADD.
-
(1996)
Cell
, vol.85
, pp. 817-827
-
-
Muzio, M.1
Chinnaiyan, A.M.2
Kischkel, F.C.3
O'Rourke, K.4
Shevchenko, A.5
Ni, J.6
Scaffidi, C.7
Bretz, J.D.8
Zhang, M.9
Gentz, R.10
-
58
-
-
0030011398
-
Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1-and TNF receptor-induced cell death
-
of outstanding interest. See annotation [57]. In addition, this paper shows that a naturally occuring truncated MACH protein can serve as a powerful dominant negative protein to block Fas-initiated apoptosis.
-
Boldin MP, Goncharov TM, Goltsev YV, Wallach D. Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1-and TNF receptor-induced cell death. of outstanding interest Cell. 85:1996;803-815 See annotation [57]. In addition, this paper shows that a naturally occuring truncated MACH protein can serve as a powerful dominant negative protein to block Fas-initiated apoptosis.
-
(1996)
Cell
, vol.85
, pp. 803-815
-
-
Boldin, M.P.1
Goncharov, T.M.2
Goltsev, Y.V.3
Wallach, D.4
-
59
-
-
0344053451
-
In vitro activation of CPP32 and Mch3 by Mch4, a novel human apoptotic cysteine protease containing two FADD-like domains
-
of special interest. See annotation [57].
-
Fernandes-Alnemri T, Armstrong RC, Krebs J, Srinivasula SM, Wang L, Bullrich F, Fritz LC, Trapani JA, Tomaselli KJ, Litwack G, Alnemri ES. In vitro activation of CPP32 and Mch3 by Mch4, a novel human apoptotic cysteine protease containing two FADD-like domains. of special interest Proc Natl Acad Sci USA. 93:1996;7464-7469 See annotation [57].
-
(1996)
Proc Natl Acad Sci USA
, vol.93
, pp. 7464-7469
-
-
Fernandes-Alnemri, T.1
Armstrong, R.C.2
Krebs, J.3
Srinivasula, S.M.4
Wang, L.5
Bullrich, F.6
Fritz, L.C.7
Trapani, J.A.8
Tomaselli, K.J.9
Litwack, G.10
Alnemri, E.S.11
-
60
-
-
0029066092
-
A protease-dependent TCR-induced death pathway in mature lymphocytes
-
Sarin A, Nakajima H, Henkart PA. A protease-dependent TCR-induced death pathway in mature lymphocytes. J Immunol. 154:1995;5806-5812.
-
(1995)
J Immunol
, vol.154
, pp. 5806-5812
-
-
Sarin, A.1
Nakajima, H.2
Henkart, P.A.3
-
61
-
-
0030472889
-
Different interleukin-1 beta converting enzyme (ICE) family protease requirements for the apoptotic death of T lymphocytes triggered by diverse stimuli
-
of special interest. This paper demonstrates that the ICE family proteases are required for apoptosis of thymocytes initiated by different stimuli.
-
Sarin A, Wu M-L, Henkart PA. Different interleukin-1 beta converting enzyme (ICE) family protease requirements for the apoptotic death of T lymphocytes triggered by diverse stimuli. of special interest J Exp Med. 184:1996;2445-2450 This paper demonstrates that the ICE family proteases are required for apoptosis of thymocytes initiated by different stimuli.
-
(1996)
J Exp Med
, vol.184
, pp. 2445-2450
-
-
Sarin, A.1
Wu, M.-L.2
Henkart, P.A.3
-
62
-
-
0027425110
-
Protease inhibitors selectively block T cell receptor-triggered programmed cell death in a murine T cell hybridoma and activated peripheral T cells
-
Sarin A, Adams DH, Henkart PA. Protease inhibitors selectively block T cell receptor-triggered programmed cell death in a murine T cell hybridoma and activated peripheral T cells. J Exp Med. 178:1993;1693-1700.
-
(1993)
J Exp Med
, vol.178
, pp. 1693-1700
-
-
Sarin, A.1
Adams, D.H.2
Henkart, P.A.3
-
63
-
-
0028984948
-
Mice deficient in IL-1 beta-converting enzyme are defective in production of mature IL-1 beta and resistant to endotoxic shock
-
Li P, Allen H, Banerjee S, Franklin S, Herzog L, Johnston C, McDowell J, Paskind M, Rodman L, Salfeld J, et al. Mice deficient in IL-1 beta-converting enzyme are defective in production of mature IL-1 beta and resistant to endotoxic shock. Cell. 80:1995;401-411.
-
(1995)
Cell
, vol.80
, pp. 401-411
-
-
Li, P.1
Allen, H.2
Banerjee, S.3
Franklin, S.4
Herzog, L.5
Johnston, C.6
McDowell, J.7
Paskind, M.8
Rodman, L.9
Salfeld, J.10
-
64
-
-
0028920863
-
Altered cytokine export and apoptosis in mice deficient in interleukin-1-beta converting enzyme
-
Kuida K, Lippke JA, Ku G, Harding MW, Livingston DJ, Su MS-S, Flavell RA. Altered cytokine export and apoptosis in mice deficient in interleukin-1-beta converting enzyme. Science. 267:1995;2000-2003.
-
(1995)
Science
, vol.267
, pp. 2000-2003
-
-
Kuida, K.1
Lippke, J.A.2
Ku, G.3
Harding, M.W.4
Livingston, D.J.5
Su, M.-S.6
Flavell, R.A.7
-
65
-
-
0029956641
-
Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice
-
Kuida K, Zheng TS, Na S, Kuan C-Y, Yang D, Karasuyama H, Rakic P, Flavell RA. Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice. Nature. 384:1996;368-372.
-
(1996)
Nature
, vol.384
, pp. 368-372
-
-
Kuida, K.1
Zheng, T.S.2
Na, S.3
Kuan, C.-Y.4
Yang, D.5
Karasuyama, H.6
Rakic, P.7
Flavell, R.A.8
-
66
-
-
0029834497
-
CrmA expression in T lymphocytes of transgenic mice inhibits CD95 (Fas/APO-1)-transduced apoptosis, but does not cause lymphadenopathy or autoimmune disease
-
of special interest. This paper provides an intriguing finding that the overexpression of CrmA in transgenic mice leads to blockage of Fas-mediated apoptosis, as expected, but does not result in an lymphoproliferative disease that is usually observed in Fas-deficient mice.
-
Smith KGC, Strasser A, Vaux DL. CrmA expression in T lymphocytes of transgenic mice inhibits CD95 (Fas/APO-1)-transduced apoptosis, but does not cause lymphadenopathy or autoimmune disease. of special interest EMBO J. 15:1996;5167-5176 This paper provides an intriguing finding that the overexpression of CrmA in transgenic mice leads to blockage of Fas-mediated apoptosis, as expected, but does not result in an lymphoproliferative disease that is usually observed in Fas-deficient mice.
-
(1996)
EMBO J
, vol.15
, pp. 5167-5176
-
-
Smith, K.G.C.1
Strasser, A.2
Vaux, D.L.3
-
67
-
-
0028873964
-
Fas- And tumor necrosis factor-induced apoptosis is inhibited by the poxvirus crmA gene product
-
Tewari M, Dixit VM. Fas- and tumor necrosis factor-induced apoptosis is inhibited by the poxvirus crmA gene product. J Biol Chem. 270:1995;3255-3260.
-
(1995)
J Biol Chem
, vol.270
, pp. 3255-3260
-
-
Tewari, M.1
Dixit, V.M.2
-
68
-
-
0029022365
-
Involvement of an ICE-like protease in Fas-mediated apoptosis
-
Enari M, Hug H, Nagata S. Involvement of an ICE-like protease in Fas-mediated apoptosis. Nature. 375:1995;78-81.
-
(1995)
Nature
, vol.375
, pp. 78-81
-
-
Enari, M.1
Hug, H.2
Nagata, S.3
-
69
-
-
0029005805
-
Requirement of an ICE/CED-3 protease for Fas/APO-1-mediated apoptosis
-
Los M, Van de Craen M, Penning LC, Schenk H, Westendorp M, Baeuerle PA, Droge W, Krammer PH, Fiers W, Schulze OK. Requirement of an ICE/CED-3 protease for Fas/APO-1-mediated apoptosis. Nature. 375:1995;81-83.
-
(1995)
Nature
, vol.375
, pp. 81-83
-
-
Los, M.1
Van De Craen, M.2
Penning, L.C.3
Schenk, H.4
Westendorp, M.5
Baeuerle, P.A.6
Droge, W.7
Krammer, P.H.8
Fiers, W.9
Schulze, O.K.10
-
70
-
-
0028102478
-
Cleavage of poly (ADP-ribose) polymerase by a proteinase with properties like ICE
-
Lazebnik YA, Kaufmann SH, Desnoyers S, Poirier GG, Earnshaw WC. Cleavage of poly (ADP-ribose) polymerase by a proteinase with properties like ICE. Nature. 371:1994;346-347.
-
(1994)
Nature
, vol.371
, pp. 346-347
-
-
Lazebnik, Y.A.1
Kaufmann, S.H.2
Desnoyers, S.3
Poirier, G.G.4
Earnshaw, W.C.5
-
71
-
-
0028990125
-
Yama/CPP32β mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase
-
Tewari M, Quan LT, O'Rourke K, Desnoyers S, Zeng Z, Beidler DR, Poirier GG, Salvesen GS, Dixit VM. Yama/CPP32β mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase. Cell. 81:1995;801-809.
-
(1995)
Cell
, vol.81
, pp. 801-809
-
-
Tewari, M.1
Quan, L.T.2
O'Rourke, K.3
Desnoyers, S.4
Zeng, Z.5
Beidler, D.R.6
Poirier, G.G.7
Salvesen, G.S.8
Dixit, V.M.9
-
72
-
-
0029890823
-
Apopain/CPP32 cleaves proteins that are essential for cellular repair: A fundamental principal of apoptotic death
-
Casciola-Rosen L, Nicholson DW, Chong T, Rowan KR, Thornberry NA, Miller DK, Rosen A. Apopain/CPP32 cleaves proteins that are essential for cellular repair: A fundamental principal of apoptotic death. J Exp Med. 183:1996;1957-1964.
-
(1996)
J Exp Med
, vol.183
, pp. 1957-1964
-
-
Casciola-Rosen, L.1
Nicholson, D.W.2
Chong, T.3
Rowan, K.R.4
Thornberry, N.A.5
Miller, D.K.6
Rosen, A.7
-
73
-
-
8944241298
-
DNA-dependent protein kinase catalytic subunit: A target for an ICE-like protease in apoptosis
-
Song Q, Lees-Miller SP, Kumar S, Zhang Z, Chan DW, Smith GC, Jackson SP, Alnemri ES, Litwack G, Khanna KK, Lavin MF. DNA-dependent protein kinase catalytic subunit: a target for an ICE-like protease in apoptosis. EMBO J. 15:1996;3238-3246.
-
(1996)
EMBO J
, vol.15
, pp. 3238-3246
-
-
Song, Q.1
Lees-Miller, S.P.2
Kumar, S.3
Zhang, Z.4
Chan, D.W.5
Smith, G.C.6
Jackson, S.P.7
Alnemri, E.S.8
Litwack, G.9
Khanna, K.K.10
Lavin, M.F.11
-
74
-
-
0030462563
-
Specific cleavage of the retinoblatoma protein by an ICE-like protease in apoptosis
-
Janicke RU, Walker PA, Lin XY, Porter AG. Specific cleavage of the retinoblatoma protein by an ICE-like protease in apoptosis. EMBO J. 15:1996;6969-6978.
-
(1996)
EMBO J
, vol.15
, pp. 6969-6978
-
-
Janicke, R.U.1
Walker, P.A.2
Lin, X.Y.3
Porter, A.G.4
-
75
-
-
0030249877
-
The involvement of an ATP-gated ion channel, P2x1, in thymocyte apoptosis
-
Chvatchko Y, Valera S, Aubry J-P, Renno T, Buell G, Bonnefoy J-Y. The involvement of an ATP-gated ion channel, P2x1, in thymocyte apoptosis. Immunity. 5:1996;275-283.
-
(1996)
Immunity
, vol.5
, pp. 275-283
-
-
Chvatchko, Y.1
Valera, S.2
Aubry, J.-P.3
Renno, T.4
Buell, G.5
Bonnefoy, J.-Y.6
-
76
-
-
0028922885
-
Massive cell death of immature hematopoietic cells and neurons in bcl-x-deficient mice
-
Motoyama N, Wang F, Roth KA, Sawa H, Nakayama K-I, Nakayama K, Negishi I, Senju S, Zhang Q, Fujii S, Loh DY. Massive cell death of immature hematopoietic cells and neurons in bcl-x-deficient mice. Science. 267:1995;1506-1510.
-
(1995)
Science
, vol.267
, pp. 1506-1510
-
-
Motoyama, N.1
Wang, F.2
Roth, K.A.3
Sawa, H.4
Nakayama, K.-I.5
Nakayama, K.6
Negishi, I.7
Senju, S.8
Zhang, Q.9
Fujii, S.10
Loh, D.Y.11
-
78
-
-
0027501839
-
Molecular cloning of a human cDNA encoding a novel protein, DAD1, whose defect causes apoptotic cell death in hamster BHK21 cells
-
Nakashima T, Sekiguchi T, Kuraoka A, Fukushima K, Shibata Y, Komiyama S, Nishimoto T. Molecular cloning of a human cDNA encoding a novel protein, DAD1, whose defect causes apoptotic cell death in hamster BHK21 cells. Mol Cell Biol. 13:1993;6367-6374.
-
(1993)
Mol Cell Biol
, vol.13
, pp. 6367-6374
-
-
Nakashima, T.1
Sekiguchi, T.2
Kuraoka, A.3
Fukushima, K.4
Shibata, Y.5
Komiyama, S.6
Nishimoto, T.7
-
79
-
-
0029115508
-
Dad-1, an endogenous programmed cell death suppressor in Caenorhabditis elegans and vertebrates
-
of special interest. This paper demonstrates that overexpression of Dad1 can rescue from some forms of apoptosis in worms.
-
Sugimoto A, Hozak RR, Nakashima T, Nishimoto T, Rothman JH. Dad-1, an endogenous programmed cell death suppressor in Caenorhabditis elegans and vertebrates. of special interest EMBO J. 14:1995;4434-4441 This paper demonstrates that overexpression of Dad1 can rescue from some forms of apoptosis in worms.
-
(1995)
EMBO J
, vol.14
, pp. 4434-4441
-
-
Sugimoto, A.1
Hozak, R.R.2
Nakashima, T.3
Nishimoto, T.4
Rothman, J.H.5
|